Unveiling BID: a key biomarker in apoptosis post-intracerebral hemorrhage

BackgroundApoptosis plays a significant role in secondary brain injury following intracerebral hemorrhage (ICH). Currently, the mechanisms related to cell apoptosis after cerebral hemorrhage are still under investigation.MethodsWe identified differentially expressed genes (DEGs) between human ICH pa...

Full description

Saved in:
Bibliographic Details
Main Authors: Kun Dai, Hong-Rong Zhang, Shuai-Yu Ren, Ming-Pei Zhao, Neng Wang, Hong-Zhi Gao, De-Zhi Kang, Zong-Qing Zheng
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-02-01
Series:Frontiers in Neurology
Subjects:
Online Access:https://www.frontiersin.org/articles/10.3389/fneur.2025.1533558/full
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850043829421342720
author Kun Dai
Hong-Rong Zhang
Shuai-Yu Ren
Ming-Pei Zhao
Ming-Pei Zhao
Neng Wang
Neng Wang
Hong-Zhi Gao
Hong-Zhi Gao
De-Zhi Kang
De-Zhi Kang
De-Zhi Kang
Zong-Qing Zheng
Zong-Qing Zheng
Zong-Qing Zheng
Zong-Qing Zheng
Zong-Qing Zheng
author_facet Kun Dai
Hong-Rong Zhang
Shuai-Yu Ren
Ming-Pei Zhao
Ming-Pei Zhao
Neng Wang
Neng Wang
Hong-Zhi Gao
Hong-Zhi Gao
De-Zhi Kang
De-Zhi Kang
De-Zhi Kang
Zong-Qing Zheng
Zong-Qing Zheng
Zong-Qing Zheng
Zong-Qing Zheng
Zong-Qing Zheng
author_sort Kun Dai
collection DOAJ
description BackgroundApoptosis plays a significant role in secondary brain injury following intracerebral hemorrhage (ICH). Currently, the mechanisms related to cell apoptosis after cerebral hemorrhage are still under investigation.MethodsWe identified differentially expressed genes (DEGs) between human ICH patients and normal individuals from the GEO database and conducted GO and KEGG functional enrichment analyses on these DEGs. We then constructed a PPI network and used the MECDE algorithm to identify key genes potentially involved in apoptosis after ICH. Additionally, we identified miRNAs that might regulate apoptotic genes in an mRNA-miRNA interaction network. Finally, we validated the bioinformatics results in a rat ICH model.ResultsIn the human ICH model, 645 DEGs were identified. GO and KEGG analyses indicated that these DEGs are primarily involved in immune response, inflammatory response, and apoptosis. GSEA analysis showed significant enrichment of DEGs in the apoptotic process. By comparing with apoptosis-related genes in the MSigDB database, we identified 110 apoptosis-related genes among the 645 DEGs. Further PPI and MOCDE analyses of these apoptosis-related genes revealed that BID might be a key gene involved in apoptosis after ICH, which was validated within the rat model of ICH. The mRNA-miRNA interactions network construction suggested that miR1225-3p may be an important miRNA involved in regulating BID expression after ICH.ConclusionBID plays a critical role in the regulation of apoptosis following intracerebral hemorrhage and serves as a key biomarker in the apoptotic process after hemorrhage.
format Article
id doaj-art-6c248eaf64e54ecd87f97c96fbb2c7de
institution DOAJ
issn 1664-2295
language English
publishDate 2025-02-01
publisher Frontiers Media S.A.
record_format Article
series Frontiers in Neurology
spelling doaj-art-6c248eaf64e54ecd87f97c96fbb2c7de2025-08-20T02:55:07ZengFrontiers Media S.A.Frontiers in Neurology1664-22952025-02-011610.3389/fneur.2025.15335581533558Unveiling BID: a key biomarker in apoptosis post-intracerebral hemorrhageKun Dai0Hong-Rong Zhang1Shuai-Yu Ren2Ming-Pei Zhao3Ming-Pei Zhao4Neng Wang5Neng Wang6Hong-Zhi Gao7Hong-Zhi Gao8De-Zhi Kang9De-Zhi Kang10De-Zhi Kang11Zong-Qing Zheng12Zong-Qing Zheng13Zong-Qing Zheng14Zong-Qing Zheng15Zong-Qing Zheng16Department of Neurosurgery, The First Affiliated Hospital of Soochow University, Suzhou, ChinaDepartment of Neurosurgery, The First Affiliated Hospital of Soochow University, Suzhou, ChinaDepartment of Neurosurgery, The First Affiliated Hospital of Soochow University, Suzhou, ChinaDepartment of Neurosurgery, Neurosurgery Research Institute, The First Affiliated Hospital, Fujian Medical University, Fuzhou, ChinaDepartment of Neurosurgery, National Regional Medical Center, Binhai Campus of the First Affiliated Hospital, Fujian Medical University, Fuzhou, ChinaDepartment of Neurosurgery, Neurosurgery Research Institute, The First Affiliated Hospital, Fujian Medical University, Fuzhou, ChinaDepartment of Neurosurgery, National Regional Medical Center, Binhai Campus of the First Affiliated Hospital, Fujian Medical University, Fuzhou, ChinaDepartment of Neurosurgery, The Second Affiliated Hospital of Fujian Medical University, Quanzhou, ChinaClinical Center for Molecular Diagnosis and Therapy, The Second Affiliated Hospital of Fujian Medical University, Quanzhou, ChinaDepartment of Neurosurgery, Neurosurgery Research Institute, The First Affiliated Hospital, Fujian Medical University, Fuzhou, ChinaDepartment of Neurosurgery, National