Anti-inflammatory effects of Stephania tetrandra S. Moore on interleukin-6 production and experimental inflammatory disease models

Deregulation of interleukin-6 (IL-6) expression caused the synthesis and release of many inflammatory mediators. It is involved in chronic inflammation, autoimmune diseases, and malignancy. Stephania tetrandra S. Moore is a Chinese medicinal herb which has been used traditionary as a remedy for neur...

Full description

Saved in:
Bibliographic Details
Main Authors: H-S. Kang, Y-H. Kim, C-S. Lee, J-J. Lee, I. Choi, K-H. Pyun
Format: Article
Language:English
Published: Wiley 1996-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/S0962935196000415
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850222379680137216
author H-S. Kang
Y-H. Kim
C-S. Lee
J-J. Lee
I. Choi
K-H. Pyun
author_facet H-S. Kang
Y-H. Kim
C-S. Lee
J-J. Lee
I. Choi
K-H. Pyun
author_sort H-S. Kang
collection DOAJ
description Deregulation of interleukin-6 (IL-6) expression caused the synthesis and release of many inflammatory mediators. It is involved in chronic inflammation, autoimmune diseases, and malignancy. Stephania tetrandra S. Moore is a Chinese medicinal herb which has been used traditionary as a remedy for neuralgia and arthritis in China. To investigate the anti-inflammatory effects of S. tetrandra S. Moore in vitro and in vivo, its effects on the production of IL-6 and inflammatory mediators were analysed. When human monocytes/macrophages stimulated with silica were treated with 0.1–10 μg/ml S. tetranda S. Moore, the production of IL-6 was inhibited up to 50%. At these concentrations, it had no cytotoxicity effect on these cells. It also suppressed the production of IL-6 by alveolar macrophages stimulated with silica. In addition, it inhibited the release of superoxide anion and hydrogen peroxide from human monocytes/macrophages. To assess the anti-fibrosis effects of S. tetrandra S. Moore, its effects on in vivo experimental inflammatory models were evaluated. In the experimental silicosis model, IL-6 activities in the sera and in the culture supernatants of pulmonary fibroblasts were also inhibited by it. In vitro and in vivo treatment of S. tetrandra S. Moore reduced collagen production by rat lung fibroblasts and lung tissue. Also, S. tetrandra S. Moore reduced the levels of serum GOT and GPT in the rat cirrhosis model induced by CCL4, and it was effective in reducing hepatic fibrosis and nodular formation. Taken together, these data indicate that it has a potent anti-inflammatory and antifibrosis effect by reducing IL-6 production.
format Article
id doaj-art-6f67db74ae7744f3bb09b844c488cd49
institution OA Journals
issn 0962-9351
1466-1861
language English
publishDate 1996-01-01
publisher Wiley
record_format Article
series Mediators of Inflammation
spelling doaj-art-6f67db74ae7744f3bb09b844c488cd492025-08-20T02:06:22ZengWileyMediators of Inflammation0962-93511466-18611996-01-015428029110.1155/S0962935196000415Anti-inflammatory effects of Stephania tetrandra S. Moore on interleukin-6 production and experimental inflammatory disease modelsH-S. Kang0Y-H. Kim1C-S. Lee2J-J. Lee3I. Choi4K-H. Pyun5Immune Cell Signal Transduction R.U., Korea Research Institute of Bioscience and Biotechnology, KIST, Yusong, P.O. Box 115, Taejon 305-600, KoreaImmune Cell Signal Transduction R.U., Korea Research Institute of Bioscience and Biotechnology, KIST, Yusong, P.O. Box 115, Taejon 305-600, KoreaDepartment of Pathology, School of Medicine, Chungnam University, Taejon, KoreaImmune Cell Signal Transduction R.U., Korea Research Institute of Bioscience and Biotechnology, KIST, Yusong, P.O. Box 115, Taejon 305-600, KoreaImmune Cell Signal Transduction R.U., Korea Research Institute of Bioscience and Biotechnology, KIST, Yusong, P.O. Box 