Effects of prolonged exposure to diclofenac sodium on strains of Staphylococcus aureus

Abstract Staphylococcus aureus is a pathogen commonly involved in the etiology of nosocomial and community-acquired infections. This microorganism adheres and forms biofilm on biotic and abiotic surfaces, which is a significant complication for infection control. More recently, non-antibiotic medica...

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Main Authors: A. A. de Lima-e-Silva, R.G. da Silva-Filho, A. S. Viana
Format: Article
Language:English
Published: Instituto Internacional de Ecologia 2025-04-01
Series:Brazilian Journal of Biology
Subjects:
Online Access:http://www.scielo.br/scielo.php?script=sci_arttext&pid=S1519-69842025000100166&lng=en&tlng=en
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author A. A. de Lima-e-Silva
R.G. da Silva-Filho
A. S. Viana
author_facet A. A. de Lima-e-Silva
R.G. da Silva-Filho
A. S. Viana
author_sort A. A. de Lima-e-Silva
collection DOAJ
description Abstract Staphylococcus aureus is a pathogen commonly involved in the etiology of nosocomial and community-acquired infections. This microorganism adheres and forms biofilm on biotic and abiotic surfaces, which is a significant complication for infection control. More recently, non-antibiotic medications, such as nonsteroidal anti-inflammatory agents (NSAIDs), have been studied as an alternative for treating infections caused by multidrug-resistant pathogens and infections associated with biofilms. This study assessed the effects of prolonged exposure of S. aureus strains to the NSAID diclofenac sodium regarding biofilm production, direct adhesion, hydrophobicity, susceptibility to antimicrobials, hemolysis, and biochemical profile. Some strains exposed to diclofenac showed increased direct adhesion to hydrophobic and hydrophilic surfaces and increased cell surface hydrophobicity. Two strains, one reference and one clinical, were subjected to repeated and prolonged passages in a culture medium with a diclofenac gradient. In one of them, the drug suppressed biofilm production in the assay with and without subinhibitory concentrations (sub-MICs) from NSAID. Additional repeated passages of this strain in a medium without diclofenac partially recovered the original biofilm-positive phenotype, but only in culture with sub-MICs of the drug. The original characteristics of the strains regarding susceptibility or resistance to antimicrobials and biochemical profile were not affected by prolonged exposure to diclofenac. However, the clinical isolate showed suppressed hemolytic activity.
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spelling doaj-art-78d4e89d679b46158f27c007769437e42025-08-20T02:14:38ZengInstituto Internacional de EcologiaBrazilian Journal of Biology1678-43752025-04-018510.1590/1519-6984.290421Effects of prolonged exposure to diclofenac sodium on strains of Staphylococcus aureusA. A. de Lima-e-Silvahttps://orcid.org/0000-0002-5247-5388R.G. da Silva-Filhohttps://orcid.org/0000-0001-6960-9314A. S. Vianahttps://orcid.org/0000-0003-0121-592XAbstract Staphylococcus aureus is a pathogen commonly involved in the etiology of nosocomial and community-acquired infections. This microorganism adheres and forms biofilm on biotic and abiotic surfaces, which is a significant complication for infection control. More recently, non-antibiotic medications, such as nonsteroidal anti-inflammatory agents (NSAIDs), have been studied as an alternative for treating infections caused by multidrug-resistant pathogens and infections associated with biofilms. This study assessed the effects of prolonged exposure of S. aureus strains to the NSAID diclofenac sodium regarding biofilm production, direct adhesion, hydrophobicity, susceptibility to antimicrobials, hemolysis, and biochemical profile. Some strains exposed to diclofenac showed increased direct adhesion to hydrophobic and hydrophilic surfaces and increased cell surface hydrophobicity. Two strains, one reference and one clinical, were subjected to repeated and prolonged passages in a culture medium with a diclofenac gradient. In one of them, the drug suppressed biofilm production in the assay with and without subinhibitory concentrations (sub-MICs) from NSAID. Additional repeated passages of this strain in a medium without diclofenac partially recovered the original biofilm-positive phenotype, but only in culture with sub-MICs of the drug. The original characteristics of the strains regarding susceptibility or resistance to antimicrobials and biochemical profile were not affected by prolonged exposure to diclofenac. However, the clinical isolate showed suppressed hemolytic activity.http://www.scielo.br/scielo.php?script=sci_arttext&pid=S1519-69842025000100166&lng=en&tlng=enStaphylococcus aureusdiclofenac sodiumresistancebiofilmdrug repositioning
spellingShingle A. A. de Lima-e-Silva
R.G. da Silva-Filho
A. S. Viana
Effects of prolonged exposure to diclofenac sodium on strains of Staphylococcus aureus
Brazilian Journal of Biology
Staphylococcus aureus
diclofenac sodium
resistance
biofilm
drug repositioning
title Effects of prolonged exposure to diclofenac sodium on strains of Staphylococcus aureus
title_full Effects of prolonged exposure to diclofenac sodium on strains of Staphylococcus aureus
title_fullStr Effects of prolonged exposure to diclofenac sodium on strains of Staphylococcus aureus
title_full_unstemmed Effects of prolonged exposure to diclofenac sodium on strains of Staphylococcus aureus
title_short Effects of prolonged exposure to diclofenac sodium on strains of Staphylococcus aureus
title_sort effects of prolonged exposure to diclofenac sodium on strains of staphylococcus aureus
topic Staphylococcus aureus
diclofenac sodium
resistance
biofilm
drug repositioning
url http://www.scielo.br/scielo.php?script=sci_arttext&pid=S1519-69842025000100166&lng=en&tlng=en
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