Endothelial sensitivity to pro-fibrotic signals links systemic exposure to pulmonary fibrosis

Abstract Pulmonary fibrosis (PF) is a life-threatening condition characterised by excessive extracellular matrix deposition and tissue scarring. While much of PF research has focused on alveolar epithelial cells and fibroblasts, endothelial cells have emerged as active contributors to the disease in...

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Main Authors: Lena Möbus, Laura Ylä-Outinen, Luca Mannino, Giorgia Migliaccio, Karoliina Kosunen, Nicoletta D’Alessandro, Angela Serra, Dario Greco
Format: Article
Language:English
Published: Nature Publishing Group 2025-07-01
Series:Cell Death and Disease
Online Access:https://doi.org/10.1038/s41419-025-07824-5
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author Lena Möbus
Laura Ylä-Outinen
Luca Mannino
Giorgia Migliaccio
Karoliina Kosunen
Nicoletta D’Alessandro
Angela Serra
Dario Greco
author_facet Lena Möbus
Laura Ylä-Outinen
Luca Mannino
Giorgia Migliaccio
Karoliina Kosunen
Nicoletta D’Alessandro
Angela Serra
Dario Greco
author_sort Lena Möbus
collection DOAJ
description Abstract Pulmonary fibrosis (PF) is a life-threatening condition characterised by excessive extracellular matrix deposition and tissue scarring. While much of PF research has focused on alveolar epithelial cells and fibroblasts, endothelial cells have emerged as active contributors to the disease initiation, especially in the context of systemic exposure to pro-fibrotic substances. Here, we investigate early transcriptomic and secretory responses of human umbilical vein endothelial cells (HUVEC) to subtoxic doses of bleomycin, a known pro-fibrotic agent, and TGF-beta, a key cytokine in fibrosis. Bleomycin exposure induced a rapid and extensive shift in the endothelial transcriptional programme, including signatures of endothelial to mesenchymal transition, cellular senescence, and immune cell recruitment. These findings suggest endothelial cells as early initiators of pro-fibrotic signals, independent of contributions from other cell types. In contrast, TGF-beta effects were limited and transient, indicating its pro-fibrotic action may require another initial stimulus and interplay with other cells like fibroblasts. This study highlights the sensitivity of endothelial cells to pro-fibrotic exposure and provides a blueprint of early pro-fibrotic mechanisms that may operate on organs such as the lungs systemically via the endothelium, emphasising its pivotal role in PF pathogenesis.
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institution Kabale University
issn 2041-4889
language English
publishDate 2025-07-01
publisher Nature Publishing Group
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series Cell Death and Disease
spelling doaj-art-7908484a507341af90fbd6da66baf39e2025-08-20T03:43:34ZengNature Publishing GroupCell Death and Disease2041-48892025-07-0116111210.1038/s41419-025-07824-5Endothelial sensitivity to pro-fibrotic signals links systemic exposure to pulmonary fibrosisLena Möbus0Laura Ylä-Outinen1Luca Mannino2Giorgia Migliaccio3Karoliina Kosunen4Nicoletta D’Alessandro5Angela Serra6Dario Greco7Finnish Hub for Development and Validation of Integrated Approaches (FHAIVE), Faculty of Medicine and Health Technology, Tampere UniversityFinnish Hub for Development and Validation of Integrated Approaches (FHAIVE), Faculty of Medicine and Health Technology, Tampere UniversityFinnish Hub for Development and Validation of Integrated Approaches (FHAIVE), Faculty of Medicine and Health Technology, Tampere UniversityFinnish Hub for Development and Validation of Integrated Approaches (FHAIVE), Faculty of Medicine and Health Technology, Tampere UniversityFinnish Hub for Development and Validation of Integrated Approaches (FHAIVE), Faculty of Medicine and Health Technology, Tampere UniversityFinnish Hub for Development and Validation of Integrated Approaches (FHAIVE), Faculty of Medicine and Health Technology, Tampere UniversityFinnish Hub for Development and Validation of Integrated Approaches (FHAIVE), Faculty of Medicine and Health Technology, Tampere UniversityFinnish Hub for Development and Validation of Integrated Approaches (FHAIVE), Faculty of Medicine and Health Technology, Tampere UniversityAbstract Pulmonary fibrosis (PF) is a life-threatening condition characterised by excessive extracellular matrix deposition and tissue scarring. While much of PF research has focused on alveolar epithelial cells and fibroblasts, endothelial cells have emerged as active contributors to the disease initiation, especially in the context of systemic exposure to pro-fibrotic substances. Here, we investigate early transcriptomic and secretory responses of human umbilical vein endothelial cells (HUVEC) to subtoxic doses of bleomycin, a known pro-fibrotic agent, and TGF-beta, a key cytokine in fibrosis. Bleomycin exposure induced a rapid and extensive shift in the endothelial transcriptional programme, including signatures of endothelial to mesenchymal transition, cellular senescence, and immune cell recruitment. These findings suggest endothelial cells as early initiators of pro-fibrotic signals, independent of contributions from other cell types. In contrast, TGF-beta effects were limited and transient, indicating its pro-fibrotic action may require another initial stimulus and interplay with other cells like fibroblasts. This study highlights the sensitivity of endothelial cells to pro-fibrotic exposure and provides a blueprint of early pro-fibrotic mechanisms that may operate on organs such as the lungs systemically via the endothelium, emphasising its pivotal role in PF pathogenesis.https://doi.org/10.1038/s41419-025-07824-5
spellingShingle Lena Möbus
Laura Ylä-Outinen
Luca Mannino
Giorgia Migliaccio
Karoliina Kosunen
Nicoletta D’Alessandro
Angela Serra
Dario Greco
Endothelial sensitivity to pro-fibrotic signals links systemic exposure to pulmonary fibrosis
Cell Death and Disease
title Endothelial sensitivity to pro-fibrotic signals links systemic exposure to pulmonary fibrosis
title_full Endothelial sensitivity to pro-fibrotic signals links systemic exposure to pulmonary fibrosis
title_fullStr Endothelial sensitivity to pro-fibrotic signals links systemic exposure to pulmonary fibrosis
title_full_unstemmed Endothelial sensitivity to pro-fibrotic signals links systemic exposure to pulmonary fibrosis
title_short Endothelial sensitivity to pro-fibrotic signals links systemic exposure to pulmonary fibrosis
title_sort endothelial sensitivity to pro fibrotic signals links systemic exposure to pulmonary fibrosis
url https://doi.org/10.1038/s41419-025-07824-5
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