Regional Medical Center, Binhai Campus of the First Affiliated Hospital, Fujian Medical University, Fuzhou, ChinaFujian Provincial Institutes of Brain Disorders and Brain Sciences, First Affiliated Hospital, Fujian Medical University, Fuzhou, ChinaDepartment of Neurosurgery, Neurosurgery Research Institute, The First Affiliated Hospital, Fujian Medical University, Fuzhou, ChinaDepartment of Neurosurgery, National Regional Medical Center, Binhai Campus of the First Affiliated Hospital, Fujian Medical University, Fuzhou, ChinaDepartment of Neurosurgery, The Second Affiliated Hospital of Fujian Medical University, Quanzhou, ChinaClinical Center for Molecular Diagnosis and Therapy, The Second Affiliated Hospital of Fujian Medical University, Quanzhou, ChinaFujian Provincial Institutes of Brain Disorders and Brain Sciences, First Affiliated Hospital, Fujian Medical University, Fuzhou, ChinaBackgroundApoptosis plays a significant role in secondary brain injury following intracerebral hemorrhage (ICH). Currently, the mechanisms related to cell apoptosis after cerebral hemorrhage are still under investigation.MethodsWe identified differentially expressed genes (DEGs) between human ICH patients and normal individuals from the GEO database and conducted GO and KEGG functional enrichment analyses on these DEGs. We then constructed a PPI network and used the MECDE algorithm to identify key genes potentially involved in apoptosis after ICH. Additionally, we identified miRNAs that might regulate apoptotic genes in an mRNA-miRNA interaction network. Finally, we validated the bioinformatics results in a rat ICH model.ResultsIn the human ICH model, 645 DEGs were identified. GO and KEGG analyses indicated that these DEGs are primarily involved in immune response, inflammatory response, and apoptosis. GSEA analysis showed significant enrichment of DEGs in the apoptotic process. By comparing with apoptosis-related genes in the MSigDB database, we identified 110 apoptosis-related genes among the 645 DEGs. Further PPI and MOCDE analyses of these apoptosis-related genes revealed that BID might be a key gene involved in apoptosis after ICH, which was validated within the rat model of ICH. The mRNA-miRNA interactions network construction suggested that miR1225-3p may be an important miRNA involved in regulating BID expression after ICH.ConclusionBID plays a critical role in the regulation of apoptosis following intracerebral hemorrhage and serves as a key biomarker in the apoptotic process after hemorrhage.https://www.frontiersin.org/articles/10.3389/fneur.2025.1533558/fullICHapoptosisbioinformatics analysismicroRNABID
spellingShingle Kun Dai
Hong-Rong Zhang
Shuai-Yu Ren
Ming-Pei Zhao
Ming-Pei Zhao
Neng Wang
Neng Wang
Hong-Zhi Gao
Hong-Zhi Gao
De-Zhi Kang
De-Zhi Kang
De-Zhi Kang
Zong-Qing Zheng
Zong-Qing Zheng
Zong-Qing Zheng
Zong-Qing Zheng
Zong-Qing Zheng
Unveiling BID: a key biomarker in apoptosis post-intracerebral hemorrhage
Frontiers in Neurology
ICH
apoptosis
bioinformatics analysis
microRNA
BID
title Unveiling BID: a key biomarker in apoptosis post-intracerebral hemorrhage
title_full Unveiling BID: a key biomarker in apoptosis post-intracerebral hemorrhage
title_fullStr Unveiling BID: a key biomarker in apoptosis post-intracerebral hemorrhage
title_full_unstemmed Unveiling BID: a key biomarker in apoptosis post-intracerebral hemorrhage
title_short Unveiling BID: a key biomarker in apoptosis post-intracerebral hemorrhage
title_sort unveiling bid a key biomarker in apoptosis post intracerebral hemorrhage
topic ICH
apoptosis
bioinformatics analysis
microRNA
BID
url https://www.frontiersin.org/articles/10.3389/fneur.2025.1533558/full
work_keys_str_mv AT kundai unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT hongrongzhang unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT shuaiyuren unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT mingpeizhao unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT mingpeizhao unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT nengwang unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT nengwang unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT hongzhigao unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT hongzhigao unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT dezhikang unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT dezhikang unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT dezhikang unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT zongqingzheng unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT zongqingzheng unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT zongqingzheng unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT zongqingzheng unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage
AT zongqingzheng unveilingbidakeybiomarkerinapoptosispostintracerebralhemorrhage