Could Perinatal Asphyxia Induce a Synaptopathy? New Highlights from an Experimental Model

Birth asphyxia also termed perinatal asphyxia is an obstetric complication that strongly affects brain structure and function. Central nervous system is highly susceptible to oxidative damage caused by perinatal asphyxia while activation and maturity of the proper pathways are relevant to avoiding a...

Full description

Saved in:
Bibliographic Details
Main Authors: María Inés Herrera, Matilde Otero-Losada, Lucas Daniel Udovin, Carlos Kusnier, Rodolfo Kölliker-Frers, Wanderley de Souza, Francisco Capani
Format: Article
Language:English
Published: Wiley 2017-01-01
Series:Neural Plasticity
Online Access:http://dx.doi.org/10.1155/2017/3436943
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849410662491488256
author María Inés Herrera
Matilde Otero-Losada
Lucas Daniel Udovin
Carlos Kusnier
Rodolfo Kölliker-Frers
Wanderley de Souza
Francisco Capani
author_facet María Inés Herrera
Matilde Otero-Losada
Lucas Daniel Udovin
Carlos Kusnier
Rodolfo Kölliker-Frers
Wanderley de Souza
Francisco Capani
author_sort María Inés Herrera
collection DOAJ
description Birth asphyxia also termed perinatal asphyxia is an obstetric complication that strongly affects brain structure and function. Central nervous system is highly susceptible to oxidative damage caused by perinatal asphyxia while activation and maturity of the proper pathways are relevant to avoiding abnormal neural development. Perinatal asphyxia is associated with high morbimortality in term and preterm neonates. Although several studies have demonstrated a variety of biochemical and molecular pathways involved in perinatal asphyxia physiopathology, little is known about the synaptic alterations induced by perinatal asphyxia. Nearly 25% of the newborns who survive perinatal asphyxia develop neurological disorders such as cerebral palsy and certain neurodevelopmental and learning disabilities where synaptic connectivity disturbances may be involved. Accordingly, here we review and discuss the association of possible synaptic dysfunction with perinatal asphyxia on the basis of updated evidence from an experimental model.
format Article
id doaj-art-67171486518e4d32ae445aafd90e23c7
institution Kabale University
issn 2090-5904
1687-5443
language English
publishDate 2017-01-01
publisher Wiley
record_format Article
series Neural Plasticity
spelling doaj-art-67171486518e4d32ae445aafd90e23c72025-08-20T03:35:01ZengWileyNeural Plasticity2090-59041687-54432017-01-01201710.1155/2017/34369433436943Could Perinatal Asphyxia Induce a Synaptopathy? New Highlights from an Experimental ModelMaría Inés Herrera0Matilde Otero-Losada1Lucas Daniel Udovin2Carlos Kusnier3Rodolfo Kölliker-Frers4Wanderley de Souza5Francisco Capani6Centro de Investigaciones en Psicología y Psicopedagogía, Facultad de Psicología, Universidad Católica Argentina, Buenos Aires, ArgentinaInstituto de Investigaciones Cardiológicas (ININCA), UBA-CONICET, CABA, Buenos Aires, ArgentinaInstituto de Investigaciones Cardiológicas (ININCA), UBA-CONICET, CABA, Buenos Aires, ArgentinaInstituto de Investigaciones Cardiológicas (ININCA), UBA-CONICET, CABA, Buenos Aires, ArgentinaInstituto de Investigaciones Cardiológicas (ININCA), UBA-CONICET, CABA, Buenos Aires, ArgentinaLaboratório de Ultraestrutura Celular Hertha Meyer, Instituto de Biofísica Carlos Chagas Filho, Universidade Federal do Rio de Janeiro (UFRJ), Rio de Janeiro, RJ, BrazilCentro de Investigaciones en Psicología y Psicopedagogía, Facultad de Psicología, Universidad Católica Argentina, Buenos Aires, ArgentinaBirth asphyxia also termed perinatal asphyxia is an obstetric complication that strongly affects brain structure and function. Central nervous system is highly susceptible to oxidative damage caused by perinatal asphyxia while activation and maturity of the proper pathways are relevant to avoiding abnormal neural development. Perinatal asphyxia is associated with high morbimortality in term and preterm neonates. Although several studies have demonstrated a variety of biochemical and molecular pathways involved in perinatal asphyxia physiopathology, little is known about the synaptic alterations induced by perinatal asphyxia. Nearly 25% of the newborns who survive perinatal asphyxia develop neurological disorders such as cerebral palsy and certain neurodevelopmental and learning disabilities where synaptic connectivity disturbances may be involved. Accordingly, here we review and discuss the association of possible synaptic dysfunction with perinatal asphyxia on the basis of updated evidence from an experimental model.http://dx.doi.org/10.1155/2017/3436943
spellingShingle María Inés Herrera
Matilde Otero-Losada
Lucas Daniel Udovin
Carlos Kusnier
Rodolfo Kölliker-Frers
Wanderley de Souza
Francisco Capani
Could Perinatal Asphyxia Induce a Synaptopathy? New Highlights from an Experimental Model
Neural Plasticity
title Could Perinatal Asphyxia Induce a Synaptopathy? New Highlights from an Experimental Model
title_full Could Perinatal Asphyxia Induce a Synaptopathy? New Highlights from an Experimental Model
title_fullStr Could Perinatal Asphyxia Induce a Synaptopathy? New Highlights from an Experimental Model
title_full_unstemmed Could Perinatal Asphyxia Induce a Synaptopathy? New Highlights from an Experimental Model
title_short Could Perinatal Asphyxia Induce a Synaptopathy? New Highlights from an Experimental Model
title_sort could perinatal asphyxia induce a synaptopathy new highlights from an experimental model
url http://dx.doi.org/10.1155/2017/3436943
work_keys_str_mv AT mariainesherrera couldperinatalasphyxiainduceasynaptopathynewhighlightsfromanexperimentalmodel
AT matildeoterolosada couldperinatalasphyxiainduceasynaptopathynewhighlightsfromanexperimentalmodel
AT lucasdanieludovin couldperinatalasphyxiainduceasynaptopathynewhighlightsfromanexperimentalmodel
AT carloskusnier couldperinatalasphyxiainduceasynaptopathynewhighlightsfromanexperimentalmodel
AT rodolfokollikerfrers couldperinatalasphyxiainduceasynaptopathynewhighlightsfromanexperimentalmodel
AT wanderleydesouza couldperinatalasphyxiainduceasynaptopathynewhighlightsfromanexperimentalmodel
AT franciscocapani couldperinatalasphyxiainduceasynaptopathynewhighlightsfromanexperimentalmodel