Release of tumor necrosis factor-α and prostanoids in whole blood cultures after in vivo exposure to low-dose aspirin

Background: The preventive effect of low-dose aspirin in cardiovascular disease is generally attributed to its antiplatelet action caused by differential inhibition of platelet cyclooxygenase-1. However, there is evidence that aspirin also affects release of inflammatory cytokines, including tumor n...

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Main Authors: Ilse Beckmann, Shlomo Ben-Efraim, Monica Vervoort, Henk C. S. Wallenburg
Format: Article
Language:English
Published: Wiley 2001-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1080/09629350120054554
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author Ilse Beckmann
Shlomo Ben-Efraim
Monica Vervoort
Henk C. S. Wallenburg
author_facet Ilse Beckmann
Shlomo Ben-Efraim
Monica Vervoort
Henk C. S. Wallenburg
author_sort Ilse Beckmann
collection DOAJ
description Background: The preventive effect of low-dose aspirin in cardiovascular disease is generally attributed to its antiplatelet action caused by differential inhibition of platelet cyclooxygenase-1. However, there is evidence that aspirin also affects release of inflammatory cytokines, including tumor necrosis factor-α (TNF-α). It is not known whether this is caused by direct action on the cytokine pathway or indirectly through cyclooxygenase inhibition and altered prostanoid synthesis, or both.
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institution Kabale University
issn 0962-9351
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publishDate 2001-01-01
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series Mediators of Inflammation
spelling doaj-art-71dce5b5d7f74367979923ef6b9a72dd2025-08-20T03:33:42ZengWileyMediators of Inflammation0962-93511466-18612001-01-01102818810.1080/09629350120054554Release of tumor necrosis factor-α and prostanoids in whole blood cultures after in vivo exposure to low-dose aspirinIlse Beckmann0Shlomo Ben-Efraim1Monica Vervoort2Henk C. S. Wallenburg3Department of Obstetrics and Gynecology, Erasmus University School of Medicine and Health Sciences, P.O.B. 1738, Rotterdam 3000 DR, The NetherlandsDepartment of Human Microbiology, Sackler School of Medicine, Tel-Aviv University, Tel Aviv, IsraelDepartment of Obstetrics and Gynecology, Erasmus University School of Medicine and Health Sciences, P.O.B. 1738, Rotterdam 3000 DR, The NetherlandsDepartment of Obstetrics and Gynecology, Erasmus University School of Medicine and Health Sciences, P.O.B. 1738, Rotterdam 3000 DR, The NetherlandsBackground: The preventive effect of low-dose aspirin in cardiovascular disease is generally attributed to its antiplatelet action caused by differential inhibition of platelet cyclooxygenase-1. However, there is evidence that aspirin also affects release of inflammatory cytokines, including tumor necrosis factor-α (TNF-α). It is not known whether this is caused by direct action on the cytokine pathway or indirectly through cyclooxygenase inhibition and altered prostanoid synthesis, or both.http://dx.doi.org/10.1080/09629350120054554
spellingShingle Ilse Beckmann
Shlomo Ben-Efraim
Monica Vervoort
Henk C. S. Wallenburg
Release of tumor necrosis factor-α and prostanoids in whole blood cultures after in vivo exposure to low-dose aspirin
Mediators of Inflammation
title Release of tumor necrosis factor-α and prostanoids in whole blood cultures after in vivo exposure to low-dose aspirin
title_full Release of tumor necrosis factor-α and prostanoids in whole blood cultures after in vivo exposure to low-dose aspirin
title_fullStr Release of tumor necrosis factor-α and prostanoids in whole blood cultures after in vivo exposure to low-dose aspirin
title_full_unstemmed Release of tumor necrosis factor-α and prostanoids in whole blood cultures after in vivo exposure to low-dose aspirin
title_short Release of tumor necrosis factor-α and prostanoids in whole blood cultures after in vivo exposure to low-dose aspirin
title_sort release of tumor necrosis factor α and prostanoids in whole blood cultures after in vivo exposure to low dose aspirin
url http://dx.doi.org/10.1080/09629350120054554
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