A shared inflammatory signature across severe malaria syndromes manifested by transcriptomic, proteomic and metabolomic analyses

Abstract Factors governing the clinical trajectory of Plasmodium falciparum infection remain an important area of investigation. Here we present transcriptomic, proteomic and metabolomic analyses comparing clinical subtypes of severe Plasmodium falciparum malaria to matched controls with uncomplicat...

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Main Authors: Rafal S. Sobota, Emily M. Stucke, Drissa Coulibaly, Jonathan G. Lawton, Bryan E. Cummings, Savy Sebastian, Antoine Dara, James B. Munro, Amed Ouattara, Abdoulaye K. Kone, Bourama Kane, Karim Traoré, Bouréima Guindo, Bourama M. Tangara, Amadou Niangaly, Noah T. Ventimiglia, Modibo Daou, Issa Diarra, Youssouf Tolo, Mody Sissoko, Fayçal Maiga, Aichatou Diawara, Amidou Traore, Ali Thera, Matthew B. Laurens, Kirsten E. Lyke, Bourema Kouriba, Ogobara K. Doumbo, Christopher V. Plowe, David R. Goodlett, Joana C. Silva, Mahamadou A. Thera, Mark A. Travassos
Format: Article
Language:English
Published: Nature Portfolio 2025-05-01
Series:Nature Communications
Online Access:https://doi.org/10.1038/s41467-025-59281-5
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author Rafal S. Sobota
Emily M. Stucke
Drissa Coulibaly
Jonathan G. Lawton
Bryan E. Cummings
Savy Sebastian
Antoine Dara
James B. Munro
Amed Ouattara
Abdoulaye K. Kone
Bourama Kane
Karim Traoré
Bouréima Guindo
Bourama M. Tangara
Amadou Niangaly
Noah T. Ventimiglia
Modibo Daou
Issa Diarra
Youssouf Tolo
Mody Sissoko
Fayçal Maiga
Aichatou Diawara
Amidou Traore
Ali Thera
Matthew B. Laurens
Kirsten E. Lyke
Bourema Kouriba
Ogobara K. Doumbo
Christopher V. Plowe
David R. Goodlett
Joana C. Silva
Mahamadou A. Thera
Mark A. Travassos
author_facet Rafal S. Sobota
Emily M. Stucke
Drissa Coulibaly
Jonathan G. Lawton
Bryan E. Cummings
Savy Sebastian
Antoine Dara
James B. Munro
Amed Ouattara
Abdoulaye K. Kone
Bourama Kane
Karim Traoré
Bouréima Guindo
Bourama M. Tangara
Amadou Niangaly
Noah T. Ventimiglia
Modibo Daou
Issa Diarra
Youssouf Tolo
Mody Sissoko
Fayçal Maiga
Aichatou Diawara
Amidou Traore
Ali Thera
Matthew B. Laurens
Kirsten E. Lyke
Bourema Kouriba
Ogobara K. Doumbo
Christopher V. Plowe
David R. Goodlett
Joana C. Silva
Mahamadou A. Thera
Mark A. Travassos
author_sort Rafal S. Sobota
collection DOAJ
description Abstract Factors governing the clinical trajectory of Plasmodium falciparum infection remain an important area of investigation. Here we present transcriptomic, proteomic and metabolomic analyses comparing clinical subtypes of severe Plasmodium falciparum malaria to matched controls with uncomplicated disease in 79 children from Mali. MMP8, IL1R2, and ARG1 transcription is higher across cerebral malaria, severe malarial anemia, and concurrent cerebral malaria and severe malarial anemia, indicating a shared inflammatory signature. Tissue inhibitor of metalloproteinases 1 is the most upregulated protein in cerebral malaria, which along with elevated MMP8 and MMP9 transcription, underscores the importance of the metalloproteinase pathway in central nervous system pathophysiology. L-arginine metabolites are decreased in cerebral malaria, which coupled with increased ARG1 transcription suggests a putative mechanism impairing cerebral vasodilation. Using multi-omics approaches, we thus describe the inflammatory cascade in severe malaria syndromes, and identify potential therapeutic targets and biological markers.
