The Treg/Th17 Paradigm in Lung Cancer

Pathogenic mechanisms underlying the development of lung cancer are very complex and not yet entirely clarified. T lymphocytes and their immune-regulatory cytokines play a pivotal role in controlling tumor growth and metastasis. Following activation by unique cytokines, CD4+ T helper cells different...

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Main Authors: Min-Chao Duan, Xiao-Ning Zhong, Guang-Nan Liu, Jin-Ru Wei
Format: Article
Language:English
Published: Wiley 2014-01-01
Series:Journal of Immunology Research
Online Access:http://dx.doi.org/10.1155/2014/730380
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author Min-Chao Duan
Xiao-Ning Zhong
Guang-Nan Liu
Jin-Ru Wei
author_facet Min-Chao Duan
Xiao-Ning Zhong
Guang-Nan Liu
Jin-Ru Wei
author_sort Min-Chao Duan
collection DOAJ
description Pathogenic mechanisms underlying the development of lung cancer are very complex and not yet entirely clarified. T lymphocytes and their immune-regulatory cytokines play a pivotal role in controlling tumor growth and metastasis. Following activation by unique cytokines, CD4+ T helper cells differentiate into Th1, Th2, Th17, and regulatory T cells (Tregs). Traditionally, research in lung cancer immunity has focused almost exclusively on Th1/Th2 cell balance. Recently, Th17 cells and Tregs represent an intriguing issue to be addressed in lung cancer pathogenesis. Tregs play an important role in the preservation of self-tolerance and modulation of overall immune responses against tumor cells. Th17 cells directly or via other proinflammatory cytokines modulate antitumor immune responses. Notably, there is a close relation between Tregs and Th17 cells. However, the possible interaction between these subsets in lung cancer remains to be elucidated. In this setting, targeting Treg/Th17 balance for therapeutic purposes may represent a useful tool for lung cancer treatment in the future. The purpose of this review is to discuss recent findings of the role of these novel populations in lung cancer immunity and to highlight the pleiotropic effects of these subsets on the development and regulation of lung cancer.
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spelling doaj-art-6912f9d48cac4ba0b2e46dfc8329b3152025-08-20T02:35:24ZengWileyJournal of Immunology Research2314-88612314-71562014-01-01201410.1155/2014/730380730380The Treg/Th17 Paradigm in Lung CancerMin-Chao Duan0Xiao-Ning Zhong1Guang-Nan Liu2Jin-Ru Wei3Department of Respiratory Medicine, The Eighth People’s Hospital of Nanning, Nanning, Guangxi 530001, ChinaDepartment of Respiratory Medicine, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi 530021, ChinaDepartment of Respiratory Medicine, The First Affiliated Hospital of Guangxi Medical University, Nanning, Guangxi 530021, ChinaDepartment of Respiratory Medicine, The First People’s Hospital of Nanning, Nanning, Guangxi 530022, ChinaPathogenic mechanisms underlying the development of lung cancer are very complex and not yet entirely clarified. T lymphocytes and their immune-regulatory cytokines play a pivotal role in controlling tumor growth and metastasis. Following activation by unique cytokines, CD4+ T helper cells differentiate into Th1, Th2, Th17, and regulatory T cells (Tregs). Traditionally, research in lung cancer immunity has focused almost exclusively on Th1/Th2 cell balance. Recently, Th17 cells and Tregs represent an intriguing issue to be addressed in lung cancer pathogenesis. Tregs play an important role in the preservation of self-tolerance and modulation of overall immune responses against tumor cells. Th17 cells directly or via other proinflammatory cytokines modulate antitumor immune responses. Notably, there is a close relation between Tregs and Th17 cells. However, the possible interaction between these subsets in lung cancer remains to be elucidated. In this setting, targeting Treg/Th17 balance for therapeutic purposes may represent a useful tool for lung cancer treatment in the future. The purpose of this review is to discuss recent findings of the role of these novel populations in lung cancer immunity and to highlight the pleiotropic effects of these subsets on the development and regulation of lung cancer.http://dx.doi.org/10.1155/2014/730380
spellingShingle Min-Chao Duan
Xiao-Ning Zhong
Guang-Nan Liu
Jin-Ru Wei
The Treg/Th17 Paradigm in Lung Cancer
Journal of Immunology Research
title The Treg/Th17 Paradigm in Lung Cancer
title_full The Treg/Th17 Paradigm in Lung Cancer
title_fullStr The Treg/Th17 Paradigm in Lung Cancer
title_full_unstemmed The Treg/Th17 Paradigm in Lung Cancer
title_short The Treg/Th17 Paradigm in Lung Cancer
title_sort treg th17 paradigm in lung cancer
url http://dx.doi.org/10.1155/2014/730380
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