Copper Metabolism and Cuproptosis in Ovarian Cancer

Objectives: This review provides a comprehensive analysis of copper metabolism and cuproptosis in ovarian cancer (OC), evaluating therapeutic strategies targeting copper to improve clinical outcomes. Mechanism:...

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Main Authors: Yuhong Xu, Qiang Wen, Haigang Ding, Jiangjing Shan, Qi Zhang
Format: Article
Language:English
Published: IMR Press 2025-05-01
Series:Clinical and Experimental Obstetrics & Gynecology
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Online Access:https://www.imrpress.com/journal/CEOG/52/5/10.31083/CEOG37476
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author Yuhong Xu
Qiang Wen
Haigang Ding
Jiangjing Shan
Qi Zhang
author_facet Yuhong Xu
Qiang Wen
Haigang Ding
Jiangjing Shan
Qi Zhang
author_sort Yuhong Xu
collection DOAJ
description Objectives: This review provides a comprehensive analysis of copper metabolism and cuproptosis in ovarian cancer (OC), evaluating therapeutic strategies targeting copper to improve clinical outcomes. Mechanism: OC exhibits the highest mortality rate among gynecological malignancies, characterized by its insidious onset and poor prognosis, underscoring the urgent need for novel treatment strategies. Copper metabolism and cuproptosis hold promising potential in regulating tumor progression and overcoming drug resistance, thereby opening new avenues for OC treatment. This article aims to elucidate the biological basis and potential therapeutic applications of copper metabolism and cuproptosis in treating OC. Findings in Brief: Cuproptosis, a copper-dependent mechanism of cell death mechanism, disrupts mitochondrial function by inducing the aggregation of lipoylated proteins and the loss of iron-sulfur (Fe-S) clusters. Copper metabolism imbalance promotes OC progression by modulating cancer cell function, contributing to chemotherapy resistance, and influencing responses to anti-angiogenesis, and immunotherapy. Copper ionophores, chelators, copper-based nanoparticles (NPs), and certain natural molecules represent potential therapeutic strategies for the treatment of OC. Conclusions: Targeting copper metabolism and cuproptosis offers innovative therapeutic strategies for OC, particularly in cases of treatment resistance. However, clinical validation of long-term safety, optimal dosing, and biomarker-guided therapies remains critical. Future research should prioritize translational studies to bridge mechanistic insights with patient-centered applications.
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issn 0390-6663
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series Clinical and Experimental Obstetrics & Gynecology
spelling doaj-art-7262b90157a94f8ea5f7a65d6027f89d2025-08-20T02:32:31ZengIMR PressClinical and Experimental Obstetrics & Gynecology0390-66632025-05-015253747610.31083/CEOG37476S0390-6663(25)02588-6Copper Metabolism and Cuproptosis in Ovarian CancerYuhong Xu0Qiang Wen1Haigang Ding2Jiangjing Shan3Qi Zhang4Department of Gynecology, Shaoxing People’s Hospital (The First Affiliated Hospital, Shaoxing University), 312000 Shaoxing, Zhejiang, ChinaDepartment of Gynecological Oncology, Zhejiang Cancer Hospital, 310022 Hangzhou, Zhejiang, ChinaDepartment of Gynecology, Shaoxing People’s Hospital (The First Affiliated Hospital, Shaoxing University), 312000 Shaoxing, Zhejiang, ChinaDepartment of Gynecology, Shaoxing People’s Hospital (The First Affiliated Hospital, Shaoxing University), 312000 Shaoxing, Zhejiang, ChinaDepartment of Ultrasound, Shaoxing People’s Hospital (The First Affiliated Hospital, Shaoxing University), 312000 Shaoxing, Zhejiang, ChinaObjectives: This review provides a comprehensive analysis of copper metabolism and cuproptosis in ovarian cancer (OC), evaluating therapeutic strategies targeting copper to improve clinical outcomes. Mechanism: OC exhibits the highest mortality rate among gynecological malignancies, characterized by its insidious onset and poor prognosis, underscoring the urgent need for novel treatment strategies. Copper metabolism and cuproptosis hold promising potential in regulating tumor progression and overcoming drug resistance, thereby opening new avenues for OC treatment. This article aims to elucidate the biological basis and potential therapeutic applications of copper metabolism and cuproptosis in treating OC. Findings in Brief: Cuproptosis, a copper-dependent mechanism of cell death mechanism, disrupts mitochondrial function by inducing the aggregation of lipoylated proteins and the loss of iron-sulfur (Fe-S) clusters. Copper metabolism imbalance promotes OC progression by modulating cancer cell function, contributing to chemotherapy resistance, and influencing responses to anti-angiogenesis, and immunotherapy. Copper ionophores, chelators, copper-based nanoparticles (NPs), and certain natural molecules represent potential therapeutic strategies for the treatment of OC. Conclusions: Targeting copper metabolism and cuproptosis offers innovative therapeutic strategies for OC, particularly in cases of treatment resistance. However, clinical validation of long-term safety, optimal dosing, and biomarker-guided therapies remains critical. Future research should prioritize translational studies to bridge mechanistic insights with patient-centered applications.https://www.imrpress.com/journal/CEOG/52/5/10.31083/CEOG37476copper metabolismcuproptosisovarian cancerreview
spellingShingle Yuhong Xu
Qiang Wen
Haigang Ding
Jiangjing Shan
Qi Zhang
Copper Metabolism and Cuproptosis in Ovarian Cancer
Clinical and Experimental Obstetrics & Gynecology
copper metabolism
cuproptosis
ovarian cancer
review
title Copper Metabolism and Cuproptosis in Ovarian Cancer
title_full Copper Metabolism and Cuproptosis in Ovarian Cancer
title_fullStr Copper Metabolism and Cuproptosis in Ovarian Cancer
title_full_unstemmed Copper Metabolism and Cuproptosis in Ovarian Cancer
title_short Copper Metabolism and Cuproptosis in Ovarian Cancer
title_sort copper metabolism and cuproptosis in ovarian cancer
topic copper metabolism
cuproptosis
ovarian cancer
review
url https://www.imrpress.com/journal/CEOG/52/5/10.31083/CEOG37476
work_keys_str_mv AT yuhongxu coppermetabolismandcuproptosisinovariancancer
AT qiangwen coppermetabolismandcuproptosisinovariancancer
AT haigangding coppermetabolismandcuproptosisinovariancancer
AT jiangjingshan coppermetabolismandcuproptosisinovariancancer
AT qizhang coppermetabolismandcuproptosisinovariancancer