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spelling doaj-art-724ff740a22a48559f133c6aef7eb65b2025-08-20T01:51:36ZengNature PortfolioNature Communications2041-17232025-05-0116111410.1038/s41467-025-59281-5A shared inflammatory signature across severe malaria syndromes manifested by transcriptomic, proteomic and metabolomic analysesRafal S. Sobota0Emily M. Stucke1Drissa Coulibaly2Jonathan G. Lawton3Bryan E. Cummings4Savy Sebastian5Antoine Dara6James B. Munro7Amed Ouattara8Abdoulaye K. Kone9Bourama Kane10Karim Traoré11Bouréima Guindo12Bourama M. Tangara13Amadou Niangaly14Noah T. Ventimiglia15Modibo Daou16Issa Diarra17Youssouf Tolo18Mody Sissoko19Fayçal Maiga20Aichatou Diawara21Amidou Traore22Ali Thera23Matthew B. Laurens24Kirsten E. Lyke25Bourema Kouriba26Ogobara K. Doumbo27Christopher V. Plowe28David R. Goodlett29Joana C. Silva30Mahamadou A. Thera31Mark A. Travassos32Malaria Research Program, Center for Vaccine Development and Global Health, University of Maryland School of MedicineMalaria Research Program, Center for Vaccine Development and Global Health, University of Maryland School of MedicineMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research Program, Center for Vaccine Development and Global Health, University of Maryland School of MedicineMalaria Research Program, Center for Vaccine Development and Global Health, University of Maryland School of MedicineMalaria Research Program, Center for Vaccine Development and Global Health, University of Maryland School of MedicineMalaria Research and Training Center, University of Sciences Techniques and TechnologiesInstitute for Genome Sciences, University of Maryland School of MedicineMalaria Research Program, Center for Vaccine Development and Global Health, University of Maryland School of MedicineMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research Program, Center for Vaccine Development and Global Health, University of Maryland School of MedicineMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research Program, Center for Vaccine Development and Global Health, University of Maryland School of MedicineMalaria Research Program, Center for Vaccine Development and Global Health, University of Maryland School of MedicineMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research Program, Center for Vaccine Development and Global Health, University of Maryland School of MedicineDepartment of Biochemistry and Microbiology, University of VictoriaInstitute for Genome Sciences, University of Maryland School of MedicineMalaria Research and Training Center, University of Sciences Techniques and TechnologiesMalaria Research Program, Center for Vaccine Development and Global Health, University of Maryland School of MedicineAbstract Factors governing the clinical trajectory of Plasmodium falciparum infection remain an important area of investigation. Here we present transcriptomic, proteomic and metabolomic analyses comparing clinical subtypes of severe Plasmodium falciparum malaria to matched controls with uncomplicated disease in 79 children from Mali. MMP8, IL1R2, and ARG1 transcription is higher across cerebral malaria, severe malarial anemia, and concurrent cerebral malaria and severe malarial anemia, indicating a shared inflammatory signature. Tissue inhibitor of metalloproteinases 1 is the most upregulated protein in cerebral malaria, which along with elevated MMP8 and MMP9 transcription, underscores the importance of the metalloproteinase pathway in central nervous system pathophysiology. L-arginine metabolites are decreased in cerebral malaria, which coupled with increased ARG1 transcription suggests a putative mechanism impairing cerebral vasodilation. Using multi-omics approaches, we thus describe the inflammatory cascade in severe malaria syndromes, and identify potential therapeutic targets and biological markers.https://doi.org/10.1038/s41467-025-59281-5
spellingShingle Rafal S. Sobota
Emily M. Stucke
Drissa Coulibaly
Jonathan G. Lawton
Bryan E. Cummings
Savy Sebastian
Antoine Dara
James B. Munro
Amed Ouattara
Abdoulaye K. Kone
Bourama Kane
Karim Traoré
Bouréima Guindo
Bourama M. Tangara
Amadou Niangaly
Noah T. Ventimiglia
Modibo Daou
Issa Diarra
Youssouf Tolo
Mody Sissoko
Fayçal Maiga
Aichatou Diawara
Amidou Traore
Ali Thera
Matthew B. Laurens
Kirsten E. Lyke
Bourema Kouriba
Ogobara K. Doumbo
Christopher V. Plowe
David R. Goodlett
Joana C. Silva
Mahamadou A. Thera
Mark A. Travassos
A shared inflammatory signature across severe malaria syndromes manifested by transcriptomic, proteomic and metabolomic analyses
Nature Communications
title A shared inflammatory signature across severe malaria syndromes manifested by transcriptomic, proteomic and metabolomic analyses
title_full A shared inflammatory signature across severe malaria syndromes manifested by transcriptomic, proteomic and metabolomic analyses
title_fullStr A shared inflammatory signature across severe malaria syndromes manifested by transcriptomic, proteomic and metabolomic analyses
title_full_unstemmed A shared inflammatory signature across severe malaria syndromes manifested by transcriptomic, proteomic and metabolomic analyses
title_short A shared inflammatory signature across severe malaria syndromes manifested by transcriptomic, proteomic and metabolomic analyses
title_sort shared inflammatory signature across severe malaria syndromes manifested by transcriptomic proteomic and metabolomic analyses
url https://doi.org/10.1038/s41467-025-59281-5
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