Marked differences in the effects of levetiracetam and its analogue brivaracetam on microglial, astrocytic, and neuronal density in the rat model of kainic acid-induced temporal lobe epilepsy

Efficient treatment of temporal lobe epilepsy (TLE) remains challenging due to limited understanding of cellular and network changes and the interference of novel antiepileptic drugs (AEDs) with tissue reorganisation. This study compared the effects of brivaracetam and levetiracetam on histological...

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Main Authors: Krisztina Kelemen, Máté Sárosi, Ágnes Csüdör, Károly Orbán-Kis, Hanga Kelemen, László Bába, Zsolt Gáll, Eszter Horváth, István Katona, Tibor Szilágyi
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-03-01
Series:Frontiers in Pharmacology
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Online Access:https://www.frontiersin.org/articles/10.3389/fphar.2025.1553545/full
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author Krisztina Kelemen
Krisztina Kelemen
Krisztina Kelemen
Máté Sárosi
Ágnes Csüdör
Károly Orbán-Kis
Hanga Kelemen
Hanga Kelemen
László Bába
Zsolt Gáll
Eszter Horváth
István Katona
István Katona
Tibor Szilágyi
author_facet Krisztina Kelemen
Krisztina Kelemen
Krisztina Kelemen
Máté Sárosi
Ágnes Csüdör
Károly Orbán-Kis
Hanga Kelemen
Hanga Kelemen
László Bába
Zsolt Gáll
Eszter Horváth
István Katona
István Katona
Tibor Szilágyi
author_sort Krisztina Kelemen
collection DOAJ
description Efficient treatment of temporal lobe epilepsy (TLE) remains challenging due to limited understanding of cellular and network changes and the interference of novel antiepileptic drugs (AEDs) with tissue reorganisation. This study compared the effects of brivaracetam and levetiracetam on histological alterations in key brain regions of the epileptic circuitry, namely, the hippocampus, amygdala, piriform cortex (PC), endopiriform nucleus (EPN) and paraventricular thalamic nucleus (PVT), using the kainic acid (KA) rat model of TLE. Male Wistar rats were assigned to sham-operated (SHAM), epileptic (EPI), brivaracetam- (BRV-EPI) and levetiracetam-treated (LEV-EPI) epileptic groups. Epileptic groups received KA in the right lateral ventricle, which induced status epilepticus followed by a 3-week recovery and latent period. Rats then underwent 3 weeks of oral brivaracetam, levetiracetam or placebo treatment with continuous video monitoring for seizure analysis. Subsequently, triple fluorescent immunolabeling assessed microglial, astrocytic, and neuronal changes. The results showed a drastic increase in microglia density in the EPI and BRV-EPI groups compared to control and LEV-EPI. The BRV-EPI group displayed a significantly higher microglia density than SHAM and EPI groups in the right CA1, CA3 and left CA1 regions, bilateral amygdalae, EPN, PVT and left PC. Astrocyte density was significantly elevated in hippocampal regions of the BRV-EPI group, while neuronal density decreased. Furthermore, brivaracetam did not reduce seizure activity in this disease phase. Significance: Brivaracetam treatment increased microglial activation under epileptic conditions in vivo in all examined brain-regions participating in the epileptic circuitry, in contrast to the effects of levetiracetam, highlighting differences in AED-induced histological alterations.
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spelling doaj-art-71028a67c15440efab5a75720d64bfc92025-08-20T03:02:07ZengFrontiers Media S.A.Frontiers in Pharmacology1663-98122025-03-011610.3389/fphar.2025.15535451553545Marked differences in the effects of levetiracetam and its analogue brivaracetam on microglial, astrocytic, and neuronal density in the rat model of kainic acid-induced temporal lobe epilepsyKrisztina Kelemen0Krisztina Kelemen1Krisztina Kelemen2Máté Sárosi3Ágnes Csüdör4Károly Orbán-Kis5Hanga Kelemen6Hanga Kelemen7László Bába8Zsolt Gáll9Eszter Horváth10István Katona11István Katona12Tibor Szilágyi13Department of Physiology, George Emil Palade University of Medicine, Pharmacy, Science, and Technology of Targu Mures, Târgu Mureș, RomaniaDoctoral School, George Emil Palade University of Medicine, Pharmacy, Science, and Technology of Targu Mures, Târgu Mureș, RomaniaMolecular Neurobiology Research Group, HUN-REN Institute of Experimental Medicine, Budapest, HungaryFaculty of Medicine, George Emil Palade University of Medicine, Pharmacy, Science, and Technology of Targu Mures, Târgu Mureș, RomaniaFaculty of Medicine, George Emil Palade University of Medicine, Pharmacy, Science, and Technology of Targu Mures, Târgu Mureș, RomaniaDepartment of Physiology, George Emil Palade University of Medicine, Pharmacy, Science, and Technology of Targu Mures, Târgu Mureș, RomaniaTranslational Behavioural Neuroscience Research Group, HUN-REN Institute of Experimental Medicine, Budapest, HungaryJános Szentágothai