Polyethylene microplastics promote nucleus pulposus cell senescence by inducing oxidative stress via TLR4/NOX2 axis

This study aimed to detect and characterize microplastics in intervertebral disc and investigate their effects and molecular mechanism on intervertebral disc degeneration. We collected intervertebral disc tissues from cervical, lumbar, and thoracolumbar segments and used Raman spectroscopy to identi...

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Main Authors: Weilin Zhang, Zhencong Li, Zhongwei Wang, Kuize Liu, Shengbang Huang, Jinguo Liang, Zhiwen Dai, Weixiong Guo, Chao Mao, Siyuan Chen, Jinsong Wei
Format: Article
Language:English
Published: Elsevier 2025-03-01
Series:Ecotoxicology and Environmental Safety
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Online Access:http://www.sciencedirect.com/science/article/pii/S0147651325002866
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author Weilin Zhang
Zhencong Li
Zhongwei Wang
Kuize Liu
Shengbang Huang
Jinguo Liang
Zhiwen Dai
Weixiong Guo
Chao Mao
Siyuan Chen
Jinsong Wei
author_facet Weilin Zhang
Zhencong Li
Zhongwei Wang
Kuize Liu
Shengbang Huang
Jinguo Liang
Zhiwen Dai
Weixiong Guo
Chao Mao
Siyuan Chen
Jinsong Wei
author_sort Weilin Zhang
collection DOAJ
description This study aimed to detect and characterize microplastics in intervertebral disc and investigate their effects and molecular mechanism on intervertebral disc degeneration. We collected intervertebral disc tissues from cervical, lumbar, and thoracolumbar segments and used Raman spectroscopy to identify and characterize microplastics. Among 80 samples, 47 contained microplastics, with polyethylene being the most prevalent type. To explore the effects of polyethylene microplastics (PE-MPs), we established a mouse model and a nucleus pulposus cell model. Reactive oxygen species (ROS) levels were assessed via immunofluorescence staining, cell viability was measured using the CCK-8 assay, and protein expression related to the Toll-like receptor 4 (TLR4)/NADPH oxidase 2 (NOX2) axis, oxidative stress, and nucleus pulposus degeneration were evaluated through western blotting and immunofluorescence staining. Results showed that PE-MPs exposure led to intervertebral disc degeneration by inducing oxidative stress and activating the TLR4 / NOX2 axis, which increased the senescence of nucleus pulposus cells. These effects were mitigated by TLR4 and NOX2 inhibitors. This research highlights the existence of microplastics in human intervertebral disc tissue and unveils a novel mechanism of nucleus pulposus cell senescence induced by PE-MPs, offering new avenues for clinical treatment of microplastic-related disc degeneration.
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spelling doaj-art-709558631c0c4f7296c23fae4e9ba4392025-08-20T02:57:33ZengElsevierEcotoxicology and Environmental Safety0147-65132025-03-0129211795010.1016/j.ecoenv.2025.117950Polyethylene microplastics promote nucleus pulposus cell senescence by inducing oxidative stress via TLR4/NOX2 axisWeilin Zhang0Zhencong Li1Zhongwei Wang2Kuize Liu3Shengbang Huang4Jinguo Liang5Zhiwen Dai6Weixiong Guo7Chao Mao8Siyuan Chen9Jinsong Wei10Department of Spinal Degeneration and Deformity Surgery, Affiliated Hospital of Guangdong Medical University, Zhanjiang 524001, ChinaDepartment of Spinal Degeneration and Deformity Surgery, Affiliated Hospital of Guangdong Medical University, Zhanjiang 524001, ChinaDepartment of Spinal Degeneration and Deformity Surgery, Affiliated Hospital of Guangdong Medical University, Zhanjiang 524001, ChinaDepartment of Spinal Degeneration and Deformity Surgery, Affiliated Hospital of Guangdong Medical University, Zhanjiang 524001, ChinaDepartment of Spinal Degeneration and Deformity Surgery, Affiliated Hospital of Guangdong Medical University, Zhanjiang 524001, ChinaDepartment of Spinal Degeneration and Deformity Surgery, Affiliated Hospital of Guangdong