Recombinant characterization and pathogenicity of a novel L1C RFLP-1-4-4 variant of porcine reproductive and respiratory syndrome virus in China

Abstract Porcine reproductive and respiratory syndrome (PRRS) is one of the most significant diseases affecting the pig industry worldwide and is caused by the PRRS virus (PRRSV), which has complex genetic variation due to frequent mutations, indels, and recombination. The emergence of PRRSV L1C.5 i...

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Main Authors: Xinyi Huang, Guoqing Liu, Tong Chang, Yongbo Yang, Tao Wang, Dasong Xia, Xinyu Qi, Xulong Zhu, Ziyi Wei, Xiaoxiao Tian, Haiwei Wang, Zhijun Tian, Xuehui Cai, Tongqing An
Format: Article
Language:English
Published: BMC 2024-11-01
Series:Veterinary Research
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Online Access:https://doi.org/10.1186/s13567-024-01401-y
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author Xinyi Huang
Guoqing Liu
Tong Chang
Yongbo Yang
Tao Wang
Dasong Xia
Xinyu Qi
Xulong Zhu
Ziyi Wei
Xiaoxiao Tian
Haiwei Wang
Zhijun Tian
Xuehui Cai
Tongqing An
author_facet Xinyi Huang
Guoqing Liu
Tong Chang
Yongbo Yang
Tao Wang
Dasong Xia
Xinyu Qi
Xulong Zhu
Ziyi Wei
Xiaoxiao Tian
Haiwei Wang
Zhijun Tian
Xuehui Cai
Tongqing An
author_sort Xinyi Huang
collection DOAJ
description Abstract Porcine reproductive and respiratory syndrome (PRRS) is one of the most significant diseases affecting the pig industry worldwide and is caused by the PRRS virus (PRRSV), which has complex genetic variation due to frequent mutations, indels, and recombination. The emergence of PRRSV L1C.5 in 2020 in the United States has raised worldwide concerns about PRRSV with the RFLP 1-4-4 pattern and lineage 1C. However, studies on the pathogenic characteristics, epidemiological distribution, and effectiveness of vaccines against PRRSV with L1C and RFLP1-4-4 pattern in China are still insufficient. In this study, a novel recombinant variant of PRRSV with RFLP 1-4-4 and lineage 1C features, different from L1C.5 in the United States, was isolated in China in 2021. In pathogenicity experiments in specific pathogen-free piglets or farm piglets, 60–100% of artificially infected experimental piglets died with high fever and respiratory symptoms. Inflammatory cytokine and chemokine levels were upregulated in infected piglets. A commercially modified live vaccine against highly pathogenic PRRSV did not provide effective protection when the vaccinated piglets were challenged with the novel L1C-1-4-4 variant. Therefore, this strain merits special attention when devising control and vaccine strategies. These findings suggest that extensive joint surveillance is urgently needed and that vaccine strategies should be updated to prevent the disease from spreading further.
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spelling doaj-art-6932ca2f819c4c1bbab00cc4b0b2f9152025-08-20T02:50:04ZengBMCVeterinary Research1297-97162024-11-0155111410.1186/s13567-024-01401-yRecombinant characterization and pathogenicity of a novel L1C RFLP-1-4-4 variant of porcine reproductive and respiratory syndrome virus in ChinaXinyi Huang0Guoqing Liu1Tong Chang2Yongbo Yang3Tao Wang4Dasong Xia5Xinyu Qi6Xulong Zhu7Ziyi Wei8Xiaoxiao Tian9Haiwei Wang10Zhijun Tian11Xuehui Cai12Tongqing An13State Key Laboratory for Animal Disease Control and Prevention, Harbin Veterinary Research Institute, Chinese Academy of Agricultural SciencesState Key Laboratory for Animal Disease Control and Prevention, Harbin Veterinary Research Institute, Chinese Academy of Agricultural SciencesState Key Laboratory for Animal Disease Control and Prevention, Harbin Veterinary Research Institute, Chinese Academy of Agricultural SciencesState Key Laboratory for Animal Disease Control and Prevention, Harbin Veterinary Research Institute, Chinese Academy of Agricultural SciencesState Key Laboratory for Animal Disease Control and Prevention, Harbin Veterinary Research Institute, Chinese Academy of Agricultural SciencesState Key Laboratory for Animal Disease Control and Prevention, Harbin Veterinary Research Institute, Chinese Academy of Agricultural SciencesState Key Laboratory for Animal Disease Control and Prevention, Harbin Veterinary Research Institute, Chinese