Personalized Nutrition for Obesity Management: A Mini-Review
Obesity is a pro-inflammatory state and a chronic health condition, which falls within the spectrum of immune-mediated inflammatory diseases (IMIDs). It is caused mainly by the consumption of excess calories and egregious food substances. A Personalized Food Avoidance Dietary Approach for Management...
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| Language: | English |
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Wolters Kluwer Medknow Publications
2024-10-01
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| Series: | International Journal of Medicine and Health Development |
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| Online Access: | https://journals.lww.com/ijmh/fulltext/2024/29040/personalized_nutrition_for_obesity_management__a.3.aspx |
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| author | Michael T. Okafor |
| author_facet | Michael T. Okafor |
| author_sort | Michael T. Okafor |
| collection | DOAJ |
| description | Obesity is a pro-inflammatory state and a chronic health condition, which falls within the spectrum of immune-mediated inflammatory diseases (IMIDs). It is caused mainly by the consumption of excess calories and egregious food substances. A Personalized Food Avoidance Dietary Approach for Management of IMIDs (PFA-DAMI) addresses primary and secondary immune intolerances of different individuals to dietary constituents. Exposure to toxic food substances is responsible for inflammatory immune dysfunction that mediates obesity. Hence, a PFA-DAMI is promising for the management of obesity. T cell dysfunction disease mediating models describing inflammatory disease processes underlying IMIDs exist. However, they do not highlight inflammatory processes underlying the diseases concerning toxin-mediated epigenetic T cell dysfunction. Online searches were conducted on databases, such as Google Scholar, PubMed, Biomed Central, and SciELO. Articles were reviewed using keywords, such as obesity, personalized nutrition, immune optimization/dysfunction, T lymphocyte activation/dysfunction, cytokines, and adipokines. There is a putative T cell toxin-mediated dysfunction disease model for IMIDs, which may apply to other diseases. The putative disease model may highlight the actual inflammatory immune dysfunctional processes underlying T cell disease mediation in inflammatory diseases, which may be validated by multiomic studies. Validation of the putative disease model using obesity as an example should pave the way for a better understanding of the role of personalized nutrition in obesity management. |
| format | Article |
| id | doaj-art-76ef0b8d55eb4c40875d3febcfe06188 |
| institution | OA Journals |
| issn | 2667-2863 |
| language | English |
| publishDate | 2024-10-01 |
| publisher | Wolters Kluwer Medknow Publications |
| record_format | Article |
| series | International Journal of Medicine and Health Development |
| spelling | doaj-art-76ef0b8d55eb4c40875d3febcfe061882025-08-20T02:13:20ZengWolters Kluwer Medknow PublicationsInternational Journal of Medicine and Health Development2667-28632024-10-0129428528810.4103/ijmh.ijmh_27_24Personalized Nutrition for Obesity Management: A Mini-ReviewMichael T. OkaforObesity is a pro-inflammatory state and a chronic health condition, which falls within the spectrum of immune-mediated inflammatory diseases (IMIDs). It is caused mainly by the consumption of excess calories and egregious food substances. A Personalized Food Avoidance Dietary Approach for Management of IMIDs (PFA-DAMI) addresses primary and secondary immune intolerances of different individuals to dietary constituents. Exposure to toxic food substances is responsible for inflammatory immune dysfunction that mediates obesity. Hence, a PFA-DAMI is promising for the management of obesity. T cell dysfunction disease mediating models describing inflammatory disease processes underlying IMIDs exist. However, they do not highlight inflammatory processes underlying the diseases concerning toxin-mediated epigenetic T cell dysfunction. Online searches were conducted on databases, such as Google Scholar, PubMed, Biomed Central, and SciELO. Articles were reviewed using keywords, such as obesity, personalized nutrition, immune optimization/dysfunction, T lymphocyte activation/dysfunction, cytokines, and adipokines. There is a putative T cell toxin-mediated dysfunction disease model for IMIDs, which may apply to other diseases. The putative disease model may highlight the actual inflammatory immune dysfunctional processes underlying T cell disease mediation in inflammatory diseases, which may be validated by multiomic studies. Validation of the putative disease model using obesity as an example should pave the way for a better understanding of the role of personalized nutrition in obesity management.https://journals.lww.com/ijmh/fulltext/2024/29040/personalized_nutrition_for_obesity_management__a.3.aspximmune optimization/dysfunction; inflammatory cytokines; obesity |
| spellingShingle | Michael T. Okafor Personalized Nutrition for Obesity Management: A Mini-Review International Journal of Medicine and Health Development immune optimization/dysfunction; inflammatory cytokines; obesity |
| title | Personalized Nutrition for Obesity Management: A Mini-Review |
| title_full | Personalized Nutrition for Obesity Management: A Mini-Review |
| title_fullStr | Personalized Nutrition for Obesity Management: A Mini-Review |
| title_full_unstemmed | Personalized Nutrition for Obesity Management: A Mini-Review |
| title_short | Personalized Nutrition for Obesity Management: A Mini-Review |
| title_sort | personalized nutrition for obesity management a mini review |
| topic | immune optimization/dysfunction; inflammatory cytokines; obesity |
| url | https://journals.lww.com/ijmh/fulltext/2024/29040/personalized_nutrition_for_obesity_management__a.3.aspx |
| work_keys_str_mv | AT michaeltokafor personalizednutritionforobesitymanagementaminireview |