An African ancestry-specific allele of CTLA4 confers protection against rheumatoid arthritis in African Americans.

Cytotoxic T-lymphocyte associated protein 4 (CTLA4) is a negative regulator of T-cell proliferation. Polymorphisms in CTLA4 have been inconsistently associated with susceptibility to rheumatoid arthritis (RA) in populations of European ancestry but have not been examined in African Americans. The pr...

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Main Authors: James M Kelley, Laura B Hughes, Jeffrey D Faggard, Maria I Danila, Monica H Crawford, Yuanqing Edberg, Miguel A Padilla, Hemant K Tiwari, Andrew O Westfall, Graciela S Alarcón, Doyt L Conn, Beth L Jonas, Leigh F Callahan, Edwin A Smith, Richard D Brasington, David B Allison, Robert P Kimberly, Larry W Moreland, Jeffrey C Edberg, S Louis Bridges
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2009-03-01
Series:PLoS Genetics
Online Access:https://journals.plos.org/plosgenetics/article/file?id=10.1371/journal.pgen.1000424&type=printable
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author James M Kelley
Laura B Hughes
Jeffrey D Faggard
Maria I Danila
Monica H Crawford
Yuanqing Edberg
Miguel A Padilla
Hemant K Tiwari
Andrew O Westfall
Graciela S Alarcón
Doyt L Conn
Beth L Jonas
Leigh F Callahan
Edwin A Smith
Richard D Brasington
David B Allison
Robert P Kimberly
Larry W Moreland
Jeffrey C Edberg
S Louis Bridges
author_facet James M Kelley
Laura B Hughes
Jeffrey D Faggard
Maria I Danila
Monica H Crawford
Yuanqing Edberg
Miguel A Padilla
Hemant K Tiwari
Andrew O Westfall
Graciela S Alarcón
Doyt L Conn
Beth L Jonas
Leigh F Callahan
Edwin A Smith
Richard D Brasington
David B Allison
Robert P Kimberly
Larry W Moreland
Jeffrey C Edberg
S Louis Bridges
author_sort James M Kelley
collection DOAJ
description Cytotoxic T-lymphocyte associated protein 4 (CTLA4) is a negative regulator of T-cell proliferation. Polymorphisms in CTLA4 have been inconsistently associated with susceptibility to rheumatoid arthritis (RA) in populations of European ancestry but have not been examined in African Americans. The prevalence of RA in most populations of European and Asian ancestry is approximately 1.0%; RA is purportedly less common in black Africans, with little known about its prevalence in African Americans. We sought to determine if CTLA4 polymorphisms are associated with RA in African Americans. We performed a 2-stage analysis of 12 haplotype tagging single nucleotide polymorphisms (SNPs) across CTLA4 in a total of 505 African American RA patients and 712 African American controls using Illumina and TaqMan platforms. The minor allele (G) of the rs231778 SNP was 0.054 in RA patients, compared to 0.209 in controls (4.462 x 10(-26), Fisher's exact). The presence of the G allele was associated with a substantially reduced odds ratio (OR) of having RA (AG+GG genotypes vs. AA genotype, OR 0.19, 95% CI: 0.13-0.26, p = 2.4 x 10(-28), Fisher's exact), suggesting a protective effect. This SNP is polymorphic in the African population (minor allele frequency [MAF] 0.09 in the Yoruba population), but is very rare in other groups (MAF = 0.002 in 530 Caucasians genotyped for this study). Markers associated with RA in populations of European ancestry (rs3087243 [+60C/T] and rs231775 [+49A/G]) were not replicated in African Americans. We found no confounding of association for rs231778 after stratifying for the HLA-DRB1 shared epitope, presence of anti-cyclic citrullinated peptide antibody, or degree of admixture from the European population. An African ancestry-specific genetic variant of CTLA4 appears to be associated with protection from RA in African Americans. This finding may explain, in part, the relatively low prevalence of RA in black African populations.