Academy of Agricultural SciencesState Key Laboratory for Animal Disease Control and Prevention, Harbin Veterinary Research Institute, Chinese Academy of Agricultural SciencesState Key Laboratory for Animal Disease Control and Prevention, Harbin Veterinary Research Institute, Chinese Academy of Agricultural SciencesState Key Laboratory for Animal Disease Control and Prevention, Harbin Veterinary Research Institute, Chinese Academy of Agricultural SciencesState Key Laboratory for Animal Disease Control and Prevention, Harbin Veterinary Research Institute, Chinese Academy of Agricultural SciencesState Key Laboratory for Animal Disease Control and Prevention, Harbin Veterinary Research Institute, Chinese Academy of Agricultural SciencesState Key Laboratory for Animal Disease Control and Prevention, Harbin Veterinary Research Institute, Chinese Academy of Agricultural SciencesState Key Laboratory for Animal Disease Control and Prevention, Harbin Veterinary Research Institute, Chinese Academy of Agricultural SciencesAbstract Porcine reproductive and respiratory syndrome (PRRS) is one of the most significant diseases affecting the pig industry worldwide and is caused by the PRRS virus (PRRSV), which has complex genetic variation due to frequent mutations, indels, and recombination. The emergence of PRRSV L1C.5 in 2020 in the United States has raised worldwide concerns about PRRSV with the RFLP 1-4-4 pattern and lineage 1C. However, studies on the pathogenic characteristics, epidemiological distribution, and effectiveness of vaccines against PRRSV with L1C and RFLP1-4-4 pattern in China are still insufficient. In this study, a novel recombinant variant of PRRSV with RFLP 1-4-4 and lineage 1C features, different from L1C.5 in the United States, was isolated in China in 2021. In pathogenicity experiments in specific pathogen-free piglets or farm piglets, 60–100% of artificially infected experimental piglets died with high fever and respiratory symptoms. Inflammatory cytokine and chemokine levels were upregulated in infected piglets. A commercially modified live vaccine against highly pathogenic PRRSV did not provide effective protection when the vaccinated piglets were challenged with the novel L1C-1-4-4 variant. Therefore, this strain merits special attention when devising control and vaccine strategies. These findings suggest that extensive joint surveillance is urgently needed and that vaccine strategies should be updated to prevent the disease from spreading further.https://doi.org/10.1186/s13567-024-01401-yPorcine reproductive and respiratory syndrome virusL1C-1-4-4 variantrecombinationpathogenicitycross-protection
spellingShingle Xinyi Huang
Guoqing Liu
Tong Chang
Yongbo Yang
Tao Wang
Dasong Xia
Xinyu Qi
Xulong Zhu
Ziyi Wei
Xiaoxiao Tian
Haiwei Wang
Zhijun Tian
Xuehui Cai
Tongqing An
Recombinant characterization and pathogenicity of a novel L1C RFLP-1-4-4 variant of porcine reproductive and respiratory syndrome virus in China
Veterinary Research
Porcine reproductive and respiratory syndrome virus
L1C-1-4-4 variant
recombination
pathogenicity
cross-protection
title Recombinant characterization and pathogenicity of a novel L1C RFLP-1-4-4 variant of porcine reproductive and respiratory syndrome virus in China
title_full Recombinant characterization and pathogenicity of a novel L1C RFLP-1-4-4 variant of porcine reproductive and respiratory syndrome virus in China
title_fullStr Recombinant characterization and pathogenicity of a novel L1C RFLP-1-4-4 variant of porcine reproductive and respiratory syndrome virus in China
title_full_unstemmed Recombinant characterization and pathogenicity of a novel L1C RFLP-1-4-4 variant of porcine reproductive and respiratory syndrome virus in China
title_short Recombinant characterization and pathogenicity of a novel L1C RFLP-1-4-4 variant of porcine reproductive and respiratory syndrome virus in China
title_sort recombinant characterization and pathogenicity of a novel l1c rflp 1 4 4 variant of porcine reproductive and respiratory syndrome virus in china
topic Porcine reproductive and respiratory syndrome virus
L1C-1-4-4 variant
recombination
pathogenicity
cross-protection
url https://doi.org/10.1186/s13567-024-01401-y
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