Research progress on the mechanism of curcumin anti-oxidative stress based on signaling pathway

Oxidative stress refers to an imbalance between oxidative capacity and antioxidant capacity, leading to oxidative damage to proteins, lipids, and DNA, which can result in cell senescence or death. It is closely associated with the occurrence and development of various diseases, including cardiovascu...

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Main Authors: Jie Cui, Haonan Li, Tianyi Zhang, Fengli Lin, Meiyun Chen, Guimin Zhang, Zhong Feng
Format: Article
Language:English
Published: Frontiers Media S.A. 2025-04-01
Series:Frontiers in Pharmacology
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Online Access:https://www.frontiersin.org/articles/10.3389/fphar.2025.1548073/full
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author Jie Cui
Haonan Li
Tianyi Zhang
Fengli Lin
Meiyun Chen
Guimin Zhang
Zhong Feng
Zhong Feng
Zhong Feng
Zhong Feng
author_facet Jie Cui
Haonan Li
Tianyi Zhang
Fengli Lin
Meiyun Chen
Guimin Zhang
Zhong Feng
Zhong Feng
Zhong Feng
Zhong Feng
author_sort Jie Cui
collection DOAJ
description Oxidative stress refers to an imbalance between oxidative capacity and antioxidant capacity, leading to oxidative damage to proteins, lipids, and DNA, which can result in cell senescence or death. It is closely associated with the occurrence and development of various diseases, including cardiovascular diseases, nephropathy, malignant tumors, neurodegenerative diseases, hypertension, diabetes, and inflammatory diseases. Curcumin is a natural polyphenol compound of β-diketone, which has a wide range of pharmacological activities such as anti-inflammatory, antibacterial, anti-oxidative stress, anti-tumor, anti-fibrosis, and hypolipidemic, demonstrating broad research and development value. It has a wide range of biological targets and can bind to various endogenous biomolecules. Additionally, it maintains the redox balance primarily by scavenging ROS, enhancing the activity of antioxidant enzymes, inhibiting lipid peroxidation, and chelating metal ions. This paper systematically describes the antioxidative stress mechanisms of curcumin from the perspective of signaling pathways, focusing on the Keap1-Nrf2/ARE, NF-κB, NOX, MAPK and other pathways. The study also discusses potential pathway targets and the complex crosstalk among these pathways, aiming to provide insights for further research on curcumin’s antioxidant mechanisms and its clinical applications.
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institution OA Journals
issn 1663-9812
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publishDate 2025-04-01
publisher Frontiers Media S.A.
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series Frontiers in Pharmacology
spelling doaj-art-64233aa52f3b4fcf8581be1bbb8a3a5b2025-08-20T02:09:02ZengFrontiers Media S.A.Frontiers in Pharmacology1663-98122025-04-011610.3389/fphar.2025.15480731548073Research progress on the mechanism of curcumin anti-oxidative stress based on signaling pathwayJie Cui0Haonan Li1Tianyi Zhang2Fengli Lin3Meiyun Chen4Guimin Zhang5Zhong Feng6Zhong Feng7Zhong Feng8Zhong Feng9School of Pharmacy, Shandong University of Traditional Chinese Medicine, Jinan, ChinaSchool of Pharmacy, Shandong University of Traditional Chinese Medicine, Jinan, ChinaGraduate School, Tianjin University of Traditional Chinese Medicine, Tianjin, ChinaGraduate School, Tianjin University of Traditional Chinese Medicine, Tianjin, ChinaSchool of Pharmaceutical Sciences (Shenzhen), Sun Yat-sen University, Shenzhen, ChinaLunan Pharmaceutical Group Co., Ltd., Linyi, ChinaSchool of Pharmacy, Shandong University of Traditional Chinese Medicine, Jinan, ChinaGraduate School, Tianjin University of Traditional Chinese Medicine, Tianjin, ChinaSchool of Pharmaceutical Sciences (Shenzhen), Sun Yat-sen University, Shenzhen, ChinaLunan Pharmaceutical Group Co., Ltd., Linyi, ChinaOxidative stress refers to an imbalance between oxidative capacity and antioxidant capacity, leading to oxidative damage to proteins, lipids, and DNA, which can result in cell senescence or death. It is closely associated with the occurrence and development of various diseases, including cardiovascular diseases, nephropathy, malignant tumors, neurodegenerative diseases, hypertension, diabetes, and inflammatory diseases. Curcumin is a natural polyphenol compound of β-diketone, which has a wide range of pharmacological activities such as anti-inflammatory, antibacterial, anti-oxidative stress, anti-tumor, anti-fibrosis, and hypolipidemic, demonstrating broad research and development value. It has a wide range of biological targets and can bind to various endogenous biomolecules. Additionally, it maintains the redox balance primarily by scavenging ROS, enhancing the activity of antioxidant enzymes, inhibiting lipid peroxidation, and chelating metal ions. This paper systematically describes the antioxidative stress mechanisms of curcumin from the perspective of signaling pathways, focusing on the Keap1-Nrf2/ARE, NF-κB, NOX, MAPK and other pathways. The study also discusses potential pathway targets and the complex crosstalk among these pathways, aiming to provide insights for further research on curcumin’s antioxidant mechanisms and its clinical applications.https://www.frontiersin.org/articles/10.3389/fphar.2025.1548073/fullcurcuminoxidative stressKeap1-Nrf2/ARENF-κBNOXMAPK
spellingShingle Jie Cui
Haonan Li
Tianyi Zhang
Fengli Lin
Meiyun Chen
Guimin Zhang
Zhong Feng
Zhong Feng
Zhong Feng
Zhong Feng
Research progress on the mechanism of curcumin anti-oxidative stress based on signaling pathway
Frontiers in Pharmacology
curcumin
oxidative stress
Keap1-Nrf2/ARE
NF-κB
NOX
MAPK
title Research progress on the mechanism of curcumin anti-oxidative stress based on signaling pathway
title_full Research progress on the mechanism of curcumin anti-oxidative stress based on signaling pathway
title_fullStr Research progress on the mechanism of curcumin anti-oxidative stress based on signaling pathway
title_full_unstemmed Research progress on the mechanism of curcumin anti-oxidative stress based on signaling pathway
title_short Research progress on the mechanism of curcumin anti-oxidative stress based on signaling pathway
title_sort research progress on the mechanism of curcumin anti oxidative stress based on signaling pathway
topic curcumin
oxidative stress
Keap1-Nrf2/ARE
NF-κB
NOX
MAPK
url https://www.frontiersin.org/articles/10.3389/fphar.2025.1548073/full
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