The E3 ligase c-Cbl modulates microglial phenotypes and contributes to Parkinson’s disease pathology

Abstract Microglial activation, particularly the polarization between classical (M1 phenotype) and alternative (M2 phenotype) states, plays pivotal roles in the immune pathogenesis of Parkinson’s disease (PD), with the M1 phenotype exerting neurotoxic effects and the M2 phenotype conferring neuropro...

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Main Authors: Shumin Deng, Zhiyuan Zhang, Lu Liu, Chen Xu, Di Zhang, Lin Dong, Chunyan Gao, Xiaomin Wang, Zheng Fan
Format: Article
Language:English
Published: Nature Publishing Group 2025-04-01
Series:Cell Death Discovery
Online Access:https://doi.org/10.1038/s41420-025-02482-0
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author Shumin Deng
Zhiyuan Zhang
Lu Liu
Chen Xu
Di Zhang
Lin Dong
Chunyan Gao
Xiaomin Wang
Zheng Fan
author_facet Shumin Deng
Zhiyuan Zhang
Lu Liu
Chen Xu
Di Zhang
Lin Dong
Chunyan Gao
Xiaomin Wang
Zheng Fan
author_sort Shumin Deng
collection DOAJ
description Abstract Microglial activation, particularly the polarization between classical (M1 phenotype) and alternative (M2 phenotype) states, plays pivotal roles in the immune pathogenesis of Parkinson’s disease (PD), with the M1 phenotype exerting neurotoxic effects and the M2 phenotype conferring neuroprotection. Modulating microglial polarization toward the M2 phenotype holds therapeutic potential for PD. This study investigated the role of c-Cbl, an E3 ubiquitin ligase implicated in modulating microglial phenotypes and protecting dopaminergic neurons. Our findings revealed that c-Cbl-/- mice exhibited motor deficits, reduced striatal dopamine levels, and progressive dopaminergic neuron loss in the substantia nigra (SN). Genetic ablation of c-Cbl significantly increased proinflammatory cytokine release and microglial activation in the SN, accompanied by a phenotypic shift from M2 to M1 polarization. Furthermore, stereotaxic c-Cbl knockdown in the SN exacerbated behavioral impairments and accelerated dopaminergic neuron degeneration in the MPTP-induced mouse model of PD. At the molecular level, c-Cbl deletion promoted M1 polarization of microglia through dysregulation of the PI3K/Akt signaling pathway, thereby impairing dopaminergic neuronal survival. Collectively, this study demonstrates that c-Cbl knockout recapitulates PD-like pathology and drives microglial activation. Our results establish that c-Cbl orchestrates the transition from neurotoxic M1 to neuroprotective M2 microglial phenotypes, highlighting its central role in PD immunopathogenesis. These findings suggest c-Cbl as a promising therapeutic target for modulating microglial polarization and alleviating PD symptoms.
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spelling doaj-art-623922d3df7249a4b018af71e2f89aae2025-08-20T03:18:30ZengNature Publishing GroupCell Death Discovery2058-77162025-04-0111111210.1038/s41420-025-02482-0The E3 ligase c-Cbl modulates microglial phenotypes and contributes to Parkinson’s disease pathologyShumin Deng0Zhiyuan Zhang1Lu Liu2Chen Xu3Di Zhang4Lin Dong5Chunyan Gao6Xiaomin Wang7Zheng Fan8Department of Pharmacology, School of Basic Medical Sciences, Capital Medical UniversityDepartment of Neurobiology, School of Basic Medical Sciences, Capital Medical UniversityDepartment of Pharmacology, School of Basic Medical Sciences, Capital Medical UniversityDepartment of Pharmacology, School of Basic Medical Sciences, Capital Medical UniversityDepartment of Pharmacology, School of Basic Medical Sciences, Capital Medical UniversityDepartment of Pathology, National Cancer Center/National Clinical Research Center for Cancer/Cancer Hospital, Chinese Academy of Medical Sciences and Peking Union Medical CollegeDepartment of Clinical Medicine, Yanjing Medical College, Capital Medical UniversityDepartment of Neurobiology, School of Basic Medical Sciences, Capital Medical UniversityDepartment of Pharmacology, School of Basic Medical Sciences, Capital Medical UniversityAbstract Microglial activation, particularly the polarization between classical (M1 phenotype) and alternative (M2 phenotype) states, plays pivotal roles in the immune pathogenesis of Parkinson’s disease (PD), with the M1 phenotype exerting neurotoxic effects and the M2 phenotype conferring neuroprotection. Modulating microglial polarization toward the M2 phenotype holds therapeutic potential for PD. This study investigated the role of c-Cbl, an E3 ubiquitin ligase implicated in modulating microglial phenotypes and protecting dopaminergic neurons. Our findings revealed that c-Cbl-/- mice exhibited motor deficits, reduced striatal dopamine levels, and progressive dopaminergic neuron loss in the substantia nigra (SN). Genetic ablation of c-Cbl significantly increased proinflammatory cytokine release and microglial activation in the SN, accompanied by a phenotypic shift from M2 to M1 polarization. Furthermore, stereotaxic c-Cbl knockdown in the SN exacerbated behavioral impairments and accelerated dopaminergic neuron degeneration in the MPTP-induced mouse model of PD. At the molecular level, c-Cbl deletion promoted M1 polarization of microglia through dysregulation of the PI3K/Akt signaling pathway, thereby impairing dopaminergic neuronal survival. Collectively, this study demonstrates that c-Cbl knockout recapitulates PD-like pathology and drives microglial activation. Our results establish that c-Cbl orchestrates the transition from neurotoxic M1 to neuroprotective M2 microglial phenotypes, highlighting its central role in PD immunopathogenesis. These findings suggest c-Cbl as a promising therapeutic target for modulating microglial polarization and alleviating PD symptoms.https://doi.org/10.1038/s41420-025-02482-0
spellingShingle Shumin Deng
Zhiyuan Zhang
Lu Liu
Chen Xu
Di Zhang
Lin Dong
Chunyan Gao
Xiaomin Wang
Zheng Fan
The E3 ligase c-Cbl modulates microglial phenotypes and contributes to Parkinson’s disease pathology
Cell Death Discovery
title The E3 ligase c-Cbl modulates microglial phenotypes and contributes to Parkinson’s disease pathology
title_full The E3 ligase c-Cbl modulates microglial phenotypes and contributes to Parkinson’s disease pathology
title_fullStr The E3 ligase c-Cbl modulates microglial phenotypes and contributes to Parkinson’s disease pathology
title_full_unstemmed The E3 ligase c-Cbl modulates microglial phenotypes and contributes to Parkinson’s disease pathology
title_short The E3 ligase c-Cbl modulates microglial phenotypes and contributes to Parkinson’s disease pathology
title_sort e3 ligase c cbl modulates microglial phenotypes and contributes to parkinson s disease pathology
url https://doi.org/10.1038/s41420-025-02482-0
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