Study on Carbohydrate Metabolism in Adult Zebrafish (Danio rerio)

Excessive carbohydrate intake leads to metabolic disorders in fish. However, few literatures have reported the appropriate carbohydrate level for zebrafish, and the metabolic response to dietary carbohydrate remains largely unknown in zebrafish. This study assessed the responses of zebrafish and zeb...

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Main Authors: Longwei Xi, Qisheng Lu, Yulong Liu, Yulong Gong, Haokun Liu, Junyan Jin, Zhimin Zhang, Yunxia Yang, Xiaoming Zhu, Dong Han, Shouqi Xie
Format: Article
Language:English
Published: Wiley 2023-01-01
Series:Aquaculture Nutrition
Online Access:http://dx.doi.org/10.1155/2023/1397508
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author Longwei Xi
Qisheng Lu
Yulong Liu
Yulong Gong
Haokun Liu
Junyan Jin
Zhimin Zhang
Yunxia Yang
Xiaoming Zhu
Dong Han
Shouqi Xie
author_facet Longwei Xi
Qisheng Lu
Yulong Liu
Yulong Gong
Haokun Liu
Junyan Jin
Zhimin Zhang
Yunxia Yang
Xiaoming Zhu
Dong Han
Shouqi Xie
author_sort Longwei Xi
collection DOAJ
description Excessive carbohydrate intake leads to metabolic disorders in fish. However, few literatures have reported the appropriate carbohydrate level for zebrafish, and the metabolic response to dietary carbohydrate remains largely unknown in zebrafish. This study assessed the responses of zebrafish and zebrafish liver cell line (ZFL) to different carbohydrate levels. In vivo results showed that ≥30% dietary dextrin levels significantly increased the plasma glucose content, activated the expression of hepatic glycolysis-related genes, and inhibited the expression of hepatic gluconeogenesis-related genes in zebrafish. Oil red O staining, triglyceride content, and Hematoxylin-Eosin staining results showed that dietary dextrin levels of ≥30% significantly increased lipid accumulation and liver damage, as well as processes related to glycolipid metabolism and inflammation in zebrafish. In ZFL, the transcription factor sterol regulatory element binding protein-1c signal intensity, 4,4-difluoro-1,3,5,7,8-pentamethyl-4-bora-3a,4a-diaza-s-indacene (BODIPY 493/503) signal intensity, and triglyceride content were also significantly increased when incubated in high glucose, along with abnormal glycolipid metabolism and increased inflammation-related genes. In conclusion, we demonstrated that the maximum dietary carbohydrate level in adult zebrafish should be less than 30%. Excess dietary carbohydrates (30%–50%) caused hepatic steatosis and damage to zebrafish, similar to that seen in aquaculture species. Thus, this study assessed responses to different carbohydrate levels in zebrafish and illustrated that zebrafish is an optimal model for investigating glucose metabolism in some aquatic animals.
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spelling doaj-art-65feafa1d5d947f89ce78b9f8c0eb6352025-08-20T02:23:46ZengWileyAquaculture Nutrition1365-20952023-01-01202310.1155/2023/1397508Study on Carbohydrate Metabolism in Adult Zebrafish (Danio rerio)Longwei Xi0Qisheng Lu1Yulong Liu2Yulong Gong3Haokun Liu4Junyan Jin5Zhimin Zhang6Yunxia Yang7Xiaoming Zhu8Dong Han9Shouqi Xie10State Key Laboratory of Freshwater Ecology and BiotechnologyState Key Laboratory of Freshwater Ecology and BiotechnologyState Key Laboratory of Freshwater Ecology and BiotechnologyState Key Laboratory of Freshwater Ecology and BiotechnologyState Key Laboratory of Freshwater Ecology and BiotechnologyState Key Laboratory of Freshwater Ecology and BiotechnologyState Key Laboratory of Freshwater Ecology and BiotechnologyState Key Laboratory of Freshwater Ecology and BiotechnologyState Key Laboratory of Freshwater Ecology and BiotechnologyState Key Laboratory of Freshwater Ecology and BiotechnologyState Key Laboratory of Freshwater Ecology and BiotechnologyExcessive carbohydrate intake leads to metabolic disorders in fish. However, few literatures have reported the appropriate carbohydrate level for zebrafish, and the metabolic response to dietary carbohydrate remains largely unknown in zebrafish. This study assessed the responses of zebrafish and zebrafish liver cell line (ZFL) to different carbohydrate levels. In vivo results showed that ≥30% dietary dextrin levels significantly increased the plasma glucose content, activated the expression of hepatic glycolysis-related genes, and inhibited the expression of hepatic gluconeogenesis-related genes in zebrafish. Oil red O staining, triglyceride content, and Hematoxylin-Eosin staining results showed that dietary dextrin levels of ≥30% significantly increased lipid accumulation and liver damage, as well as processes related to glycolipid metabolism and inflammation in zebrafish. In ZFL, the transcription factor sterol regulatory element binding protein-1c signal intensity, 4,4-difluoro-1,3,5,7,8-pentamethyl-4-bora-3a,4a-diaza-s-indacene (BODIPY 493/503) signal intensity, and triglyceride content were also significantly increased when incubated in high glucose, along with abnormal glycolipid metabolism and increased inflammation-related genes. In conclusion, we demonstrated that the maximum dietary carbohydrate level in adult zebrafish should be less than 30%. Excess dietary carbohydrates (30%–50%) caused hepatic steatosis and damage to zebrafish, similar to that seen in aquaculture species. Thus, this study assessed responses to different carbohydrate levels in zebrafish and illustrated that zebrafish is an optimal model for investigating glucose metabolism in some aquatic animals.http://dx.doi.org/10.1155/2023/1397508
spellingShingle Longwei Xi
Qisheng Lu
Yulong Liu
Yulong Gong
Haokun Liu
Junyan Jin
Zhimin Zhang
Yunxia Yang
Xiaoming Zhu
Dong Han
Shouqi Xie
Study on Carbohydrate Metabolism in Adult Zebrafish (Danio rerio)
Aquaculture Nutrition
title Study on Carbohydrate Metabolism in Adult Zebrafish (Danio rerio)
title_full Study on Carbohydrate Metabolism in Adult Zebrafish (Danio rerio)
title_fullStr Study on Carbohydrate Metabolism in Adult Zebrafish (Danio rerio)
title_full_unstemmed Study on Carbohydrate Metabolism in Adult Zebrafish (Danio rerio)
title_short Study on Carbohydrate Metabolism in Adult Zebrafish (Danio rerio)
title_sort study on carbohydrate metabolism in adult zebrafish danio rerio
url http://dx.doi.org/10.1155/2023/1397508
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