LINC00365 promotes miR-221-5p to inhibit pyroptosis via Dicer in colorectal cancer

Pyroptosis, a newly discovered form of programmed cell death, is involved in the occurrence, development and drug resistance of a variety of tumors and has attracted increasing attention in recent years. LINC00365 is a novel lncRNA that has...

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Main Authors: Yang Weiqing, Huang Xiang, Lv Weibin, Jin Yuelong, Zhu Yiping
Format: Article
Language:English
Published: China Science Publishing & Media Ltd. 2024-10-01
Series:Acta Biochimica et Biophysica Sinica
Subjects:
Online Access:https://www.sciengine.com/doi/10.3724/abbs.2024173
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author Yang Weiqing
Huang Xiang
Lv Weibin
Jin Yuelong
Zhu Yiping
author_facet Yang Weiqing
Huang Xiang
Lv Weibin
Jin Yuelong
Zhu Yiping
author_sort Yang Weiqing
collection DOAJ
description Pyroptosis, a newly discovered form of programmed cell death, is involved in the occurrence, development and drug resistance of a variety of tumors and has attracted increasing attention in recent years. LINC00365 is a novel lncRNA that has rarely been reported before. We previously reported that LINC00365 expression in colorectal cancer is closely associated with poor patient outcomes. Additionally, LINC00365 was confirmed to be positively correlated with miR-221-5p, and miR-221-5p is negatively correlated with gasdermin-D (GSDMD) in colorectal cancer tissues. Bioinformatics analysis and luciferase reporter gene experiments revealed that GSDMD is the target gene of miR-221-5p. Cell function experiments and nude mouse tumor transplantation assays confirmed that LINC00365 could regulate the expressions of pyroptosis-related proteins such as Caspase-1, Caspase-11, NLRP3 and GSDMD. RNA pulldown and RNA immunoprecipitation experiments further elucidated the mechanism by which LINC00365 regulates miR-221-5p. In the present study, we observe that LINC00365 promotes the expression of miR-221-5p by binding to the Dicer enzyme to inhibit GSDMD and plays an antipyroptotic role. Our findings suggest that LINC00365 may serve as a molecular biomarker for estimating the prognosis of patients with colorectal cancer and as a potential therapeutic target for colorectal cancer.
format Article
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institution DOAJ
issn 1672-9145
language English
publishDate 2024-10-01
publisher China Science Publishing & Media Ltd.
record_format Article
series Acta Biochimica et Biophysica Sinica
spelling doaj-art-700fa6c0af464d3d94240dfec754b5e92025-08-20T03:10:42ZengChina Science Publishing & Media Ltd.Acta Biochimica et Biophysica Sinica1672-91452024-10-015752954110.3724/abbs.202417320d259ccLINC00365 promotes miR-221-5p to inhibit pyroptosis via Dicer in colorectal cancerYang Weiqing0Huang Xiang1Lv Weibin2Jin Yuelong3Zhu Yiping4["School of Graduate Studies, Wannan Medical College, Wuhu 241002, China","Department of Oncology, the First Affiliated Hospital of Wannan Medical College, Wuhu 241002, China"]["School of Public Health, Wannan Medical College, Wuhu 241002, China"]["School of Graduate Studies, Wannan Medical College, Wuhu 241002, China","Department of Oncology, the First Affiliated Hospital of Wannan Medical College, Wuhu 241002, China"]["School of Public Health, Wannan Medical College, Wuhu 241002, China"]["Department of Oncology, the First Affiliated Hospital of Wannan Medical College, Wuhu 241002, China"]Pyroptosis, a newly discovered form of programmed cell death, is involved in the occurrence, development and drug resistance of a variety of tumors and has attracted increasing attention in recent years. LINC00365 is a novel lncRNA that has rarely been reported before. We previously reported that LINC00365 expression in colorectal cancer is closely associated with poor patient outcomes. Additionally, LINC00365 was confirmed to be positively correlated with miR-221-5p, and miR-221-5p is negatively correlated with gasdermin-D (GSDMD) in colorectal cancer tissues. Bioinformatics analysis and luciferase reporter gene experiments revealed that GSDMD is the target gene of miR-221-5p. Cell function experiments and nude mouse tumor transplantation assays confirmed that LINC00365 could regulate the expressions of pyroptosis-related proteins such as Caspase-1, Caspase-11, NLRP3 and GSDMD. RNA pulldown and RNA immunoprecipitation experiments further elucidated the mechanism by which LINC00365 regulates miR-221-5p. In the present study, we observe that LINC00365 promotes the expression of miR-221-5p by binding to the Dicer enzyme to inhibit GSDMD and plays an antipyroptotic role. Our findings suggest that LINC00365 may serve as a molecular biomarker for estimating the prognosis of patients with colorectal cancer and as a potential therapeutic target for colorectal cancer.https://www.sciengine.com/doi/10.3724/abbs.2024173LINC00365miR-221-5ppyroptosiscolorectal cancer
spellingShingle Yang Weiqing
Huang Xiang
Lv Weibin
Jin Yuelong
Zhu Yiping
LINC00365 promotes miR-221-5p to inhibit pyroptosis via Dicer in colorectal cancer
Acta Biochimica et Biophysica Sinica
LINC00365
miR-221-5p
pyroptosis
colorectal cancer
title LINC00365 promotes miR-221-5p to inhibit pyroptosis via Dicer in colorectal cancer
title_full LINC00365 promotes miR-221-5p to inhibit pyroptosis via Dicer in colorectal cancer
title_fullStr LINC00365 promotes miR-221-5p to inhibit pyroptosis via Dicer in colorectal cancer
title_full_unstemmed LINC00365 promotes miR-221-5p to inhibit pyroptosis via Dicer in colorectal cancer
title_short LINC00365 promotes miR-221-5p to inhibit pyroptosis via Dicer in colorectal cancer
title_sort linc00365 promotes mir 221 5p to inhibit pyroptosis via dicer in colorectal cancer
topic LINC00365
miR-221-5p
pyroptosis
colorectal cancer
url https://www.sciengine.com/doi/10.3724/abbs.2024173
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AT huangxiang linc00365promotesmir2215ptoinhibitpyroptosisviadicerincolorectalcancer
AT lvweibin linc00365promotesmir2215ptoinhibitpyroptosisviadicerincolorectalcancer
AT jinyuelong linc00365promotesmir2215ptoinhibitpyroptosisviadicerincolorectalcancer
AT zhuyiping linc00365promotesmir2215ptoinhibitpyroptosisviadicerincolorectalcancer