The mitochondrial calcium uniporter regulates breast cancer progression via HIF‐1α

Abstract Triple‐negative breast cancer (TNBC) represents the most aggressive breast tumor subtype. However, the molecular determinants responsible for the metastatic TNBC phenotype are only partially understood. We here show that expression of the mitochondrial calcium uniporter (MCU), the selective...

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Main Authors: Anna Tosatto, Roberta Sommaggio, Carsten Kummerow, Robert B Bentham, Thomas S Blacker, Tunde Berecz, Michael R Duchen, Antonio Rosato, Ivan Bogeski, Gyorgy Szabadkai, Rosario Rizzuto, Cristina Mammucari
Format: Article
Language:English
Published: Springer Nature 2016-04-01
Series:EMBO Molecular Medicine
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Online Access:https://doi.org/10.15252/emmm.201606255
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author Anna Tosatto
Roberta Sommaggio
Carsten Kummerow
Robert B Bentham
Thomas S Blacker
Tunde Berecz
Michael R Duchen
Antonio Rosato
Ivan Bogeski
Gyorgy Szabadkai
Rosario Rizzuto
Cristina Mammucari
author_facet Anna Tosatto
Roberta Sommaggio
Carsten Kummerow
Robert B Bentham
Thomas S Blacker
Tunde Berecz
Michael R Duchen
Antonio Rosato
Ivan Bogeski
Gyorgy Szabadkai
Rosario Rizzuto
Cristina Mammucari
author_sort Anna Tosatto
collection DOAJ
description Abstract Triple‐negative breast cancer (TNBC) represents the most aggressive breast tumor subtype. However, the molecular determinants responsible for the metastatic TNBC phenotype are only partially understood. We here show that expression of the mitochondrial calcium uniporter (MCU), the selective channel responsible for mitochondrial Ca2+ uptake, correlates with tumor size and lymph node infiltration, suggesting that mitochondrial Ca2+ uptake might be instrumental for tumor growth and metastatic formation. Accordingly, MCU downregulation hampered cell motility and invasiveness and reduced tumor growth, lymph node infiltration, and lung metastasis in TNBC xenografts. In MCU‐silenced cells, production of mitochondrial reactive oxygen species (mROS) is blunted and expression of the hypoxia‐inducible factor‐1α (HIF‐1α) is reduced, suggesting a signaling role for mROS and HIF‐1α, downstream of mitochondrial Ca2+. Finally, in breast cancer mRNA samples, a positive correlation of MCU expression with HIF‐1α signaling route is present. Our results indicate that MCU plays a central role in TNBC growth and metastasis formation and suggest that mitochondrial Ca2+ uptake is a potential novel therapeutic target for clinical intervention.
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spelling doaj-art-772fae7597ff450391056d14ec5509ff2025-08-20T03:42:52ZengSpringer NatureEMBO Molecular Medicine1757-46761757-46842016-04-018556958510.15252/emmm.201606255The mitochondrial calcium uniporter regulates breast cancer progression via HIF‐1αAnna Tosatto0Roberta Sommaggio1Carsten Kummerow2Robert B Bentham3Thomas S Blacker4Tunde Berecz5Michael R Duchen6Antonio Rosato7Ivan Bogeski8Gyorgy Szabadkai9Rosario Rizzuto10Cristina Mammucari11Department of Biomedical Sciences, University of PaduaDepartment of Surgery, Oncology and Gastroenterology, University of PaduaDepartment of Biophysics, Center for Integrative Physiology and Molecular Medicine (CIPMM), School of Medicine, Saarland UniversityDepartment of Cell and Developmental Biology, Consortium for Mitochondrial Research, University College LondonDepartment of Cell and Developmental Biology, Consortium for Mitochondrial Research, University College LondonDepartment of Cell and Developmental Biology, Consortium for Mitochondrial Research, University College LondonDepartment of Cell and Developmental Biology, Consortium for Mitochondrial Research, University College LondonDepartment of Surgery, Oncology and Gastroenterology, University of PaduaDepartment of Biophysics, Center for Integrative Physiology and Molecular Medicine (CIPMM), School of Medicine, Saarland UniversityDepartment of Biomedical Sciences, University of PaduaDepartment of Biomedical Sciences, University of PaduaDepartment of Biomedical Sciences, University of PaduaAbstract Triple‐negative breast cancer (TNBC) represents the most aggressive breast tumor subtype. However, the molecular determinants responsible for the metastatic TNBC phenotype are only partially understood. We here show that expression of the mitochondrial calcium uniporter (MCU), the selective channel responsible for mitochondrial Ca2+ uptake, correlates with tumor size and lymph node infiltration, suggesting that mitochondrial Ca2+ uptake might be instrumental for tumor growth and metastatic formation. Accordingly, MCU downregulation hampered cell motility and invasiveness and reduced tumor growth, lymph node infiltration, and lung metastasis in TNBC xenografts. In MCU‐silenced cells, production of mitochondrial reactive oxygen species (mROS) is blunted and expression of the hypoxia‐inducible factor‐1α (HIF‐1α) is reduced, suggesting a signaling role for mROS and HIF‐1α, downstream of mitochondrial Ca2+. Finally, in breast cancer mRNA samples, a positive correlation of MCU expression with HIF‐1α signaling route is present. Our results indicate that MCU plays a central role in TNBC growth and metastasis formation and suggest that mitochondrial Ca2+ uptake is a potential novel therapeutic target for clinical intervention.https://doi.org/10.15252/emmm.201606255breast cancerHIF‐1αmetastasismitochondrial Ca2+ uptakereactive oxygen species
spellingShingle Anna Tosatto
Roberta Sommaggio
Carsten Kummerow
Robert B Bentham
Thomas S Blacker
Tunde Berecz
Michael R Duchen
Antonio Rosato
Ivan Bogeski
Gyorgy Szabadkai
Rosario Rizzuto
Cristina Mammucari
The mitochondrial calcium uniporter regulates breast cancer progression via HIF‐1α
EMBO Molecular Medicine
breast cancer
HIF‐1α
metastasis
mitochondrial Ca2+ uptake
reactive oxygen species
title The mitochondrial calcium uniporter regulates breast cancer progression via HIF‐1α
title_full The mitochondrial calcium uniporter regulates breast cancer progression via HIF‐1α
title_fullStr The mitochondrial calcium uniporter regulates breast cancer progression via HIF‐1α
title_full_unstemmed The mitochondrial calcium uniporter regulates breast cancer progression via HIF‐1α
title_short The mitochondrial calcium uniporter regulates breast cancer progression via HIF‐1α
title_sort mitochondrial calcium uniporter regulates breast cancer progression via hif 1α
topic breast cancer
HIF‐1α
metastasis
mitochondrial Ca2+ uptake
reactive oxygen species
url https://doi.org/10.15252/emmm.201606255
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