Microvascular dysfunction across organs in heart failure with preserved ejection fraction: the PROSE-HFpEF case-control study

Abstract Background Systemic microvascular dysfunction is proposed as a key pathophysiological process in heart failure with preserved ejection fraction (HFpEF). This study compared microvasculature across vascular beds in HFpEF patients and controls. Methods This prospective, case-control study inc...

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Main Authors: Jerremy Weerts, Blanche L. M. Schroen, Arantxa Barandiarán Aizpurua, Tos T. J. M. Berendschot, Lloyd Brandts, Carroll A. B. Webers, Sami O. Simons, Steven J. R. Meex, Ronald Henry, Carla J. H. van der Kallen, Hans-Peter Brunner-La Rocca, Christian Knackstedt, Stephane R. B. Heymans, Rudolf A. de Boer, Vanessa P. M. van Empel, Alfons J. H. M. Houben
Format: Article
Language:English
Published: BMC 2025-07-01
Series:Cardiovascular Diabetology
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Online Access:https://doi.org/10.1186/s12933-025-02850-1
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author Jerremy Weerts
Blanche L. M. Schroen
Arantxa Barandiarán Aizpurua
Tos T. J. M. Berendschot
Lloyd Brandts
Carroll A. B. Webers
Sami O. Simons
Steven J. R. Meex
Ronald Henry
Carla J. H. van der Kallen
Hans-Peter Brunner-La Rocca
Christian Knackstedt
Stephane R. B. Heymans
Rudolf A. de Boer
Vanessa P. M. van Empel
Alfons J. H. M. Houben
author_facet Jerremy Weerts
Blanche L. M. Schroen
Arantxa Barandiarán Aizpurua
Tos T. J. M. Berendschot
Lloyd Brandts
Carroll A. B. Webers
Sami O. Simons
Steven J. R. Meex
Ronald Henry
Carla J. H. van der Kallen
Hans-Peter Brunner-La Rocca
Christian Knackstedt
Stephane R. B. Heymans
Rudolf A. de Boer
Vanessa P. M. van Empel
Alfons J. H. M. Houben
author_sort Jerremy Weerts
collection DOAJ
description Abstract Background Systemic microvascular dysfunction is proposed as a key pathophysiological process in heart failure with preserved ejection fraction (HFpEF). This study compared microvasculature across vascular beds in HFpEF patients and controls. Methods This prospective, case-control study included subjects ≥ 60years. HFpEF patients were diagnosed in an expert centre. Controls without HF were selected from the Maastricht Study, a population cohort enriched with diabetes mellitus. Microvascular assessments included central retinal venular/arteriolar calibres (CRVE/CRAE), flicker-light-induced retinal dilation, skin microvascular flowmotion and heat-induced hyperemia, and urinary albumin-to-creatinine ratio (UACR). Group differences were evaluated with confounder-adjustments (age, sex, blood pressure, body mass index, diabetes, haemoglobin, smoking). Interactions with sex and diabetes mellitus were tested, and stratified analyses were performed when significant interactions were present. Results Microvascular assessments were performed in 138 HFpEF patients and 2140 controls. Microvascular differences were present between groups in all vascular beds. However, confounder-adjusted analyses attenuated differences. Confounder-adjusted analyses indicated that HFpEF patients versus controls still had retinal differences: narrower CRVE (− 8.1 μm, p = 0.008) and narrower CRAE trend (− 3.5 μm, p = 0.073), but similar flicker-light-induced retinal venular/arteriolar dilation (− 0.23%, p = 0.392; − 0.18%, p = 0.593, respectively). Confounder-adjusted analyses showed similar skin flowmotion measures (i.e. endothelial power − 0.09log(PU2), p = 0.181), and heat-induced hyperemia (0.02log(%), p = 0.605) between groups. UACR remained higher in HFpEF after confounder adjustments (0.56log(g/mol), p = < 0.001). Interaction analyses revealed that female patients had narrower CRVE versus controls (pint=0.023; females − 13.8 μm, p < 0.001; males 1.2 μm, p = 0.812). Patients had lower skin endothelial flowmotion power only when diabetes was co-occurring (pint=0.048; − 0.36 log(PU2 ), p = 0.014). UACR was higher in male and female patients versus controls, but was more pronounced in males (pint=0.002). Conclusions HFpEF patients showed microvascular differences versus controls across all vascular beds studied. However, confounder-adjusted differences remained significant in eyes and kidneys. The findings across multiple organs support that MVD is likely a more systemic process than only local MVD in HFpEF, and possible sex-specific underlying pathophysiology. Registration URL: https://onderzoekmetmensen.nl ; Unique identifier: NL7655. Graphical abstract Figure created in BioRender (i88u523).