Neurosciences Division, Doctoral College, Semmelweis University, Budapest, HungaryDepartment of Pharmacology and Clinical Pharmacy, George Emil Palade University of Medicine, Pharmacy, Science and Technology of Targu Mures, Târgu Mures, RomaniaDepartment of Pharmacology and Clinical Pharmacy, George Emil Palade University of Medicine, Pharmacy, Science and Technology of Targu Mures, Târgu Mures, RomaniaMolecular Neurobiology Research Group, HUN-REN Institute of Experimental Medicine, Budapest, HungaryMolecular Neurobiology Research Group, HUN-REN Institute of Experimental Medicine, Budapest, HungaryDepartment of Psychological and Brain Sciences, Indiana University Bloomington, Bloomington, IN, United StatesDepartment of Physiology, George Emil Palade University of Medicine, Pharmacy, Science, and Technology of Targu Mures, Târgu Mureș, RomaniaEfficient treatment of temporal lobe epilepsy (TLE) remains challenging due to limited understanding of cellular and network changes and the interference of novel antiepileptic drugs (AEDs) with tissue reorganisation. This study compared the effects of brivaracetam and levetiracetam on histological alterations in key brain regions of the epileptic circuitry, namely, the hippocampus, amygdala, piriform cortex (PC), endopiriform nucleus (EPN) and paraventricular thalamic nucleus (PVT), using the kainic acid (KA) rat model of TLE. Male Wistar rats were assigned to sham-operated (SHAM), epileptic (EPI), brivaracetam- (BRV-EPI) and levetiracetam-treated (LEV-EPI) epileptic groups. Epileptic groups received KA in the right lateral ventricle, which induced status epilepticus followed by a 3-week recovery and latent period. Rats then underwent 3 weeks of oral brivaracetam, levetiracetam or placebo treatment with continuous video monitoring for seizure analysis. Subsequently, triple fluorescent immunolabeling assessed microglial, astrocytic, and neuronal changes. The results showed a drastic increase in microglia density in the EPI and BRV-EPI groups compared to control and LEV-EPI. The BRV-EPI group displayed a significantly higher microglia density than SHAM and EPI groups in the right CA1, CA3 and left CA1 regions, bilateral amygdalae, EPN, PVT and left PC. Astrocyte density was significantly elevated in hippocampal regions of the BRV-EPI group, while neuronal density decreased. Furthermore, brivaracetam did not reduce seizure activity in this disease phase. Significance: Brivaracetam treatment increased microglial activation under epileptic conditions in vivo in all examined brain-regions participating in the epileptic circuitry, in contrast to the effects of levetiracetam, highlighting differences in AED-induced histological alterations.https://www.frontiersin.org/articles/10.3389/fphar.2025.1553545/fulltemporal lobe epilepsy (TLE)antiepileptic drugsmicroglia-activationbrain-regionsepileptic circuitry
spellingShingle Krisztina Kelemen
Krisztina Kelemen
Krisztina Kelemen
Máté Sárosi
Ágnes Csüdör
Károly Orbán-Kis
Hanga Kelemen
Hanga Kelemen
László Bába
Zsolt Gáll
Eszter Horváth
István Katona
István Katona
Tibor Szilágyi
Marked differences in the effects of levetiracetam and its analogue brivaracetam on microglial, astrocytic, and neuronal density in the rat model of kainic acid-induced temporal lobe epilepsy
Frontiers in Pharmacology
temporal lobe epilepsy (TLE)
antiepileptic drugs
microglia-activation
brain-regions
epileptic circuitry
title Marked differences in the effects of levetiracetam and its analogue brivaracetam on microglial, astrocytic, and neuronal density in the rat model of kainic acid-induced temporal lobe epilepsy
title_full Marked differences in the effects of levetiracetam and its analogue brivaracetam on microglial, astrocytic, and neuronal density in the rat model of kainic acid-induced temporal lobe epilepsy
title_fullStr Marked differences in the effects of levetiracetam and its analogue brivaracetam on microglial, astrocytic, and neuronal density in the rat model of kainic acid-induced temporal lobe epilepsy
title_full_unstemmed Marked differences in the effects of levetiracetam and its analogue brivaracetam on microglial, astrocytic, and neuronal density in the rat model of kainic acid-induced temporal lobe epilepsy
title_short Marked differences in the effects of levetiracetam and its analogue brivaracetam on microglial, astrocytic, and neuronal density in the rat model of kainic acid-induced temporal lobe epilepsy
title_sort marked differences in the effects of levetiracetam and its analogue brivaracetam on microglial astrocytic and neuronal density in the rat model of kainic acid induced temporal lobe epilepsy
topic temporal lobe epilepsy (TLE)
antiepileptic drugs
microglia-activation
brain-regions
epileptic circuitry
url https://www.frontiersin.org/articles/10.3389/fphar.2025.1553545/full
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