Juvenile neuronal ceroid-lipofuscinosis: clinical and molecular investigation in a large family in Brazil

OBJECTIVE: Juvenile Neuronal Ceroid-Lipofuscinosis (JNCL, CLN 3, Batten Disease) (OMIM #204200) belongs to the most common group of neurodegenerative disorders of childhood. We report the clinical data and molecular analysis of a large Brazilian family. METHOD: Family composed of two consanguineous...

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Main Authors: Eugênia Ribeiro Valadares, Mayara Xavier Pizarro, Luiz Roberto Oliveira, Regina Helena Caldas de Amorim, Tarcísio Márcio Magalhães Pinheiro, Ulrike Grieben, Helena Hollanda Santos, Rachel Rabelo Queiroz, Guilherme de Castro Lopes, Ana Lúcia Brunialti Godard
Format: Article
Language:English
Published: Thieme Revinter Publicações 2011-02-01
Series:Arquivos de Neuro-Psiquiatria
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Online Access:http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0004-282X2011000100004&lng=en&tlng=en
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author Eugênia Ribeiro Valadares
Mayara Xavier Pizarro
Luiz Roberto Oliveira
Regina Helena Caldas de Amorim
Tarcísio Márcio Magalhães Pinheiro
Ulrike Grieben
Helena Hollanda Santos
Rachel Rabelo Queiroz
Guilherme de Castro Lopes
Ana Lúcia Brunialti Godard
author_facet Eugênia Ribeiro Valadares
Mayara Xavier Pizarro
Luiz Roberto Oliveira
Regina Helena Caldas de Amorim
Tarcísio Márcio Magalhães Pinheiro
Ulrike Grieben
Helena Hollanda Santos
Rachel Rabelo Queiroz
Guilherme de Castro Lopes
Ana Lúcia Brunialti Godard
author_sort Eugênia Ribeiro Valadares
collection DOAJ
description OBJECTIVE: Juvenile Neuronal Ceroid-Lipofuscinosis (JNCL, CLN 3, Batten Disease) (OMIM #204200) belongs to the most common group of neurodegenerative disorders of childhood. We report the clinical data and molecular analysis of a large Brazilian family. METHOD: Family composed of two consanguineous couples and thirty-two children. Clinical data of ten JNCL patients and molecular analyses on 13 participants were obtained. RESULTS: The large 1.02 kb deletion was detected. The most severe phenotype, with autistic behavior, tics and parkinsonism was seen in a 12-year-old female and a milder phenotype in a 14-year-old male. Nyctalopia was the first symptom in one deceased child. The visual loss of six patients has been first observed in the school and not at home. CONCLUSION: The report highlights the phenotypical intrafamily variation in 10 affected children of this family. The molecular investigation of this large family in our metabolic center turned possible the diagnosis, right approach and genetic counseling.
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spelling doaj-art-7385c7f4afed459e9f59dda6be8797af2025-08-20T02:06:19ZengThieme Revinter PublicaçõesArquivos de Neuro-Psiquiatria1678-42272011-02-01691131810.1590/S0004-282X2011000100004S0004-282X2011000100004Juvenile neuronal ceroid-lipofuscinosis: clinical and molecular investigation in a large family in BrazilEugênia Ribeiro Valadares0Mayara Xavier Pizarro1Luiz Roberto Oliveira2Regina Helena Caldas de Amorim3Tarcísio Márcio Magalhães Pinheiro4Ulrike Grieben5Helena Hollanda Santos6Rachel Rabelo Queiroz7Guilherme de Castro Lopes8Ana Lúcia Brunialti Godard9Universidade Federal de Minas GeraisUniversidade Federal de Minas GeraisUniversidade Federal de Minas GeraisUniversidade Federal de Minas GeraisUniversidade Federal de Minas GeraisNeuropädiatrieUniversidade Federal de Minas GeraisUniversidade Federal de Minas GeraisUniversidade Federal de Minas GeraisUniversidade Federal de Minas GeraisOBJECTIVE: Juvenile Neuronal Ceroid-Lipofuscinosis (JNCL, CLN 3, Batten Disease) (OMIM #204200) belongs to the most common group of neurodegenerative disorders of childhood. We report the clinical data and molecular analysis of a large Brazilian family. METHOD: Family composed of two consanguineous couples and thirty-two children. Clinical data of ten JNCL patients and molecular analyses on 13 participants were obtained. RESULTS: The large 1.02 kb deletion was detected. The most severe phenotype, with autistic behavior, tics and parkinsonism was seen in a 12-year-old female and a milder phenotype in a 14-year-old male. Nyctalopia was the first symptom in one deceased child. The visual loss of six patients has been first observed in the school and not at home. CONCLUSION: The report highlights the phenotypical intrafamily variation in 10 affected children of this family. The molecular investigation of this large family in our metabolic center turned possible the diagnosis, right approach and genetic counseling.http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0004-282X2011000100004&lng=en&tlng=endoença de Battenlipofuscinoses ceróides neuronaisreação em cadeia da polimerase
spellingShingle Eugênia Ribeiro Valadares
Mayara Xavier Pizarro
Luiz Roberto Oliveira
Regina Helena Caldas de Amorim
Tarcísio Márcio Magalhães Pinheiro
Ulrike Grieben
Helena Hollanda Santos
Rachel Rabelo Queiroz
Guilherme de Castro Lopes
Ana Lúcia Brunialti Godard
Juvenile neuronal ceroid-lipofuscinosis: clinical and molecular investigation in a large family in Brazil
Arquivos de Neuro-Psiquiatria
doença de Batten
lipofuscinoses ceróides neuronais
reação em cadeia da polimerase
title Juvenile neuronal ceroid-lipofuscinosis: clinical and molecular investigation in a large family in Brazil
title_full Juvenile neuronal ceroid-lipofuscinosis: clinical and molecular investigation in a large family in Brazil
title_fullStr Juvenile neuronal ceroid-lipofuscinosis: clinical and molecular investigation in a large family in Brazil
title_full_unstemmed Juvenile neuronal ceroid-lipofuscinosis: clinical and molecular investigation in a large family in Brazil
title_short Juvenile neuronal ceroid-lipofuscinosis: clinical and molecular investigation in a large family in Brazil
title_sort juvenile neuronal ceroid lipofuscinosis clinical and molecular investigation in a large family in brazil
topic doença de Batten
lipofuscinoses ceróides neuronais
reação em cadeia da polimerase
url http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0004-282X2011000100004&lng=en&tlng=en
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