Medical University, Zhanjiang 524001, ChinaDepartment of Spinal Degeneration and Deformity Surgery, Affiliated Hospital of Guangdong Medical University, Zhanjiang 524001, ChinaDepartment of Spinal Degeneration and Deformity Surgery, Affiliated Hospital of Guangdong Medical University, Zhanjiang 524001, ChinaDepartment of Spinal Degeneration and Deformity Surgery, Affiliated Hospital of Guangdong Medical University, Zhanjiang 524001, ChinaDepartment of Spinal Degeneration and Deformity Surgery, Affiliated Hospital of Guangdong Medical University, Zhanjiang 524001, ChinaCorrespondence to: Department of Spinal Degeneration and Deformity Surgery, Affiliated Hospital of Guangdong Medical University, 57 South Renmin Avenue, Xiashan District, Zhanjiang, Guangdong 524001, China.; Department of Spinal Degeneration and Deformity Surgery, Affiliated Hospital of Guangdong Medical University, Zhanjiang 524001, ChinaThis study aimed to detect and characterize microplastics in intervertebral disc and investigate their effects and molecular mechanism on intervertebral disc degeneration. We collected intervertebral disc tissues from cervical, lumbar, and thoracolumbar segments and used Raman spectroscopy to identify and characterize microplastics. Among 80 samples, 47 contained microplastics, with polyethylene being the most prevalent type. To explore the effects of polyethylene microplastics (PE-MPs), we established a mouse model and a nucleus pulposus cell model. Reactive oxygen species (ROS) levels were assessed via immunofluorescence staining, cell viability was measured using the CCK-8 assay, and protein expression related to the Toll-like receptor 4 (TLR4)/NADPH oxidase 2 (NOX2) axis, oxidative stress, and nucleus pulposus degeneration were evaluated through western blotting and immunofluorescence staining. Results showed that PE-MPs exposure led to intervertebral disc degeneration by inducing oxidative stress and activating the TLR4 / NOX2 axis, which increased the senescence of nucleus pulposus cells. These effects were mitigated by TLR4 and NOX2 inhibitors. This research highlights the existence of microplastics in human intervertebral disc tissue and unveils a novel mechanism of nucleus pulposus cell senescence induced by PE-MPs, offering new avenues for clinical treatment of microplastic-related disc degeneration.http://www.sciencedirect.com/science/article/pii/S0147651325002866Intervertebral disc degenerationMicroplasticsReactive oxygen speciesToll-like receptor 4NADPH oxidase 2
spellingShingle Weilin Zhang
Zhencong Li
Zhongwei Wang
Kuize Liu
Shengbang Huang
Jinguo Liang
Zhiwen Dai
Weixiong Guo
Chao Mao
Siyuan Chen
Jinsong Wei
Polyethylene microplastics promote nucleus pulposus cell senescence by inducing oxidative stress via TLR4/NOX2 axis
Ecotoxicology and Environmental Safety
Intervertebral disc degeneration
Microplastics
Reactive oxygen species
Toll-like receptor 4
NADPH oxidase 2
title Polyethylene microplastics promote nucleus pulposus cell senescence by inducing oxidative stress via TLR4/NOX2 axis
title_full Polyethylene microplastics promote nucleus pulposus cell senescence by inducing oxidative stress via TLR4/NOX2 axis
title_fullStr Polyethylene microplastics promote nucleus pulposus cell senescence by inducing oxidative stress via TLR4/NOX2 axis
title_full_unstemmed Polyethylene microplastics promote nucleus pulposus cell senescence by inducing oxidative stress via TLR4/NOX2 axis
title_short Polyethylene microplastics promote nucleus pulposus cell senescence by inducing oxidative stress via TLR4/NOX2 axis
title_sort polyethylene microplastics promote nucleus pulposus cell senescence by inducing oxidative stress via tlr4 nox2 axis
topic Intervertebral disc degeneration
Microplastics
Reactive oxygen species
Toll-like receptor 4
NADPH oxidase 2
url http://www.sciencedirect.com/science/article/pii/S0147651325002866
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