Circulating intercellular adhesion molecules in blood and bronchoalveolar lavage in Behçet's disease

The aim of this study was to evaluate circulating intercellular adhesion molecule-1 (cICAM-1) in serum and in bronchoalveolar lavage (BAL), as a marker for the inflammatory process in patients with active Behçet's disease (BD). Circulating ICAM-1 was tested by an enzyme linked immuno-sorbent as...

Full description

Saved in:
Bibliographic Details
Main Authors: A. Hamzaoui, K. Hamzaoui, A. Chabbou, K. Ayed
Format: Article
Language:English
Published: Wiley 1995-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/S0962935195000573
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849413266100453376
author A. Hamzaoui
K. Hamzaoui
A. Chabbou
K. Ayed
author_facet A. Hamzaoui
K. Hamzaoui
A. Chabbou
K. Ayed
author_sort A. Hamzaoui
collection DOAJ
description The aim of this study was to evaluate circulating intercellular adhesion molecule-1 (cICAM-1) in serum and in bronchoalveolar lavage (BAL), as a marker for the inflammatory process in patients with active Behçet's disease (BD). Circulating ICAM-1 was tested by an enzyme linked immuno-sorbent assay in serum and in BAL of patients with BD. These values were compared to those of patients with tuberculosis and to healthy controls. Increased levels of circulating ICAM-1 were found in serum from patients with active BD compared to healthy controls (p < 0.01). Similar levels of serum cICAM-1 were found in BD and tuberculosis. Additionally, both BD and tuberculosis patients exhibited high levels of cICAM-1 in BAL fluid, suggesting that this increase may be a result of the immune system activation in inflammatory sites. Circulating ICAM-1 seemed to have a good discriminative power in identifying active BD, being elevated in all active stages (p < 0.01) compared to remission BD stage. No differences were found in active BD patients depending upon the clinical manifestations. These results suggest that cICAM-1 may be involved in leucocyte adhesion and migration into the vessel wall of the lung. Circulating forms are derived from molecules expressed on the surface of activated cells, as a result of an inflammatory process.
format Article
id doaj-art-6cc34784ef5a4fc29a3b04d3f0fffbfe
institution Kabale University
issn 0962-9351
1466-1861
language English
publishDate 1995-01-01
publisher Wiley
record_format Article
series Mediators of Inflammation
spelling doaj-art-6cc34784ef5a4fc29a3b04d3f0fffbfe2025-08-20T03:34:10ZengWileyMediators of Inflammation0962-93511466-18611995-01-014535535810.1155/S0962935195000573Circulating intercellular adhesion molecules in blood and bronchoalveolar lavage in Behçet's diseaseA. Hamzaoui0K. Hamzaoui1A. Chabbou2K. Ayed3Department of Respiratory Diseases, Faculté de Médecine de Tunis, 9 rue Z. Essafi, Tunis 1007, TunisiaDepartment of Histology, Faculté de Médecine de Tunis, 9 rue Z. Essafi, Tunis 1007, TunisiaDepartment of Respiratory Diseases, Faculté de Médecine de Tunis, 9 rue Z. Essafi, Tunis 1007, TunisiaDepartment of Immunology, Faculté de Médecine de Tunis, 9 rue Z. Essafi, Tunis 1007, TunisiaThe aim of this study was to evaluate circulating intercellular adhesion molecule-1 (cICAM-1) in serum and in bronchoalveolar lavage (BAL), as a marker for the inflammatory process in patients with active Behçet's disease (BD). Circulating ICAM-1 was tested by an enzyme linked immuno-sorbent assay in serum and in BAL of patients with BD. These values were compared to those of patients with tuberculosis and to healthy controls. Increased levels of circulating ICAM-1 were found in serum from patients with active BD compared to healthy controls (p < 0.01). Similar levels of serum cICAM-1 were found in BD and tuberculosis. Additionally, both BD and tuberculosis patients exhibited high levels of cICAM-1 in BAL fluid, suggesting that this increase may be a result of the immune system activation in inflammatory sites. Circulating ICAM-1 seemed to have a good discriminative power in identifying active BD, being elevated in all active stages (p < 0.01) compared to remission BD stage. No differences were found in active BD patients depending upon the clinical manifestations. These results suggest that cICAM-1 may be involved in leucocyte adhesion and migration into the vessel wall of the lung. Circulating forms are derived from molecules expressed on the surface of activated cells, as a result of an inflammatory process.http://dx.doi.org/10.1155/S0962935195000573
spellingShingle A. Hamzaoui
K. Hamzaoui
A. Chabbou
K. Ayed
Circulating intercellular adhesion molecules in blood and bronchoalveolar lavage in Behçet's disease
Mediators of Inflammation
title Circulating intercellular adhesion molecules in blood and bronchoalveolar lavage in Behçet's disease
title_full Circulating intercellular adhesion molecules in blood and bronchoalveolar lavage in Behçet's disease
title_fullStr Circulating intercellular adhesion molecules in blood and bronchoalveolar lavage in Behçet's disease
title_full_unstemmed Circulating intercellular adhesion molecules in blood and bronchoalveolar lavage in Behçet's disease
title_short Circulating intercellular adhesion molecules in blood and bronchoalveolar lavage in Behçet's disease
title_sort circulating intercellular adhesion molecules in blood and bronchoalveolar lavage in behcet s disease
url http://dx.doi.org/10.1155/S0962935195000573
work_keys_str_mv AT ahamzaoui circulatingintercellularadhesionmoleculesinbloodandbronchoalveolarlavageinbehcetsdisease
AT khamzaoui circulatingintercellularadhesionmoleculesinbloodandbronchoalveolarlavageinbehcetsdisease
AT achabbou circulatingintercellularadhesionmoleculesinbloodandbronchoalveolarlavageinbehcetsdisease
AT kayed circulatingintercellularadhesionmoleculesinbloodandbronchoalveolarlavageinbehcetsdisease