Alterations of red cell membrane properties in neuroacanthocytosis.

Neuroacanthocytosis (NA) refers to a group of heterogenous, rare genetic disorders, namely chorea acanthocytosis (ChAc), McLeod syndrome (MLS), Huntington's disease-like 2 (HDL2) and pantothenate kinase associated neurodegeneration (PKAN), that mainly affect the basal ganglia and are associated...

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Main Authors: Claudia Siegl, Patricia Hamminger, Herbert Jank, Uwe Ahting, Benedikt Bader, Adrian Danek, Allison Gregory, Monika Hartig, Susan Hayflick, Andreas Hermann, Holger Prokisch, Esther M Sammler, Zuhal Yapici, Rainer Prohaska, Ulrich Salzer
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2013-01-01
Series:PLoS ONE
Online Access:https://doi.org/10.1371/journal.pone.0076715
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author Claudia Siegl
Patricia Hamminger
Herbert Jank
Uwe Ahting
Benedikt Bader
Adrian Danek
Allison Gregory
Monika Hartig
Susan Hayflick
Andreas Hermann
Holger Prokisch
Esther M Sammler
Zuhal Yapici
Rainer Prohaska
Ulrich Salzer
author_facet Claudia Siegl
Patricia Hamminger
Herbert Jank
Uwe Ahting
Benedikt Bader
Adrian Danek
Allison Gregory
Monika Hartig
Susan Hayflick
Andreas Hermann
Holger Prokisch
Esther M Sammler
Zuhal Yapici
Rainer Prohaska
Ulrich Salzer
author_sort Claudia Siegl
collection DOAJ
description Neuroacanthocytosis (NA) refers to a group of heterogenous, rare genetic disorders, namely chorea acanthocytosis (ChAc), McLeod syndrome (MLS), Huntington's disease-like 2 (HDL2) and pantothenate kinase associated neurodegeneration (PKAN), that mainly affect the basal ganglia and are associated with similar neurological symptoms. PKAN is also assigned to a group of rare neurodegenerative diseases, known as NBIA (neurodegeneration with brain iron accumulation), associated with iron accumulation in the basal ganglia and progressive movement disorder. Acanthocytosis, the occurrence of misshaped erythrocytes with thorny protrusions, is frequently observed in ChAc and MLS patients but less prevalent in PKAN (about 10%) and HDL2 patients. The pathological factors that lead to the formation of the acanthocytic red blood cell shape are currently unknown. The aim of this study was to determine whether NA/NBIA acanthocytes differ in their functionality from normal erythrocytes. Several flow-cytometry-based assays were applied to test the physiological responses of the plasma membrane, namely drug-induced endocytosis, phosphatidylserine exposure and calcium uptake upon treatment with lysophosphatidic acid. ChAc red cell samples clearly showed a reduced response in drug-induced endovesiculation, lysophosphatidic acid-induced phosphatidylserine exposure, and calcium uptake. Impaired responses were also observed in acanthocyte-positive NBIA (PKAN) red cells but not in patient cells without shape abnormalities. These data suggest an "acanthocytic state" of the red cell where alterations in functional and interdependent membrane properties arise together with an acanthocytic cell shape. Further elucidation of the aberrant molecular mechanisms that cause this acanthocytic state may possibly help to evaluate the pathological pathways leading to neurodegeneration.
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spelling doaj-art-6943ff913f5e491d87cd38f275fec6142025-08-20T03:46:24ZengPublic Library of Science (PLoS)PLoS ONE1932-62032013-01-01810e7671510.1371/journal.pone.0076715Alterations of red cell membrane properties in neuroacanthocytosis.Claudia SieglPatricia HammingerHerbert JankUwe AhtingBenedikt BaderAdrian DanekAllison GregoryMonika HartigSusan HayflickAndreas HermannHolger ProkischEsther M SammlerZuhal YapiciRainer ProhaskaUlrich SalzerNeuroacanthocytosis (NA) refers to a group of heterogenous, rare genetic disorders, namely chorea acanthocytosis (ChAc), McLeod syndrome (MLS), Huntington's disease-like 2 (HDL2) and pantothenate kinase associated neurodegeneration (PKAN), that mainly affect the basal ganglia and are associated with similar neurological symptoms. PKAN is also assigned to a group of rare neurodegenerative diseases, known as NBIA (neurodegeneration with brain iron accumulation), associated with iron accumulation in the basal ganglia and progressive movement disorder. Acanthocytosis, the occurrence of misshaped erythrocytes with thorny protrusions, is frequently observed in ChAc and MLS patients but less prevalent in PKAN (about 10%) and HDL2 patients. The pathological factors that lead to the formation of the acanthocytic red blood cell shape are currently unknown. The aim of this study was to determine whether NA/NBIA acanthocytes differ in their functionality from normal erythrocytes. Several flow-cytometry-based assays were applied to test the physiological responses of the plasma membrane, namely drug-induced endocytosis, phosphatidylserine exposure and calcium uptake upon treatment with lysophosphatidic acid. ChAc red cell samples clearly showed a reduced response in drug-induced endovesiculation, lysophosphatidic acid-induced phosphatidylserine exposure, and calcium uptake. Impaired responses were also observed in acanthocyte-positive NBIA (PKAN) red cells but not in patient cells without shape abnormalities. These data suggest an "acanthocytic state" of the red cell where alterations in functional and interdependent membrane properties arise together with an acanthocytic cell shape. Further elucidation of the aberrant molecular mechanisms that cause this acanthocytic state may possibly help to evaluate the pathological pathways leading to neurodegeneration.https://doi.org/10.1371/journal.pone.0076715
spellingShingle Claudia Siegl
Patricia Hamminger
Herbert Jank
Uwe Ahting
Benedikt Bader
Adrian Danek
Allison Gregory
Monika Hartig
Susan Hayflick
Andreas Hermann
Holger Prokisch
Esther M Sammler
Zuhal Yapici
Rainer Prohaska
Ulrich Salzer
Alterations of red cell membrane properties in neuroacanthocytosis.
PLoS ONE
title Alterations of red cell membrane properties in neuroacanthocytosis.
title_full Alterations of red cell membrane properties in neuroacanthocytosis.
title_fullStr Alterations of red cell membrane properties in neuroacanthocytosis.
title_full_unstemmed Alterations of red cell membrane properties in neuroacanthocytosis.
title_short Alterations of red cell membrane properties in neuroacanthocytosis.
title_sort alterations of red cell membrane properties in neuroacanthocytosis
url https://doi.org/10.1371/journal.pone.0076715
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