Clinical and genetic characteristics of distal arthrogryposis caused by mutations in the PIEZO2 gene

Mutations in the PIEZO2 gene, which is involved in the formation of the mechanosensitive cation channel Piezo2, can cause distal arthrogryposis type 3 (Gordon’s syndrome), type 5, and Marden–Walker syndrome. Clinical and genetic characteristics of two patients with distal arthrogryposis with autosom...

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Main Authors: T. V. Markova, E. L. Dadali, S. S. Nikitin, A.  F . Murtazina, O. L.  Mironovich, I.  V.   Kanivets
Format: Article
Language:Russian
Published: ABV-press 2021-09-01
Series:Нервно-мышечные болезни
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Online Access:https://nmb.abvpress.ru/jour/article/view/449
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author T. V. Markova
E. L. Dadali
S. S. Nikitin
A.  F . Murtazina
O. L.  Mironovich
I.  V.   Kanivets
author_facet T. V. Markova
E. L. Dadali
S. S. Nikitin
A.  F . Murtazina
O. L.  Mironovich
I.  V.   Kanivets
author_sort T. V. Markova
collection DOAJ
description Mutations in the PIEZO2 gene, which is involved in the formation of the mechanosensitive cation channel Piezo2, can cause distal arthrogryposis type 3 (Gordon’s syndrome), type 5, and Marden–Walker syndrome. Clinical and genetic characteristics of two patients with distal arthrogryposis with autosomal dominant inheritance and one with autosomal recessive inheritance are presented. Exome sequencing in one case revealed a de novo mutation in exon 52 of the PIEZO2gene c.8238G>A (p.Trp2746*, NM_022068.3), in the second, a known deletion of three nucleotides in exon 52 of the PIEZO2 gene c.8181_8183delAGA (p Glu2727del, NM_022068.3) was found, in the third, two mutations in the compound heterozygous state – a deletion of four nucleotides leading to a shift in the reading frame in c.1863_1866delTCAG(p.Ser621fs, NM_022068) and a deletion with putative coordinates 10785050–10789339 bp, spanning 15–16 exons of the PIEZO2 gene (NM_022068; LOD 2.40). The third patient was found to have two newly detected mutations in the compound heterozygous state – a deletion of four nucleotides, leading to a shift in the reading frame in exon 14, p.1863_1866delTCAG (p.Ser621fs, NM_022068) and a deletion with assumed coordinates 10785050–10789339 b. o., (NM_022068; LOD 2.40), spanning 15–16 exons of the PIEZO2 gene. The previous assumption was confirmed that heterozygous mutations are more often localized in exon 52 of the PIEZO2 gene and disrupt the amino acid sequence of the C‑terminal region of the protein molecule, while in patients with an autosomal recessive mode of inheritance of the mutation, the N‑terminal region is more often found.
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series Нервно-мышечные болезни
spelling doaj-art-7b02dadbbcc84e028d1b72a97d5240b02025-08-20T04:00:03ZrusABV-pressНервно-мышечные болезни2222-87212413-04432021-09-01112485510.17650/2222-8721-2021-11-2-48-55298Clinical and genetic characteristics of distal arthrogryposis caused by mutations in the PIEZO2 geneT. V. Markova0E. L. Dadali1S. S. Nikitin2A.  F . Murtazina3O. L.  Mironovich4I.  V.   Kanivets5Research Centre for Medical GeneticsResearch Centre for Medical GeneticsResearch Centre for Medical GeneticsResearch Centre for Medical GeneticsResearch Centre for Medical GeneticsMedical Genetic Center “Genomed”Mutations in the PIEZO2 gene, which is involved in the formation of the mechanosensitive cation channel Piezo2, can cause distal arthrogryposis type 3 (Gordon’s syndrome), type 5, and Marden–Walker syndrome. Clinical and genetic characteristics of two patients with distal arthrogryposis with autosomal dominant inheritance and one with autosomal recessive inheritance are presented. Exome sequencing in one case revealed a de novo mutation in exon 52 of the PIEZO2gene c.8238G>A (p.Trp2746*, NM_022068.3), in the second, a known deletion of three nucleotides in exon 52 of the PIEZO2 gene c.8181_8183delAGA (p Glu2727del, NM_022068.3) was found, in the third, two mutations in the compound heterozygous state – a deletion of four nucleotides leading to a shift in the reading frame in c.1863_1866delTCAG(p.Ser621fs, NM_022068) and a deletion with putative coordinates 10785050–10789339 bp, spanning 15–16 exons of the PIEZO2 gene (NM_022068; LOD 2.40). The third patient was found to have two newly detected mutations in the compound heterozygous state – a deletion of four nucleotides, leading to a shift in the reading frame in exon 14, p.1863_1866delTCAG (p.Ser621fs, NM_022068) and a deletion with assumed coordinates 10785050–10789339 b. o., (NM_022068; LOD 2.40), spanning 15–16 exons of the PIEZO2 gene. The previous assumption was confirmed that heterozygous mutations are more often localized in exon 52 of the PIEZO2 gene and disrupt the amino acid sequence of the C‑terminal region of the protein molecule, while in patients with an autosomal recessive mode of inheritance of the mutation, the N‑terminal region is more often found.https://nmb.abvpress.ru/jour/article/view/449distal arthrogryposismechanosensitive receptorgene piezo2mutationsmechanotransduction
spellingShingle T. V. Markova
E. L. Dadali
S. S. Nikitin
A.  F . Murtazina
O. L.  Mironovich
I.  V.   Kanivets
Clinical and genetic characteristics of distal arthrogryposis caused by mutations in the PIEZO2 gene
Нервно-мышечные болезни
distal arthrogryposis
mechanosensitive receptor
gene piezo2
mutations
mechanotransduction
title Clinical and genetic characteristics of distal arthrogryposis caused by mutations in the PIEZO2 gene
title_full Clinical and genetic characteristics of distal arthrogryposis caused by mutations in the PIEZO2 gene
title_fullStr Clinical and genetic characteristics of distal arthrogryposis caused by mutations in the PIEZO2 gene
title_full_unstemmed Clinical and genetic characteristics of distal arthrogryposis caused by mutations in the PIEZO2 gene
title_short Clinical and genetic characteristics of distal arthrogryposis caused by mutations in the PIEZO2 gene
title_sort clinical and genetic characteristics of distal arthrogryposis caused by mutations in the piezo2 gene
topic distal arthrogryposis
mechanosensitive receptor
gene piezo2
mutations
mechanotransduction
url https://nmb.abvpress.ru/jour/article/view/449
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