Effects of oral gavage with periodontal pathogens and plaque biofilm on gut microbiota ecology and intestinal tissue architecture in mice: a mechanistic study

ObjectiveThis study aimed to establish an in vitro model simulating periodontal biofilm architecture with three representative periodontal pathogens and evaluate its systemic impact through oral gavage administration in C57BL/6 mice. The findings provide mechanistic insights into the oral-gut axis d...

Full description

Saved in:
Bibliographic Details
Main Authors: Lan Huang, Song Ge, Kun Yang, Lian Duan, Li Gao, Yu Zhen Li, Yu Shi Yi
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-08-01
Series:Frontiers in Cellular and Infection Microbiology
Subjects:
Online Access:https://www.frontiersin.org/articles/10.3389/fcimb.2025.1589055/full
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850043831229087744
author Lan Huang
Song Ge
Kun Yang
Lian Duan
Li Gao
Yu Zhen Li
Yu Shi Yi
author_facet Lan Huang
Song Ge
Kun Yang
Lian Duan
Li Gao
Yu Zhen Li
Yu Shi Yi
author_sort Lan Huang
collection DOAJ
description ObjectiveThis study aimed to establish an in vitro model simulating periodontal biofilm architecture with three representative periodontal pathogens and evaluate its systemic impact through oral gavage administration in C57BL/6 mice. The findings provide mechanistic insights into the oral-gut axis dysbiosis, elucidating potential pathways linking periodontal inflammation to gastrointestinal pathophysiology.MethodsFifty 7-week-old male C57BL/6 mice were randomized into five groups(n=10/group): control (H), F. nucleatum (F), P.gingivalis (P), S.sanguinis (S) and biofilm (BF, F.n + P.g + S.s) groups. Mice were gavaged twice weekly for 6 weeks with 1×109 CFU (F, P, BF groups) and 1×108 CFU (S group) of bacterial suspensions or PBS (H group). Post-intervention, fecal and colon tissues were collected for 16S rRNA sequencing, H&E staining, immunohistochemistry (Occludin expression), and qRT-PCR analysis of inflammatory markers(IL18, TNF-α, IL-1β, B220, F4/80, NOS2, ARG1).ResultsA stable in vitro three-species biofilm model was successfully established to mimic the ecology of periodontal plaque. Gavage with F.n, P.g or the biofilm consortium (BF group) induced intestinal barrier disruption and elevated pro-inflammatory cytokines levels. PCR indicated a significant increase in the expression of IL-1β, TNF-α, B220, F4/80, and NOS2 in the P group (P < 0.001), while Arg-1 expression exhibited a significant decrease (P < 0.01). In the BF group, only TNF-α expression demonstrated a significant increase (P < 0.01). The expression of occludin is significantly reduced in the F/P/BF group, with the most pronounced decrease observed in the P group (P < 0.01). Gut microbiota alterations occurred in all groups. At the phylum level, the Firmicutes/Bacteroidetes (F/B) ratio increased in all three groups (F/P/BF group). Proteobacteria abundance rose substantially in the P group, while Desulfovibrio increased and Verrucomicrobia decreased in the F/P/BF and F/S groups, respectively. Genus-level analysis showed reduced Muribaculaceae in the F/P/BF group, alongside elevated pro-inflammatory bacteria (e.g., Enterococcus, Acinetobacter) and diminished beneficial bacteria (e.g., Bifidobacterium, Parabacteroides).ConclusionThese findings demonstrate that periodontal pathogens induce gut barrier compromise through microbiome-driven immunomodulation, with P. gingivalis exhibiting predominant pro-inflammatory effects.
format Article
id doaj-art-776bbd0a2d5c4df7a280f8b32a6c0762
institution DOAJ
issn 2235-2988
language English
publishDate 2025-08-01
publisher Frontiers Media S.A.
record_format Article
series Frontiers in Cellular and Infection Microbiology
spelling doaj-art-776bbd0a2d5c4df7a280f8b32a6c07622025-08-20T02:55:07ZengFrontiers Media S.A.Frontiers in Cellular and Infection Microbiology2235-29882025-08-011510.3389/fcimb.2025.15890551589055Effects of oral gavage with periodontal pathogens and plaque biofilm on gut microbiota ecology and intestinal tissue architecture in mice: a mechanistic studyLan HuangSong GeKun YangLian DuanLi GaoYu Zhen LiYu Shi YiObjectiveThis study aimed to establish an in vitro model simulating periodontal biofilm architecture with three representative periodontal pathogens and evaluate its systemic impact through oral gavage administration in C57BL/6 mice. The findings provide mechanistic insights into the oral-gut axis dysbiosis, elucidating potential pathways linking periodontal inflammation to gastrointestinal pathophysiology.MethodsFifty 7-week-old male C57BL/6 mice were randomized into five groups(n=10/group): control (H), F. nucleatum (F), P.gingivalis (P), S.sanguinis (S) and biofilm (BF, F.n + P.g + S.s) groups. Mice were gavaged twice weekly for 6 weeks with 1×109 CFU (F, P, BF groups) and 1×108 CFU (S group) of bacterial suspensions or PBS (H group). Post-intervention, fecal and colon tissues were collected for 16S rRNA sequencing, H&E staining, immunohistochemistry (Occludin expression), and qRT-PCR analysis of inflammatory markers(IL18, TNF-α, IL-1β, B220, F4/80, NOS2, ARG1).ResultsA stable in vitro three-species biofilm model was successfully established to mimic the ecology of periodontal plaque. Gavage with F.n, P.g or the biofilm consortium (BF group) induced intestinal barrier disruption and elevated pro-inflammatory cytokines levels. PCR indicated a significant increase in the expression of IL-1β, TNF-α, B220, F4/80, and NOS2 in the P group (P < 0.001), while Arg-1 expression exhibited a significant decrease (P < 0.01). In the BF group, only TNF-α expression demonstrated a significant increase (P < 0.01). The expression of occludin is significantly reduced in the F/P/BF group, with the most pronounced decrease observed in the P group (P < 0.01). Gut microbiota alterations occurred in all groups. At the phylum level, the Firmicutes/Bacteroidetes (F/B) ratio increased in all three groups (F/P/BF group). Proteobacteria abundance rose substantially in the P group, while Desulfovibrio increased and Verrucomicrobia decreased in the F/P/BF and F/S groups, respectively. Genus-level analysis showed reduced Muribaculaceae in the F/P/BF group, alongside elevated pro-inflammatory bacteria (e.g., Enterococcus, Acinetobacter) and diminished beneficial bacteria (e.g., Bifidobacterium, Parabacteroides).ConclusionThese findings demonstrate that periodontal pathogens induce gut barrier compromise through microbiome-driven immunomodulation, with P. gingivalis exhibiting predominant pro-inflammatory effects.https://www.frontiersin.org/articles/10.3389/fcimb.2025.1589055/fullFusobacterium nucleatumPorphyromonas gingivalisStreptococcus sanguinisoral-gut axisdysbiosistight junctions
spellingShingle Lan Huang
Song Ge
Kun Yang
Lian Duan
Li Gao
Yu Zhen Li
Yu Shi Yi
Effects of oral gavage with periodontal pathogens and plaque biofilm on gut microbiota ecology and intestinal tissue architecture in mice: a mechanistic study
Frontiers in Cellular and Infection Microbiology
Fusobacterium nucleatum
Porphyromonas gingivalis
Streptococcus sanguinis
oral-gut axis
dysbiosis
tight junctions
title Effects of oral gavage with periodontal pathogens and plaque biofilm on gut microbiota ecology and intestinal tissue architecture in mice: a mechanistic study
title_full Effects of oral gavage with periodontal pathogens and plaque biofilm on gut microbiota ecology and intestinal tissue architecture in mice: a mechanistic study
title_fullStr Effects of oral gavage with periodontal pathogens and plaque biofilm on gut microbiota ecology and intestinal tissue architecture in mice: a mechanistic study
title_full_unstemmed Effects of oral gavage with periodontal pathogens and plaque biofilm on gut microbiota ecology and intestinal tissue architecture in mice: a mechanistic study
title_short Effects of oral gavage with periodontal pathogens and plaque biofilm on gut microbiota ecology and intestinal tissue architecture in mice: a mechanistic study
title_sort effects of oral gavage with periodontal pathogens and plaque biofilm on gut microbiota ecology and intestinal tissue architecture in mice a mechanistic study
topic Fusobacterium nucleatum
Porphyromonas gingivalis
Streptococcus sanguinis
oral-gut axis
dysbiosis
tight junctions
url https://www.frontiersin.org/articles/10.3389/fcimb.2025.1589055/full
work_keys_str_mv AT lanhuang effectsoforalgavagewithperiodontalpathogensandplaquebiofilmongutmicrobiotaecologyandintestinaltissuearchitectureinmiceamechanisticstudy
AT songge effectsoforalgavagewithperiodontalpathogensandplaquebiofilmongutmicrobiotaecologyandintestinaltissuearchitectureinmiceamechanisticstudy
AT kunyang effectsoforalgavagewithperiodontalpathogensandplaquebiofilmongutmicrobiotaecologyandintestinaltissuearchitectureinmiceamechanisticstudy
AT lianduan effectsoforalgavagewithperiodontalpathogensandplaquebiofilmongutmicrobiotaecologyandintestinaltissuearchitectureinmiceamechanisticstudy
AT ligao effectsoforalgavagewithperiodontalpathogensandplaquebiofilmongutmicrobiotaecologyandintestinaltissuearchitectureinmiceamechanisticstudy
AT yuzhenli effectsoforalgavagewithperiodontalpathogensandplaquebiofilmongutmicrobiotaecologyandintestinaltissuearchitectureinmiceamechanisticstudy
AT yushiyi effectsoforalgavagewithperiodontalpathogensandplaquebiofilmongutmicrobiotaecologyandintestinaltissuearchitectureinmiceamechanisticstudy