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spelling doaj-art-63c87542403e42f1ac984fa134d133be2025-08-20T03:42:29ZengBMCCardiovascular Diabetology1475-28402025-07-0124111410.1186/s12933-025-02850-1Microvascular dysfunction across organs in heart failure with preserved ejection fraction: the PROSE-HFpEF case-control studyJerremy Weerts0Blanche L. M. Schroen1Arantxa Barandiarán Aizpurua2Tos T. J. M. Berendschot3Lloyd Brandts4Carroll A. B. Webers5Sami O. Simons6Steven J. R. Meex7Ronald Henry8Carla J. H. van der Kallen9Hans-Peter Brunner-La Rocca10Christian Knackstedt11Stephane R. B. Heymans12Rudolf A. de Boer13Vanessa P. M. van Empel14Alfons J. H. M. Houben15Department of Cardiology, CARIM Cardiovascular Research Institute Maastricht, Maastricht University Medical Centre (MUMC+)Department of Cardiology, CARIM Cardiovascular Research Institute Maastricht, Maastricht University Medical Centre (MUMC+)Department of Cardiology, CARIM Cardiovascular Research Institute Maastricht, Maastricht University Medical Centre (MUMC+)University Eye Clinic Maastricht, Maastricht University Medical Centre (MUMC+)Department of Clinical Epidemiology and Medical Technology Assessment, Maastricht University Medical Centre (MUMC+)University Eye Clinic Maastricht, Maastricht University Medical Centre (MUMC+)Department of Respiratory Medicine, NUTRIM Research Institute of Nutrition and Translational Research in Metabolism, Maastricht University Medical Centre (MUMC+)Department of Clinical Chemistry, CARIM School for Cardiovascular Diseases, Maastricht University Medical Centre (MUMC+)Department of Internal Medicine, CARIM Cardiovascular Research Institute Maastricht, Maastricht University Medical Centre (MUMC+)Department of Internal Medicine, CARIM Cardiovascular Research Institute Maastricht, Maastricht University Medical Centre (MUMC+)Department of Cardiology, CARIM Cardiovascular Research Institute Maastricht, Maastricht University Medical Centre (MUMC+)Department of Cardiology, CARIM Cardiovascular Research Institute Maastricht, Maastricht University Medical Centre (MUMC+)Department of Cardiology, CARIM Cardiovascular Research Institute Maastricht, Maastricht University Medical Centre (MUMC+)Department of Cardiology, Thorax Center, Cardiovascular Institute, Erasmus MCDepartment of Cardiology, CARIM Cardiovascular Research Institute Maastricht, Maastricht University Medical Centre (MUMC+)Department of Internal Medicine, CARIM Cardiovascular Research Institute Maastricht, Maastricht University Medical Centre (MUMC+)Abstract Background Systemic microvascular dysfunction is proposed as a key pathophysiological process in heart failure with preserved ejection fraction (HFpEF). This study compared microvasculature across vascular beds in HFpEF patients and controls. Methods This prospective, case-control study included subjects ≥ 60years. HFpEF patients were diagnosed in an expert centre. Controls without HF were selected from the Maastricht Study, a population cohort enriched with diabetes mellitus. Microvascular assessments included central retinal venular/arteriolar calibres (CRVE/CRAE), flicker-light-induced retinal dilation, skin microvascular flowmotion and heat-induced hyperemia, and urinary albumin-to-creatinine ratio (UACR). Group differences were evaluated with confounder-adjustments (age, sex, blood pressure, body mass index, diabetes, haemoglobin, smoking). Interactions with sex and diabetes mellitus were tested, and stratified analyses were performed when significant interactions were present. Results