Radiation dermatitis in the hairless mouse model mimics human radiation dermatitis

Abstract Over half of all people diagnosed with cancer receive radiation therapy. Moderate to severe radiation dermatitis occurs in most human radiation patients, causing pain, aesthetic distress, and a negative impact on tumor control. No effective prevention or treatment for radiation dermatitis e...

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Main Authors: Jessica Lawrence, Davis Seelig, Kimberly Demos-Davies, Clara Ferreira, Yanan Ren, Li Wang, Sk. Kayum Alam, Rendong Yang, Alonso Guedes, Angela Craig, Luke H. Hoeppner
Format: Article
Language:English
Published: Nature Portfolio 2024-10-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-024-76021-9
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author Jessica Lawrence
Davis Seelig
Kimberly Demos-Davies
Clara Ferreira
Yanan Ren
Li Wang
Sk. Kayum Alam
Rendong Yang
Alonso Guedes
Angela Craig
Luke H. Hoeppner
author_facet Jessica Lawrence
Davis Seelig
Kimberly Demos-Davies
Clara Ferreira
Yanan Ren
Li Wang
Sk. Kayum Alam
Rendong Yang
Alonso Guedes
Angela Craig
Luke H. Hoeppner
author_sort Jessica Lawrence
collection DOAJ
description Abstract Over half of all people diagnosed with cancer receive radiation therapy. Moderate to severe radiation dermatitis occurs in most human radiation patients, causing pain, aesthetic distress, and a negative impact on tumor control. No effective prevention or treatment for radiation dermatitis exists. The lack of well-characterized, clinically relevant animal models of human radiation dermatitis contributes to the absence of strategies to mitigate radiation dermatitis. Here, we establish and characterize a hairless SKH-1 mouse model of human radiation dermatitis by correlating temporal stages of clinical and pathological skin injury. We demonstrate that a single ionizing radiation treatment of 30 Gy using 6 MeV electrons induces severe clinical grade 3 peak toxicity at 12 days, defined by marked erythema, desquamation and partial ulceration, with resolution occurring by 25 days. Histopathology reveals that radiation-induced skin injury features temporally unique inflammatory changes. Upregulation of epidermal and dermal TGF-ß1 and COX-2 protein expression occurs at peak dermatitis, with sustained epidermal TGF-ß1 expression beyond resolution. Specific histopathological variables that remain substantially high at peak toxicity and early clinical resolution, including epidermal thickening, hyperkeratosis and dermal fibroplasia/fibrosis, serve as specific measurable parameters for in vivo interventional preclinical studies that seek to mitigate radiation-induced skin injury.
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spelling doaj-art-60fa46210bd04a829af4e25c508b4ca12025-08-20T02:11:18ZengNature PortfolioScientific Reports2045-23222024-10-0114111310.1038/s41598-024-76021-9Radiation dermatitis in the hairless mouse model mimics human radiation dermatitisJessica Lawrence0Davis Seelig1Kimberly Demos-Davies2Clara Ferreira3Yanan Ren4Li Wang5Sk. Kayum Alam6Rendong Yang7Alonso Guedes8Angela Craig9Luke H. Hoeppner10Department of Veterinary Clinical Sciences, College of Veterinary Medicine, University of MinnesotaDepartment of Veterinary Clinical Sciences, College of Veterinary Medicine, University of MinnesotaDepartment of Veterinary Clinical Sciences, College of Veterinary Medicine, University of MinnesotaDepartment of Radiation Oncology, Medical School, University of MinnesotaThe Hormel Institute, University of MinnesotaThe Hormel Institute, University of MinnesotaThe Hormel Institute, University of MinnesotaThe Hormel Institute, University of MinnesotaDepartment of Veterinary Clinical Sciences, College of Veterinary Medicine, University of MinnesotaDepartment of Veterinary Clinical Sciences, College of Veterinary Medicine, University of MinnesotaMasonic Cancer Center, University of MinnesotaAbstract Over half of all people diagnosed with cancer receive radiation therapy. Moderate to severe radiation dermatitis occurs in most human radiation patients, causing pain, aesthetic distress, and a negative impact on tumor control. No effective prevention or treatment for radiation dermatitis exists. The lack of well-characterized, clinically relevant animal models of human radiation dermatitis contributes to the absence of strategies to mitigate radiation dermatitis. Here, we establish and characterize a hairless SKH-1 mouse model of human radiation dermatitis by correlating temporal stages of clinical and pathological skin injury. We demonstrate that a single ionizing radiation treatment of 30 Gy using 6 MeV electrons induces severe clinical grade 3 peak toxicity at 12 days, defined by marked erythema, desquamation and partial ulceration, with resolution occurring by 25 days. Histopathology reveals that radiation-induced skin injury features temporally unique inflammatory changes. Upregulation of epidermal and dermal TGF-ß1 and COX-2 protein expression occurs at peak dermatitis, with sustained epidermal TGF-ß1 expression beyond resolution. Specific histopathological variables that remain substantially high at peak toxicity and early clinical resolution, including epidermal thickening, hyperkeratosis and dermal fibroplasia/fibrosis, serve as specific measurable parameters for in vivo interventional preclinical studies that seek to mitigate radiation-induced skin injury.https://doi.org/10.1038/s41598-024-76021-9TranslationalDermatitisRadiationSkinInflammationTGF-ß1
spellingShingle Jessica Lawrence
Davis Seelig
Kimberly Demos-Davies
Clara Ferreira
Yanan Ren
Li Wang
Sk. Kayum Alam
Rendong Yang
Alonso Guedes
Angela Craig
Luke H. Hoeppner
Radiation dermatitis in the hairless mouse model mimics human radiation dermatitis
Scientific Reports
Translational
Dermatitis
Radiation
Skin
Inflammation
TGF-ß1
title Radiation dermatitis in the hairless mouse model mimics human radiation dermatitis
title_full Radiation dermatitis in the hairless mouse model mimics human radiation dermatitis
title_fullStr Radiation dermatitis in the hairless mouse model mimics human radiation dermatitis
title_full_unstemmed Radiation dermatitis in the hairless mouse model mimics human radiation dermatitis
title_short Radiation dermatitis in the hairless mouse model mimics human radiation dermatitis
title_sort radiation dermatitis in the hairless mouse model mimics human radiation dermatitis
topic Translational
Dermatitis
Radiation
Skin
Inflammation
TGF-ß1
url https://doi.org/10.1038/s41598-024-76021-9
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