115, Taejon 305-600, KoreaImmune Cell Signal Transduction R.U., Korea Research Institute of Bioscience and Biotechnology, KIST, Yusong, P.O. Box 115, Taejon 305-600, KoreaDeregulation of interleukin-6 (IL-6) expression caused the synthesis and release of many inflammatory mediators. It is involved in chronic inflammation, autoimmune diseases, and malignancy. Stephania tetrandra S. Moore is a Chinese medicinal herb which has been used traditionary as a remedy for neuralgia and arthritis in China. To investigate the anti-inflammatory effects of S. tetrandra S. Moore in vitro and in vivo, its effects on the production of IL-6 and inflammatory mediators were analysed. When human monocytes/macrophages stimulated with silica were treated with 0.1–10 μg/ml S. tetranda S. Moore, the production of IL-6 was inhibited up to 50%. At these concentrations, it had no cytotoxicity effect on these cells. It also suppressed the production of IL-6 by alveolar macrophages stimulated with silica. In addition, it inhibited the release of superoxide anion and hydrogen peroxide from human monocytes/macrophages. To assess the anti-fibrosis effects of S. tetrandra S. Moore, its effects on in vivo experimental inflammatory models were evaluated. In the experimental silicosis model, IL-6 activities in the sera and in the culture supernatants of pulmonary fibroblasts were also inhibited by it. In vitro and in vivo treatment of S. tetrandra S. Moore reduced collagen production by rat lung fibroblasts and lung tissue. Also, S. tetrandra S. Moore reduced the levels of serum GOT and GPT in the rat cirrhosis model induced by CCL4, and it was effective in reducing hepatic fibrosis and nodular formation. Taken together, these data indicate that it has a potent anti-inflammatory and antifibrosis effect by reducing IL-6 production.http://dx.doi.org/10.1155/S0962935196000415
spellingShingle H-S. Kang
Y-H. Kim
C-S. Lee
J-J. Lee
I. Choi
K-H. Pyun
Anti-inflammatory effects of Stephania tetrandra S. Moore on interleukin-6 production and experimental inflammatory disease models
Mediators of Inflammation
title Anti-inflammatory effects of Stephania tetrandra S. Moore on interleukin-6 production and experimental inflammatory disease models
title_full Anti-inflammatory effects of Stephania tetrandra S. Moore on interleukin-6 production and experimental inflammatory disease models
title_fullStr Anti-inflammatory effects of Stephania tetrandra S. Moore on interleukin-6 production and experimental inflammatory disease models
title_full_unstemmed Anti-inflammatory effects of Stephania tetrandra S. Moore on interleukin-6 production and experimental inflammatory disease models
title_short Anti-inflammatory effects of Stephania tetrandra S. Moore on interleukin-6 production and experimental inflammatory disease models
title_sort anti inflammatory effects of stephania tetrandra s moore on interleukin 6 production and experimental inflammatory disease models
url http://dx.doi.org/10.1155/S0962935196000415
work_keys_str_mv AT hskang antiinflammatoryeffectsofstephaniatetrandrasmooreoninterleukin6productionandexperimentalinflammatorydiseasemodels
AT yhkim antiinflammatoryeffectsofstephaniatetrandrasmooreoninterleukin6productionandexperimentalinflammatorydiseasemodels
AT cslee antiinflammatoryeffectsofstephaniatetrandrasmooreoninterleukin6productionandexperimentalinflammatorydiseasemodels
AT jjlee antiinflammatoryeffectsofstephaniatetrandrasmooreoninterleukin6productionandexperimentalinflammatorydiseasemodels
AT ichoi antiinflammatoryeffectsofstephaniatetrandrasmooreoninterleukin6productionandexperimentalinflammatorydiseasemodels
AT khpyun antiinflammatoryeffectsofstephaniatetrandrasmooreoninterleukin6productionandexperimentalinflammatorydiseasemodels