Autosomal Dominant Pseudohypoaldosteronism Type 1 in an Infant with Salt Wasting Crisis Associated with Urinary Tract Infection and Obstructive Uropathy

Type 1 pseudohypoaldosteronism (PHA1) is a salt wasting syndrome caused by renal resistance to aldosterone. Primary renal PHA1 or autosomal dominant PHA1 is caused by mutations in mineralocorticoids receptor gene (NR3C2), while secondary PHA1 is frequently associated with urinary tract infection (UT...

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Main Authors: Sasigarn A. Bowden, Corin Cozzi, Scott E. Hickey, Devon Lamb Thrush, Caroline Astbury, Sushma Nuthakki
Format: Article
Language:English
Published: Wiley 2013-01-01
Series:Case Reports in Endocrinology
Online Access:http://dx.doi.org/10.1155/2013/524647
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author Sasigarn A. Bowden
Corin Cozzi
Scott E. Hickey
Devon Lamb Thrush
Caroline Astbury
Sushma Nuthakki
author_facet Sasigarn A. Bowden
Corin Cozzi
Scott E. Hickey
Devon Lamb Thrush
Caroline Astbury
Sushma Nuthakki
author_sort Sasigarn A. Bowden
collection DOAJ
description Type 1 pseudohypoaldosteronism (PHA1) is a salt wasting syndrome caused by renal resistance to aldosterone. Primary renal PHA1 or autosomal dominant PHA1 is caused by mutations in mineralocorticoids receptor gene (NR3C2), while secondary PHA1 is frequently associated with urinary tract infection (UTI) and/or urinary tract malformations (UTM). We report a 14-day-old male infant presenting with severe hyperkalemia, hyponatremic dehydration, metabolic acidosis, and markedly elevated serum aldosterone level, initially thought to have secondary PHA1 due to the associated UTI and posterior urethral valves. His serum aldosterone remained elevated at 5 months of age, despite resolution of salt wasting symptoms. Chromosomal microarray analysis revealed a deletion of exons 3–5 in NR3C2 in the patient and his asymptomatic mother who also had elevated serum aldosterone level, confirming that he had primary or autosomal dominant PHA1. Our case raises the possibility that some patients with secondary PHA1 attributed to UTI and/or UTM may instead have primary autosomal dominant PHA1, for which genetic testing should be considered to identify the cause, determine future recurrence risk, and possibly prevent the life-threatening salt wasting in a subsequent family member. Future clinical research is needed to investigate the potential overlapping between secondary PHA1 and primary autosomal dominant PHA1.
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spelling doaj-art-658a799f21d240ee8eca358a459f2cd62025-08-20T03:34:17ZengWileyCase Reports in Endocrinology2090-65012090-651X2013-01-01201310.1155/2013/524647524647Autosomal Dominant Pseudohypoaldosteronism Type 1 in an Infant with Salt Wasting Crisis Associated with Urinary Tract Infection and Obstructive UropathySasigarn A. Bowden0Corin Cozzi1Scott E. Hickey2Devon Lamb Thrush3Caroline Astbury4Sushma Nuthakki5Division of Endocrinology, Nationwide Children’s Hospital/The Ohio State University College of Medicine, Columbus, OH 43205, USADivision of Neonatology, Nationwide Children’s Hospital/The Ohio State University College of Medicine, Columbus, OH 43205, USADivision of Molecular & Human Genetics, Department of Pediatrics, Nationwide Children’s Hospital/The Ohio State University College of Medicine, Columbus, OH 43205, USADepartment of Pathology and Laboratory Medicine, Nationwide Children’s Hospital/The Ohio State University College of Medicine, Columbus, OH 43205, USADepartment of Pathology and Laboratory Medicine, Nationwide Children’s Hospital/The Ohio State University College of Medicine, Columbus, OH 43205, USADivision of Neonatology, Nationwide Children’s Hospital/The Ohio State University College of Medicine, Columbus, OH 43205, USAType 1 pseudohypoaldosteronism (PHA1) is a salt wasting syndrome caused by renal resistance to aldosterone. Primary renal PHA1 or autosomal dominant PHA1 is caused by mutations in mineralocorticoids receptor gene (NR3C2), while secondary PHA1 is frequently associated with urinary tract infection (UTI) and/or urinary tract malformations (UTM). We report a 14-day-old male infant presenting with severe hyperkalemia, hyponatremic dehydration, metabolic acidosis, and markedly elevated serum aldosterone level, initially thought to have secondary PHA1 due to the associated UTI and posterior urethral valves. His serum aldosterone remained elevated at 5 months of age, despite resolution of salt wasting symptoms. Chromosomal microarray analysis revealed a deletion of exons 3–5 in NR3C2 in the patient and his asymptomatic mother who also had elevated serum aldosterone level, confirming that he had primary or autosomal dominant PHA1. Our case raises the possibility that some patients with secondary PHA1 attributed to UTI and/or UTM may instead have primary autosomal dominant PHA1, for which genetic testing should be considered to identify the cause, determine future recurrence risk, and possibly prevent the life-threatening salt wasting in a subsequent family member. Future clinical research is needed to investigate the potential overlapping between secondary PHA1 and primary autosomal dominant PHA1.http://dx.doi.org/10.1155/2013/524647
spellingShingle Sasigarn A. Bowden
Corin Cozzi
Scott E. Hickey
Devon Lamb Thrush
Caroline Astbury
Sushma Nuthakki
Autosomal Dominant Pseudohypoaldosteronism Type 1 in an Infant with Salt Wasting Crisis Associated with Urinary Tract Infection and Obstructive Uropathy
Case Reports in Endocrinology
title Autosomal Dominant Pseudohypoaldosteronism Type 1 in an Infant with Salt Wasting Crisis Associated with Urinary Tract Infection and Obstructive Uropathy
title_full Autosomal Dominant Pseudohypoaldosteronism Type 1 in an Infant with Salt Wasting Crisis Associated with Urinary Tract Infection and Obstructive Uropathy
title_fullStr Autosomal Dominant Pseudohypoaldosteronism Type 1 in an Infant with Salt Wasting Crisis Associated with Urinary Tract Infection and Obstructive Uropathy
title_full_unstemmed Autosomal Dominant Pseudohypoaldosteronism Type 1 in an Infant with Salt Wasting Crisis Associated with Urinary Tract Infection and Obstructive Uropathy
title_short Autosomal Dominant Pseudohypoaldosteronism Type 1 in an Infant with Salt Wasting Crisis Associated with Urinary Tract Infection and Obstructive Uropathy
title_sort autosomal dominant pseudohypoaldosteronism type 1 in an infant with salt wasting crisis associated with urinary tract infection and obstructive uropathy
url http://dx.doi.org/10.1155/2013/524647
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