Microvascular assessments were performed in 138 HFpEF patients and 2140 controls. Microvascular differences were present between groups in all vascular beds. However, confounder-adjusted analyses attenuated differences. Confounder-adjusted analyses indicated that HFpEF patients versus controls still had retinal differences: narrower CRVE (− 8.1 μm, p = 0.008) and narrower CRAE trend (− 3.5 μm, p = 0.073), but similar flicker-light-induced retinal venular/arteriolar dilation (− 0.23%, p = 0.392; − 0.18%, p = 0.593, respectively). Confounder-adjusted analyses showed similar skin flowmotion measures (i.e. endothelial power − 0.09log(PU2), p = 0.181), and heat-induced hyperemia (0.02log(%), p = 0.605) between groups. UACR remained higher in HFpEF after confounder adjustments (0.56log(g/mol), p = < 0.001). Interaction analyses revealed that female patients had narrower CRVE versus controls (pint=0.023; females − 13.8 μm, p < 0.001; males 1.2 μm, p = 0.812). Patients had lower skin endothelial flowmotion power only when diabetes was co-occurring (pint=0.048; − 0.36 log(PU2 ), p = 0.014). UACR was higher in male and female patients versus controls, but was more pronounced in males (pint=0.002). Conclusions HFpEF patients showed microvascular differences versus controls across all vascular beds studied. However, confounder-adjusted differences remained significant in eyes and kidneys. The findings across multiple organs support that MVD is likely a more systemic process than only local MVD in HFpEF, and possible sex-specific underlying pathophysiology. Registration URL: https://onderzoekmetmensen.nl ; Unique identifier: NL7655. Graphical abstract Figure created in BioRender (i88u523).https://doi.org/10.1186/s12933-025-02850-1Heart failure with preserved ejection fractionDiastolic heart failureMicrocirculationPathophysiologySex differencesHFpEF
spellingShingle Jerremy Weerts
Blanche L. M. Schroen
Arantxa Barandiarán Aizpurua
Tos T. J. M. Berendschot
Lloyd Brandts
Carroll A. B. Webers
Sami O. Simons
Steven J. R. Meex
Ronald Henry
Carla J. H. van der Kallen
Hans-Peter Brunner-La Rocca
Christian Knackstedt
Stephane R. B. Heymans
Rudolf A. de Boer
Vanessa P. M. van Empel
Alfons J. H. M. Houben
Microvascular dysfunction across organs in heart failure with preserved ejection fraction: the PROSE-HFpEF case-control study
Cardiovascular Diabetology
Heart failure with preserved ejection fraction
Diastolic heart failure
Microcirculation
Pathophysiology
Sex differences
HFpEF
title Microvascular dysfunction across organs in heart failure with preserved ejection fraction: the PROSE-HFpEF case-control study
title_full Microvascular dysfunction across organs in heart failure with preserved ejection fraction: the PROSE-HFpEF case-control study
title_fullStr Microvascular dysfunction across organs in heart failure with preserved ejection fraction: the PROSE-HFpEF case-control study
title_full_unstemmed Microvascular dysfunction across organs in heart failure with preserved ejection fraction: the PROSE-HFpEF case-control study
title_short Microvascular dysfunction across organs in heart failure with preserved ejection fraction: the PROSE-HFpEF case-control study
title_sort microvascular dysfunction across organs in heart failure with preserved ejection fraction the prose hfpef case control study
topic Heart failure with preserved ejection fraction
Diastolic heart failure
Microcirculation
Pathophysiology
Sex differences
HFpEF
url https://doi.org/10.1186/s12933-025-02850-1
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