Antiviral activity of Vigna radiata extract against feline coronavirus in vitro

Feline infectious peritonitis (FIP) is a fatal illness caused by a mutated feline coronavirus (FCoV). This disease is characterized by its complexity, resulting from systemic infection, antibody-dependent enhancement (ADE), and challenges in accessing effective therapeutics. Extract derived from Vig...

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Main Authors: Ai-Ai Chou, Chung-Hui Lin, Yen-Chen Chang, Hui-Wen Chang, Yi-Chen Lin, Chia-Chen Pi, Yao-Ming Kan, Hao-Fen Chuang, Hui-Wen Chen
Format: Article
Language:English
Published: Taylor & Francis Group 2024-12-01
Series:Veterinary Quarterly
Subjects:
Online Access:https://www.tandfonline.com/doi/10.1080/01652176.2024.2349665
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author Ai-Ai Chou
Chung-Hui Lin
Yen-Chen Chang
Hui-Wen Chang
Yi-Chen Lin
Chia-Chen Pi
Yao-Ming Kan
Hao-Fen Chuang
Hui-Wen Chen
author_facet Ai-Ai Chou
Chung-Hui Lin
Yen-Chen Chang
Hui-Wen Chang
Yi-Chen Lin
Chia-Chen Pi
Yao-Ming Kan
Hao-Fen Chuang
Hui-Wen Chen
author_sort Ai-Ai Chou
collection DOAJ
description Feline infectious peritonitis (FIP) is a fatal illness caused by a mutated feline coronavirus (FCoV). This disease is characterized by its complexity, resulting from systemic infection, antibody-dependent enhancement (ADE), and challenges in accessing effective therapeutics. Extract derived from Vigna radiata (L.) R. Wilczek (VRE) exhibits various pharmacological effects, including antiviral activity. This study aimed to investigate the antiviral potential of VRE against FCoV, addressing the urgent need to advance the treatment of FIP. We explored the anti-FCoV activity, antiviral mechanism, and combinational application of VRE by means of in vitro antiviral assays. Our findings reveal that VRE effectively inhibited the cytopathic effect induced by FCoV, reduced viral proliferation, and downregulated spike protein expression. Moreover, VRE blocked FCoV in the early and late infection stages and was effective under in vitro ADE infection. Notably, when combined with VRE, the polymerase inhibitor GS-441524 or protease inhibitor GC376 suppressed FCoV more effectively than monotherapy. In conclusion, this study characterizes the antiviral property of VRE against FCoV in vitro, and VRE possesses therapeutic potential for FCoV treatment.
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publishDate 2024-12-01
publisher Taylor & Francis Group
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spelling doaj-art-7bbf5bf824654019a28ede2c756607212025-08-20T02:19:51ZengTaylor & Francis GroupVeterinary Quarterly0165-21761875-59412024-12-0144111310.1080/01652176.2024.2349665Antiviral activity of Vigna radiata extract against feline coronavirus in vitroAi-Ai Chou0Chung-Hui Lin1Yen-Chen Chang2Hui-Wen Chang3Yi-Chen Lin4Chia-Chen Pi5Yao-Ming Kan6Hao-Fen Chuang7Hui-Wen Chen8Department of Veterinary Medicine, National Taiwan University, Taipei, TaiwanNational Taiwan University Veterinary Hospital, National Taiwan University, Taipei, TaiwanGraduate Institute of Molecular and Comparative Pathobiology, School of Veterinary Medicine, National Taiwan University, Taipei, TaiwanGraduate Institute of Molecular and Comparative Pathobiology, School of Veterinary Medicine, National Taiwan University, Taipei, TaiwanKing’s Ground Biotech Co., Ltd., Pingtung, TaiwanKing’s Ground Biotech Co., Ltd., Pingtung, TaiwanDepartment of Veterinary Medicine, National Taiwan University, Taipei, TaiwanDepartment of Veterinary Medicine, National Taiwan University, Taipei, TaiwanDepartment of Veterinary Medicine, National Taiwan University, Taipei, TaiwanFeline infectious peritonitis (FIP) is a fatal illness caused by a mutated feline coronavirus (FCoV). This disease is characterized by its complexity, resulting from systemic infection, antibody-dependent enhancement (ADE), and challenges in accessing effective therapeutics. Extract derived from Vigna radiata (L.) R. Wilczek (VRE) exhibits various pharmacological effects, including antiviral activity. This study aimed to investigate the antiviral potential of VRE against FCoV, addressing the urgent need to advance the treatment of FIP. We explored the anti-FCoV activity, antiviral mechanism, and combinational application of VRE by means of in vitro antiviral assays. Our findings reveal that VRE effectively inhibited the cytopathic effect induced by FCoV, reduced viral proliferation, and downregulated spike protein expression. Moreover, VRE blocked FCoV in the early and late infection stages and was effective under in vitro ADE infection. Notably, when combined with VRE, the polymerase inhibitor GS-441524 or protease inhibitor GC376 suppressed FCoV more effectively than monotherapy. In conclusion, this study characterizes the antiviral property of VRE against FCoV in vitro, and VRE possesses therapeutic potential for FCoV treatment.https://www.tandfonline.com/doi/10.1080/01652176.2024.2349665Feline coronavirusfeline infectious peritonitisantiviralVigna radiata extractGS-441524GC376
spellingShingle Ai-Ai Chou
Chung-Hui Lin
Yen-Chen Chang
Hui-Wen Chang
Yi-Chen Lin
Chia-Chen Pi
Yao-Ming Kan
Hao-Fen Chuang
Hui-Wen Chen
Antiviral activity of Vigna radiata extract against feline coronavirus in vitro
Veterinary Quarterly
Feline coronavirus
feline infectious peritonitis
antiviral
Vigna radiata extract
GS-441524
GC376
title Antiviral activity of Vigna radiata extract against feline coronavirus in vitro
title_full Antiviral activity of Vigna radiata extract against feline coronavirus in vitro
title_fullStr Antiviral activity of Vigna radiata extract against feline coronavirus in vitro
title_full_unstemmed Antiviral activity of Vigna radiata extract against feline coronavirus in vitro
title_short Antiviral activity of Vigna radiata extract against feline coronavirus in vitro
title_sort antiviral activity of vigna radiata extract against feline coronavirus in vitro
topic Feline coronavirus
feline infectious peritonitis
antiviral
Vigna radiata extract
GS-441524
GC376
url https://www.tandfonline.com/doi/10.1080/01652176.2024.2349665
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