Dysregulation of autophagy during photoaging reduce oxidative stress and inflammatory damage caused by UV

Photoaging, the premature aging of skin due to chronic ultraviolet (UV) exposure, is a growing concern in dermatology and cosmetic science. While UV radiation is known to induce DNA damage, oxidative stress, and inflammation in skin cells, recent research unveils a promising countermeasure: autophag...

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Main Authors: Zhongsong Zhang, Run Tan, Zuanyu Xiong, Yanyan Feng, Long Chen
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-05-01
Series:Frontiers in Pharmacology
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Online Access:https://www.frontiersin.org/articles/10.3389/fphar.2025.1562845/full
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author Zhongsong Zhang
Run Tan
Run Tan
Zuanyu Xiong
Yanyan Feng
Yanyan Feng
Long Chen
author_facet Zhongsong Zhang
Run Tan
Run Tan
Zuanyu Xiong
Yanyan Feng
Yanyan Feng
Long Chen
author_sort Zhongsong Zhang
collection DOAJ
description Photoaging, the premature aging of skin due to chronic ultraviolet (UV) exposure, is a growing concern in dermatology and cosmetic science. While UV radiation is known to induce DNA damage, oxidative stress, and inflammation in skin cells, recent research unveils a promising countermeasure: autophagy. This review explores the intricate relationship between autophagy and photoaging, highlighting how this cellular recycling process can mitigate UV-induced damage. We begin by examining the differential impacts of UVA and UVB radiation on skin cells and the role of oxidative stress in accelerating photoaging. Next, we delve into the molecular mechanisms of autophagy, including its various forms and regulatory pathways. Central to this review is the discussion of autophagy’s protective functions, such as the clearance of damaged organelles and proteins, and its role in maintaining genomic integrity. Furthermore, we address the current challenges in harnessing autophagy for therapeutic purposes, including the need for selective autophagy inducers and a deeper understanding of its context-dependent effects. By synthesizing recent advancements and proposing future research directions, this review underscores the potential of autophagy modulation as a novel strategy to prevent and treat photoaging. This comprehensive analysis aims to inspire further investigation into autophagy-based interventions, offering new hope for preserving skin health in the face of environmental stressors.
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publishDate 2025-05-01
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spelling doaj-art-64eee337c2e94987b72f340fc9da69f12025-08-20T03:52:47ZengFrontiers Media S.A.Frontiers in Pharmacology1663-98122025-05-011610.3389/fphar.2025.15628451562845Dysregulation of autophagy during photoaging reduce oxidative stress and inflammatory damage caused by UVZhongsong Zhang0Run Tan1Run Tan2Zuanyu Xiong3Yanyan Feng4Yanyan Feng5Long Chen6School of Basic Medical Sciences, Chengdu Medical College, Chengdu, ChinaDepartment of Dermatology, Chengdu Second People‘s Hospital, Chengdu, Sichuan Province, ChinaSchool of Clinical Medicine, Chengdu Medical College, Chengdu, ChinaDepartment of Medical Aesthetics, Nanbu People‘s Hospital, Nanchong, ChinaDepartment of Dermatology, Chengdu Second People‘s Hospital, Chengdu, Sichuan Province, ChinaSchool of Clinical Medicine, Chengdu Medical College, Chengdu, ChinaSchool of Basic Medical Sciences, Chengdu Medical College, Chengdu, ChinaPhotoaging, the premature aging of skin due to chronic ultraviolet (UV) exposure, is a growing concern in dermatology and cosmetic science. While UV radiation is known to induce DNA damage, oxidative stress, and inflammation in skin cells, recent research unveils a promising countermeasure: autophagy. This review explores the intricate relationship between autophagy and photoaging, highlighting how this cellular recycling process can mitigate UV-induced damage. We begin by examining the differential impacts of UVA and UVB radiation on skin cells and the role of oxidative stress in accelerating photoaging. Next, we delve into the molecular mechanisms of autophagy, including its various forms and regulatory pathways. Central to this review is the discussion of autophagy’s protective functions, such as the clearance of damaged organelles and proteins, and its role in maintaining genomic integrity. Furthermore, we address the current challenges in harnessing autophagy for therapeutic purposes, including the need for selective autophagy inducers and a deeper understanding of its context-dependent effects. By synthesizing recent advancements and proposing future research directions, this review underscores the potential of autophagy modulation as a novel strategy to prevent and treat photoaging. This comprehensive analysis aims to inspire further investigation into autophagy-based interventions, offering new hope for preserving skin health in the face of environmental stressors.https://www.frontiersin.org/articles/10.3389/fphar.2025.1562845/fullphotoagingautophagyUVROSskin aging
spellingShingle Zhongsong Zhang
Run Tan
Run Tan
Zuanyu Xiong
Yanyan Feng
Yanyan Feng
Long Chen
Dysregulation of autophagy during photoaging reduce oxidative stress and inflammatory damage caused by UV
Frontiers in Pharmacology
photoaging
autophagy
UV
ROS
skin aging
title Dysregulation of autophagy during photoaging reduce oxidative stress and inflammatory damage caused by UV
title_full Dysregulation of autophagy during photoaging reduce oxidative stress and inflammatory damage caused by UV
title_fullStr Dysregulation of autophagy during photoaging reduce oxidative stress and inflammatory damage caused by UV
title_full_unstemmed Dysregulation of autophagy during photoaging reduce oxidative stress and inflammatory damage caused by UV
title_short Dysregulation of autophagy during photoaging reduce oxidative stress and inflammatory damage caused by UV
title_sort dysregulation of autophagy during photoaging reduce oxidative stress and inflammatory damage caused by uv
topic photoaging
autophagy
UV
ROS
skin aging
url https://www.frontiersin.org/articles/10.3389/fphar.2025.1562845/full
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