FLZ attenuates Parkinson's disease pathological damage by increasing glycoursodeoxycholic acid production via down-regulating Clostridium innocuum

Increasing evidence shows that the early lesions of Parkinson's disease (PD) originate from gut, and correction of microbiota dysbiosis is a promising therapy for PD. FLZ is a neuroprotective agent on PD, which has been validated capable of alleviating microbiota dysbiosis in PD mice. However,...

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Main Authors: Meiyu Shang, Jingwen Ning, Caixia Zang, Jingwei Ma, Yang Yang, Yueqi Jiang, Qiuzhu Chen, Yirong Dong, Jinrong Wang, Fangfang Li, Xiuqi Bao, Dan Zhang
Format: Article
Language:English
Published: Elsevier 2025-02-01
Series:Acta Pharmaceutica Sinica B
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Online Access:http://www.sciencedirect.com/science/article/pii/S221138352400412X
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author Meiyu Shang
Jingwen Ning
Caixia Zang
Jingwei Ma
Yang Yang
Yueqi Jiang
Qiuzhu Chen
Yirong Dong
Jinrong Wang
Fangfang Li
Xiuqi Bao
Dan Zhang
author_facet Meiyu Shang
Jingwen Ning
Caixia Zang
Jingwei Ma
Yang Yang
Yueqi Jiang
Qiuzhu Chen
Yirong Dong
Jinrong Wang
Fangfang Li
Xiuqi Bao
Dan Zhang
author_sort Meiyu Shang
collection DOAJ
description Increasing evidence shows that the early lesions of Parkinson's disease (PD) originate from gut, and correction of microbiota dysbiosis is a promising therapy for PD. FLZ is a neuroprotective agent on PD, which has been validated capable of alleviating microbiota dysbiosis in PD mice. However, the detailed mechanisms still need elucidated. Through metabolomics and 16S rRNA analysis, we identified glycoursodeoxycholic acid (GUDCA) was the most affected differential microbial metabolite by FLZ treatment, which was specially and negatively regulated by Clostridium innocuum, a differential microbiota with the strongest correlation to GUDCA production, through inhibiting bile salt hydrolase (BSH) enzyme. The protection of GUDCA on colon and brain were also clarified in PD models, showing that it could activate Nrf2 pathway, further validating that FLZ protected dopaminergic neurons through promoting GUDCA production. Our study uncovered that FLZ improved PD through microbiota–gut–brain axis, and also gave insights into modulation of microbial metabolites may serve as an important strategy for treating PD.
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publishDate 2025-02-01
publisher Elsevier
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series Acta Pharmaceutica Sinica B
spelling doaj-art-6781e1dae1694c6d80f8a9cd3fb9ead02025-08-20T01:58:28ZengElsevierActa Pharmaceutica Sinica B2211-38352025-02-0115297399010.1016/j.apsb.2024.10.011FLZ attenuates Parkinson's disease pathological damage by increasing glycoursodeoxycholic acid production via down-regulating Clostridium innocuumMeiyu Shang0Jingwen Ning1Caixia Zang2Jingwei Ma3Yang Yang4Yueqi Jiang5Qiuzhu Chen6Yirong Dong7Jinrong Wang8Fangfang Li9Xiuqi Bao10Dan Zhang11State Key Laboratory of Bioactive Substrate and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050, ChinaState Key Laboratory of Bioactive Substrate and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050, ChinaState Key Laboratory of Bioactive Substrate and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050, ChinaState Key Laboratory of Bioactive Substrate and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050, ChinaState Key Laboratory of Bioactive Substrate and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050, ChinaState Key Laboratory of Bioactive Substrate and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050, ChinaState Key Laboratory of Bioactive Substrate and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050, ChinaState Key Laboratory of Bioactive Substrate and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050, ChinaState Key Laboratory of Bioactive Substrate and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050, ChinaState Key Laboratory of Bioactive Substrate and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050, ChinaCorresponding authors.; State Key Laboratory of Bioactive Substrate and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050, ChinaCorresponding authors.; State Key Laboratory of Bioactive Substrate and Function of Natural Medicine, Institute of Materia Medica, Chinese Academy of Medical Sciences and Peking Union Medical College, Beijing 100050, ChinaIncreasing evidence shows that the early lesions of Parkinson's disease (PD) originate from gut, and correction of microbiota dysbiosis is a promising therapy for PD. FLZ is a neuroprotective agent on PD, which has been validated capable of alleviating microbiota dysbiosis in PD mice. However, the detailed mechanisms still need elucidated. Through metabolomics and 16S rRNA analysis, we identified glycoursodeoxycholic acid (GUDCA) was the most affected differential microbial metabolite by FLZ treatment, which was specially and negatively regulated by Clostridium innocuum, a differential microbiota with the strongest correlation to GUDCA production, through inhibiting bile salt hydrolase (BSH) enzyme. The protection of GUDCA on colon and brain were also clarified in PD models, showing that it could activate Nrf2 pathway, further validating that FLZ protected dopaminergic neurons through promoting GUDCA production. Our study uncovered that FLZ improved PD through microbiota–gut–brain axis, and also gave insights into modulation of microbial metabolites may serve as an important strategy for treating PD.http://www.sciencedirect.com/science/article/pii/S221138352400412XParkinson's diseaseFLZGlycoursodeoxycholic acidClostridium innocuumGut–brain axisMicrobiota dysbiosis
spellingShingle Meiyu Shang
Jingwen Ning
Caixia Zang
Jingwei Ma
Yang Yang
Yueqi Jiang
Qiuzhu Chen
Yirong Dong
Jinrong Wang
Fangfang Li
Xiuqi Bao
Dan Zhang
FLZ attenuates Parkinson's disease pathological damage by increasing glycoursodeoxycholic acid production via down-regulating Clostridium innocuum
Acta Pharmaceutica Sinica B
Parkinson's disease
FLZ
Glycoursodeoxycholic acid
Clostridium innocuum
Gut–brain axis
Microbiota dysbiosis
title FLZ attenuates Parkinson's disease pathological damage by increasing glycoursodeoxycholic acid production via down-regulating Clostridium innocuum
title_full FLZ attenuates Parkinson's disease pathological damage by increasing glycoursodeoxycholic acid production via down-regulating Clostridium innocuum
title_fullStr FLZ attenuates Parkinson's disease pathological damage by increasing glycoursodeoxycholic acid production via down-regulating Clostridium innocuum
title_full_unstemmed FLZ attenuates Parkinson's disease pathological damage by increasing glycoursodeoxycholic acid production via down-regulating Clostridium innocuum
title_short FLZ attenuates Parkinson's disease pathological damage by increasing glycoursodeoxycholic acid production via down-regulating Clostridium innocuum
title_sort flz attenuates parkinson s disease pathological damage by increasing glycoursodeoxycholic acid production via down regulating clostridium innocuum
topic Parkinson's disease
FLZ
Glycoursodeoxycholic acid
Clostridium innocuum
Gut–brain axis
Microbiota dysbiosis
url http://www.sciencedirect.com/science/article/pii/S221138352400412X
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