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spelling doaj-art-750a2b5d380e4bf59fa98a6bbe662f522025-08-20T02:00:51ZengPublic Library of Science (PLoS)PLoS Genetics1553-73901553-74042009-03-0153e100042410.1371/journal.pgen.1000424An African ancestry-specific allele of CTLA4 confers protection against rheumatoid arthritis in African Americans.James M KelleyLaura B HughesJeffrey D FaggardMaria I DanilaMonica H CrawfordYuanqing EdbergMiguel A PadillaHemant K TiwariAndrew O WestfallGraciela S AlarcónDoyt L ConnBeth L JonasLeigh F CallahanEdwin A SmithRichard D BrasingtonDavid B AllisonRobert P KimberlyLarry W MorelandJeffrey C EdbergS Louis BridgesCytotoxic T-lymphocyte associated protein 4 (CTLA4) is a negative regulator of T-cell proliferation. Polymorphisms in CTLA4 have been inconsistently associated with susceptibility to rheumatoid arthritis (RA) in populations of European ancestry but have not been examined in African Americans. The prevalence of RA in most populations of European and Asian ancestry is approximately 1.0%; RA is purportedly less common in black Africans, with little known about its prevalence in African Americans. We sought to determine if CTLA4 polymorphisms are associated with RA in African Americans. We performed a 2-stage analysis of 12 haplotype tagging single nucleotide polymorphisms (SNPs) across CTLA4 in a total of 505 African American RA patients and 712 African American controls using Illumina and TaqMan platforms. The minor allele (G) of the rs231778 SNP was 0.054 in RA patients, compared to 0.209 in controls (4.462 x 10(-26), Fisher's exact). The presence of the G allele was associated with a substantially reduced odds ratio (OR) of having RA (AG+GG genotypes vs. AA genotype, OR 0.19, 95% CI: 0.13-0.26, p = 2.4 x 10(-28), Fisher's exact), suggesting a protective effect. This SNP is polymorphic in the African population (minor allele frequency [MAF] 0.09 in the Yoruba population), but is very rare in other groups (MAF = 0.002 in 530 Caucasians genotyped for this study). Markers associated with RA in populations of European ancestry (rs3087243 [+60C/T] and rs231775 [+49A/G]) were not replicated in African Americans. We found no confounding of association for rs231778 after stratifying for the HLA-DRB1 shared epitope, presence of anti-cyclic citrullinated peptide antibody, or degree of admixture from the European population. An African ancestry-specific genetic variant of CTLA4 appears to be associated with protection from RA in African Americans. This finding may explain, in part, the relatively low prevalence of RA in black African populations.https://journals.plos.org/plosgenetics/article/file?id=10.1371/journal.pgen.1000424&type=printable
spellingShingle James M Kelley
Laura B Hughes
Jeffrey D Faggard
Maria I Danila
Monica H Crawford
Yuanqing Edberg
Miguel A Padilla
Hemant K Tiwari
Andrew O Westfall
Graciela S Alarcón
Doyt L Conn
Beth L Jonas
Leigh F Callahan
Edwin A Smith
Richard D Brasington
David B Allison
Robert P Kimberly
Larry W Moreland
Jeffrey C Edberg
S Louis Bridges
An African ancestry-specific allele of CTLA4 confers protection against rheumatoid arthritis in African Americans.
PLoS Genetics
title An African ancestry-specific allele of CTLA4 confers protection against rheumatoid arthritis in African Americans.
title_full An African ancestry-specific allele of CTLA4 confers protection against rheumatoid arthritis in African Americans.
title_fullStr An African ancestry-specific allele of CTLA4 confers protection against rheumatoid arthritis in African Americans.
title_full_unstemmed An African ancestry-specific allele of CTLA4 confers protection against rheumatoid arthritis in African Americans.
title_short An African ancestry-specific allele of CTLA4 confers protection against rheumatoid arthritis in African Americans.
title_sort african ancestry specific allele of ctla4 confers protection against rheumatoid arthritis in african americans
url https://journals.plos.org/plosgenetics/article/file?id=10.1371/journal.pgen.1000424&type=printable
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