The peculiarities of eosinophil apoptosis damage in patients with severe therapy-resistant bronchial asthma

The study aim: to investigate the dynamics of transcription factor p53, proapoptoic (bax, bcl-XS), antiapoptotic (bcl-2, bcl-XL) effectors, IL-5 expression in peripheral blood eosinophils of patients with severe therapy-resistant bronchial asthma (BA). Methods: clinical, functional, allergologic obs...

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Main Authors: L. Yu. Nikitina, F. I. Petrovsky, I. I. Ivanchuk, A. E. Sazonov
Format: Article
Language:English
Published: Siberian State Medical University (Tomsk) 2005-12-01
Series:Бюллетень сибирской медицины
Subjects:
Online Access:https://bulletin.ssmu.ru/jour/article/view/3697
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author L. Yu. Nikitina
F. I. Petrovsky
I. I. Ivanchuk
A. E. Sazonov
author_facet L. Yu. Nikitina
F. I. Petrovsky
I. I. Ivanchuk
A. E. Sazonov
author_sort L. Yu. Nikitina
collection DOAJ
description The study aim: to investigate the dynamics of transcription factor p53, proapoptoic (bax, bcl-XS), antiapoptotic (bcl-2, bcl-XL) effectors, IL-5 expression in peripheral blood eosinophils of patients with severe therapy-resistant bronchial asthma (BA). Methods: clinical, functional, allergologic observation, the study of apoptosis effectors expression (Polymerase Chain Reaction). Material: 60 patients with severe BA. Results: low p53 expression and eosinophils apoptosis depression were revealed in therapy-resistant asthmatics. Conclusion: eosinophil apoptosis disregulation – the key mechanism of persistent inflammation of the airways in therapy-resistant BA.
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publisher Siberian State Medical University (Tomsk)
record_format Article
series Бюллетень сибирской медицины
spelling doaj-art-69faad5b1bf94635a26da2f5ec88cdd32025-08-20T03:01:54ZengSiberian State Medical University (Tomsk)Бюллетень сибирской медицины1682-03631819-36842005-12-0144647010.20538/1682-0363-2005-4-64-702336The peculiarities of eosinophil apoptosis damage in patients with severe therapy-resistant bronchial asthmaL. Yu. Nikitina0F. I. Petrovsky1I. I. Ivanchuk2A. E. Sazonov3Сибирский государственный медицинский университетСибирский государственный медицинский университетСибирский государственный медицинский университетСибирский государственный медицинский университетThe study aim: to investigate the dynamics of transcription factor p53, proapoptoic (bax, bcl-XS), antiapoptotic (bcl-2, bcl-XL) effectors, IL-5 expression in peripheral blood eosinophils of patients with severe therapy-resistant bronchial asthma (BA). Methods: clinical, functional, allergologic observation, the study of apoptosis effectors expression (Polymerase Chain Reaction). Material: 60 patients with severe BA. Results: low p53 expression and eosinophils apoptosis depression were revealed in therapy-resistant asthmatics. Conclusion: eosinophil apoptosis disregulation – the key mechanism of persistent inflammation of the airways in therapy-resistant BA.https://bulletin.ssmu.ru/jour/article/view/3697therapy-resistant severe asthmaeosinophilapoptosisp53bcl-2bax
spellingShingle L. Yu. Nikitina
F. I. Petrovsky
I. I. Ivanchuk
A. E. Sazonov
The peculiarities of eosinophil apoptosis damage in patients with severe therapy-resistant bronchial asthma
Бюллетень сибирской медицины
therapy-resistant severe asthma
eosinophil
apoptosis
p53
bcl-2
bax
title The peculiarities of eosinophil apoptosis damage in patients with severe therapy-resistant bronchial asthma
title_full The peculiarities of eosinophil apoptosis damage in patients with severe therapy-resistant bronchial asthma
title_fullStr The peculiarities of eosinophil apoptosis damage in patients with severe therapy-resistant bronchial asthma
title_full_unstemmed The peculiarities of eosinophil apoptosis damage in patients with severe therapy-resistant bronchial asthma
title_short The peculiarities of eosinophil apoptosis damage in patients with severe therapy-resistant bronchial asthma
title_sort peculiarities of eosinophil apoptosis damage in patients with severe therapy resistant bronchial asthma
topic therapy-resistant severe asthma
eosinophil
apoptosis
p53
bcl-2
bax
url https://bulletin.ssmu.ru/jour/article/view/3697
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