Proanthocyanidins delaying the premature ovarian insufficiency through regulatory sirt1-p53-p21 signaling pathway in female germline stem cells

Abstract Background As women age, their ovarian follicle pool naturally declines. However, female germline stem cells (FGSCs) possess a unique ability to differentiate into oocytes and continuously self-renew, providing an effective means of delaying ovarian aging by replenishing the primordial foll...

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Main Authors: Wenbo Wu, Mengying Bai, Wenli Hong, Shuyi Ling, Yuan Li, Yuqing Dai, Ruoxin Weng, Haifeng Wu, Chongyang Ren, Liujuan Zhang, Ziqiong Zhou, Zhisheng Zhong, Xinxin Fu, Yuehui Zheng
Format: Article
Language:English
Published: BMC 2025-05-01
Series:Journal of Ovarian Research
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Online Access:https://doi.org/10.1186/s13048-025-01661-y
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author Wenbo Wu
Mengying Bai
Wenli Hong
Shuyi Ling
Yuan Li
Yuqing Dai
Ruoxin Weng
Haifeng Wu
Chongyang Ren
Liujuan Zhang
Ziqiong Zhou
Zhisheng Zhong
Xinxin Fu
Yuehui Zheng
author_facet Wenbo Wu
Mengying Bai
Wenli Hong
Shuyi Ling
Yuan Li
Yuqing Dai
Ruoxin Weng
Haifeng Wu
Chongyang Ren
Liujuan Zhang
Ziqiong Zhou
Zhisheng Zhong
Xinxin Fu
Yuehui Zheng
author_sort Wenbo Wu
collection DOAJ
description Abstract Background As women age, their ovarian follicle pool naturally declines. However, female germline stem cells (FGSCs) possess a unique ability to differentiate into oocytes and continuously self-renew, providing an effective means of delaying ovarian aging by replenishing the primordial follicle pool. Therefore, activating FGSCs is critical in reshaping and safeguarding ovarian function. Methods In this study, we investigated the biological activity of proanthocyanidins (PACs), natural antioxidants that exhibit anti-aging and anti-inflammatory properties beneficial for both male and female reproduction. Our in vivo and in vitro experiments demonstrate that PACs promote FGSCs proliferation while delaying ovarian aging. Results PACs increase the number of primordial follicles, primary follicles, corpus luteum while reducing cystic follicles, and elevate estradiol (E2) levels along with anti-mullerian hormone (AMH) concentration levels in mice. Additionally, PACs significantly boost FGSCs proliferation time- and dose-dependently by upregulating mRNA & protein expressions for FGSCs-specific markers such as MVH and OCT4 while downregulating p53/p21 via activation of silent information regulator 1(Sirt1) signaling pathway. The effects of PACs on FGCSs were found to be impeded by the Sirt1 inhibitor EX527. Conclusion PACS delay premature ovarian insufficiency (POI) through regulating the Sirt1-p53-p21 signaling pathway involving FGSCs.
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record_format Article
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spelling doaj-art-69f60b9fa5474fbea708bcadcc6645662025-08-20T03:53:13ZengBMCJournal of Ovarian Research1757-22152025-05-0118111210.1186/s13048-025-01661-yProanthocyanidins delaying the premature ovarian insufficiency through regulatory sirt1-p53-p21 signaling pathway in female germline stem cellsWenbo Wu0Mengying Bai1Wenli Hong2Shuyi Ling3Yuan Li4Yuqing Dai5Ruoxin Weng6Haifeng Wu7Chongyang Ren8Liujuan Zhang9Ziqiong Zhou10Zhisheng Zhong11Xinxin Fu12Yuehui Zheng13The Fourth Clinical Medical College of Guangzhou University of Traditional Chinese Medicine, Shenzhen Traditional Chinese Medicine HospitalThe Fourth Clinical Medical College of Guangzhou University of Traditional Chinese Medicine, Shenzhen Traditional Chinese Medicine HospitalThe Fourth Clinical Medical College of Guangzhou University of Traditional Chinese Medicine, Shenzhen Traditional Chinese Medicine HospitalThe Fourth Clinical Medical College of Guangzhou University of Traditional Chinese Medicine, Shenzhen Traditional Chinese Medicine HospitalThe Fourth Clinical Medical College of Guangzhou University of Traditional Chinese Medicine, Shenzhen Traditional Chinese Medicine HospitalThe Fourth Clinical Medical College of Guangzhou University of Traditional Chinese Medicine, Shenzhen Traditional Chinese Medicine HospitalThe Fourth Clinical Medical College of Guangzhou University