The role of astrocytes from synaptic to non-synaptic plasticity

Information storage and transfer in the brain require a high computational power. Neuronal network display various local or global mechanisms to allow information storage and transfer in the brain. From synaptic to intrinsic plasticity, the rules of input–output function modulation have been well ch...

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Main Authors: Rafael Sanz-Gálvez, Dominic Falardeau, Arlette Kolta, Yanis Inglebert
Format: Article
Language:English
Published: Frontiers Media S.A. 2024-10-01
Series:Frontiers in Cellular Neuroscience
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Online Access:https://www.frontiersin.org/articles/10.3389/fncel.2024.1477985/full
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author Rafael Sanz-Gálvez
Rafael Sanz-Gálvez
Dominic Falardeau
Dominic Falardeau
Arlette Kolta
Arlette Kolta
Arlette Kolta
Yanis Inglebert
Yanis Inglebert
author_facet Rafael Sanz-Gálvez
Rafael Sanz-Gálvez
Dominic Falardeau
Dominic Falardeau
Arlette Kolta
Arlette Kolta
Arlette Kolta
Yanis Inglebert
Yanis Inglebert
author_sort Rafael Sanz-Gálvez
collection DOAJ
description Information storage and transfer in the brain require a high computational power. Neuronal network display various local or global mechanisms to allow information storage and transfer in the brain. From synaptic to intrinsic plasticity, the rules of input–output function modulation have been well characterized in neurons. In the past years, astrocytes have been suggested to increase the computational power of the brain and we are only just starting to uncover their role in information processing. Astrocytes maintain a close bidirectional communication with neurons to modify neuronal network excitability, transmission, axonal conduction, and plasticity through various mechanisms including the release of gliotransmitters or local ion homeostasis. Astrocytes have been significantly studied in the context of long-term or short-term synaptic plasticity, but this is not the only mechanism involved in memory formation. Plasticity of intrinsic neuronal excitability also participates in memory storage through regulation of voltage-gated ion channels or axonal morphological changes. Yet, the contribution of astrocytes to these other forms of non-synaptic plasticity remains to be investigated. In this review, we summarized the recent advances on the role of astrocytes in different forms of plasticity and discuss new directions and ideas to be explored regarding astrocytes-neuronal communication and regulation of plasticity.
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spelling doaj-art-757f56f296f94689a51b6b2b204a33d52025-08-20T01:47:50ZengFrontiers Media S.A.Frontiers in Cellular Neuroscience1662-51022024-10-011810.3389/fncel.2024.14779851477985The role of astrocytes from synaptic to non-synaptic plasticityRafael Sanz-Gálvez0Rafael Sanz-Gálvez1Dominic Falardeau2Dominic Falardeau3Arlette Kolta4Arlette Kolta5Arlette Kolta6Yanis Inglebert7Yanis Inglebert8Department of Neurosciences, Université de Montréal, Montréal, QC, CanadaCentre Interdisciplinaire de Recherche sur le Cerveau et l’Apprentissage (CIRCA), Montréal, QC, CanadaDepartment of Neurosciences, Université de Montréal, Montréal, QC, CanadaCentre Interdisciplinaire de Recherche sur le Cerveau et l’Apprentissage (CIRCA), Montréal, QC, CanadaDepartment of Neurosciences, Université de Montréal, Montréal, QC, CanadaCentre Interdisciplinaire de Recherche sur le Cerveau et l’Apprentissage (CIRCA), Montréal, QC, CanadaDepartment of Stomatology, Université de Montréal, Montréal, QC, CanadaDepartment of Neurosciences, Université de Montréal, Montréal, QC, CanadaCentre Interdisciplinaire de Recherche sur le Cerveau et l’Apprentissage (CIRCA), Montréal, QC, CanadaInformation storage and transfer in the brain require a high computational power. Neuronal network display various local or global mechanisms to allow information storage and transfer in the brain. From synaptic to intrinsic plasticity, the rules of input–output function modulation have been well characterized in neurons. In the past years, astrocytes have been suggested to increase the computational power of the brain and we are only just starting to uncover their role in information processing. Astrocytes maintain a close bidirectional communication with neurons to modify neuronal network excitability, transmission, axonal conduction, and plasticity through various mechanisms including the release of gliotransmitters or local ion homeostasis. Astrocytes have been significantly studied in the context of long-term or short-term synaptic plasticity, but this is not the only mechanism involved in memory formation. Plasticity of intrinsic neuronal excitability also participates in memory storage through regulation of voltage-gated ion channels or axonal morphological changes. Yet, the contribution of astrocytes to these other forms of non-synaptic plasticity remains to be investigated. In this review, we summarized the recent advances on the role of astrocytes in different forms of plasticity and discuss new directions and ideas to be explored regarding astrocytes-neuronal communication and regulation of plasticity.https://www.frontiersin.org/articles/10.3389/fncel.2024.1477985/fullastrocytessynaptic plasticitySTDPneuronal excitabilityaxonal plasticity
spellingShingle Rafael Sanz-Gálvez
Rafael Sanz-Gálvez
Dominic Falardeau
Dominic Falardeau
Arlette Kolta
Arlette Kolta
Arlette Kolta
Yanis Inglebert
Yanis Inglebert
The role of astrocytes from synaptic to non-synaptic plasticity
Frontiers in Cellular Neuroscience
astrocytes
synaptic plasticity
STDP
neuronal excitability
axonal plasticity
title The role of astrocytes from synaptic to non-synaptic plasticity
title_full The role of astrocytes from synaptic to non-synaptic plasticity
title_fullStr The role of astrocytes from synaptic to non-synaptic plasticity
title_full_unstemmed The role of astrocytes from synaptic to non-synaptic plasticity
title_short The role of astrocytes from synaptic to non-synaptic plasticity
title_sort role of astrocytes from synaptic to non synaptic plasticity
topic astrocytes
synaptic plasticity
STDP
neuronal excitability
axonal plasticity
url https://www.frontiersin.org/articles/10.3389/fncel.2024.1477985/full
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