Epstein-Barr virus large tegument protein BPLF1 contributes to innate immune evasion through interference with toll-like receptor signaling.

Epstein-Barr virus large tegument protein BPLF1 contributes to innate immune evasion through interference with toll-like receptor signaling.

Viral infection triggers an early host response through activation of pattern recognition receptors, including Toll-like receptors (TLR). TLR signaling cascades induce production of type I interferons and proinflammatory cytokines involved in establishing an anti-viral state as well as in orchestrat...

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Bibliographic Details
Main Authors: Michiel van Gent, Steven G E Braem, Annemieke de Jong, Nezira Delagic, Janneke G C Peeters, Ingrid G J Boer, Paul N Moynagh, Elisabeth Kremmer, Emmanuel J Wiertz, Huib Ovaa, Bryan D Griffin, Maaike E Ressing
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2014-02-01
Series:PLoS Pathogens
Online Access:https://journals.plos.org/plospathogens/article/file?id=10.1371/journal.ppat.1003960&type=printable
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