of Traditional Chinese Medicine, Shenzhen Traditional Chinese Medicine HospitalThe Fourth Clinical Medical College of Guangzhou University of Traditional Chinese Medicine, Shenzhen Traditional Chinese Medicine HospitalThe Fourth Clinical Medical College of Guangzhou University of Traditional Chinese Medicine, Shenzhen Traditional Chinese Medicine HospitalThe Fourth Clinical Medical College of Guangzhou University of Traditional Chinese Medicine, Shenzhen Traditional Chinese Medicine HospitalThe Fourth Clinical Medical College of Guangzhou University of Traditional Chinese Medicine, Shenzhen Traditional Chinese Medicine HospitalThe Fourth Clinical Medical College of Guangzhou University of Traditional Chinese Medicine, Shenzhen Traditional Chinese Medicine HospitalDepartment of National Demonstration Center for Clinical Teaching &Training, School of Medicine, Xiang’an Hospital of Xiamen University, Xiamen UniversityThe Fourth Clinical Medical College of Guangzhou University of Traditional Chinese Medicine, Shenzhen Traditional Chinese Medicine HospitalAbstract Background As women age, their ovarian follicle pool naturally declines. However, female germline stem cells (FGSCs) possess a unique ability to differentiate into oocytes and continuously self-renew, providing an effective means of delaying ovarian aging by replenishing the primordial follicle pool. Therefore, activating FGSCs is critical in reshaping and safeguarding ovarian function. Methods In this study, we investigated the biological activity of proanthocyanidins (PACs), natural antioxidants that exhibit anti-aging and anti-inflammatory properties beneficial for both male and female reproduction. Our in vivo and in vitro experiments demonstrate that PACs promote FGSCs proliferation while delaying ovarian aging. Results PACs increase the number of primordial follicles, primary follicles, corpus luteum while reducing cystic follicles, and elevate estradiol (E2) levels along with anti-mullerian hormone (AMH) concentration levels in mice. Additionally, PACs significantly boost FGSCs proliferation time- and dose-dependently by upregulating mRNA & protein expressions for FGSCs-specific markers such as MVH and OCT4 while downregulating p53/p21 via activation of silent information regulator 1(Sirt1) signaling pathway. The effects of PACs on FGCSs were found to be impeded by the Sirt1 inhibitor EX527. Conclusion PACS delay premature ovarian insufficiency (POI) through regulating the Sirt1-p53-p21 signaling pathway involving FGSCs.https://doi.org/10.1186/s13048-025-01661-yProanthocyanidinsFemale germline stem cellsSilent information regulator 1Premature ovarian insufficiencyMice
spellingShingle Wenbo Wu
Mengying Bai
Wenli Hong
Shuyi Ling
Yuan Li
Yuqing Dai
Ruoxin Weng
Haifeng Wu
Chongyang Ren
Liujuan Zhang
Ziqiong Zhou
Zhisheng Zhong
Xinxin Fu
Yuehui Zheng
Proanthocyanidins delaying the premature ovarian insufficiency through regulatory sirt1-p53-p21 signaling pathway in female germline stem cells
Journal of Ovarian Research
Proanthocyanidins
Female germline stem cells
Silent information regulator 1
Premature ovarian insufficiency
Mice
title Proanthocyanidins delaying the premature ovarian insufficiency through regulatory sirt1-p53-p21 signaling pathway in female germline stem cells
title_full Proanthocyanidins delaying the premature ovarian insufficiency through regulatory sirt1-p53-p21 signaling pathway in female germline stem cells
title_fullStr Proanthocyanidins delaying the premature ovarian insufficiency through regulatory sirt1-p53-p21 signaling pathway in female germline stem cells
title_full_unstemmed Proanthocyanidins delaying the premature ovarian insufficiency through regulatory sirt1-p53-p21 signaling pathway in female germline stem cells
title_short Proanthocyanidins delaying the premature ovarian insufficiency through regulatory sirt1-p53-p21 signaling pathway in female germline stem cells
title_sort proanthocyanidins delaying the premature ovarian insufficiency through regulatory sirt1 p53 p21 signaling pathway in female germline stem cells
topic Proanthocyanidins
Female germline stem cells
Silent information regulator 1
Premature ovarian insufficiency
Mice
url https://doi.org/10.1186/s13048-025-01661-y
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