Sex differences in respiration and redox homeostasis of heart mitochondria in rats on high-fructose diet

Sex hormones play a leading role in the sexual dimorphism of mitochondrial dysfunction and oxidative stress that are associated with Metabolic Syndrome (MetS) and considered as possible causes of cardiovascular disease. The aim of the work was to determine mitochondrial respiration and redox homeost...

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Main Authors: O. Ivanova, N. Gorbenko, O. Borikov, T. Kiprych, K. Taran
Format: Article
Language:English
Published: National Academy of Sciences of Ukraine, Palladin Institute of Biochemistry 2024-08-01
Series:The Ukrainian Biochemical Journal
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Online Access:http://ukrbiochemjournal.org/wp-content/uploads/2024/07/Ivanova_96_4.pdf
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author O. Ivanova
N. Gorbenko
O. Borikov
T. Kiprych
K. Taran
author_facet O. Ivanova
N. Gorbenko
O. Borikov
T. Kiprych
K. Taran
author_sort O. Ivanova
collection DOAJ
description Sex hormones play a leading role in the sexual dimorphism of mitochondrial dysfunction and oxidative stress that are associated with Metabolic Syndrome (MetS) and considered as possible causes of cardiovascular disease. The aim of the work was to determine mitochondrial respiration and redox homeostasis in the heart mitochondria of high-fructose diet-fed (НFD) rats depending on sex. MetS was induced in Wistar rats by 8 weeks intake of fructose (200 g/l) with drinking water. The experiment was performed on 30 rats divided into five groups: control males, control females, HFD-fed males, HFD- fed females with intact ovaries, ovariectomized HFD-fed females. Heart mitochondria were isolated and indicators of redox homeostasis as well as mitochondrial oxygen consumption rate were determined. Heart mitochondria of intact female rats were characterized by a lower intensity of lipid peroxidation, a higher activity of antioxidant defense system and state 3 respiration in comparison with control males. HFD was shown to induce more expressed oxidative stress due to significant inhibition of enzymatic and non-enzymatic components of antioxidant defese and more pronounced dysregulation of mitochondrial respiration in the heart mitochondria of ovariectomized females as compared to males. This data may partially explain the greater cardiovascular risk in women with low estrogen sufficiency and justify the necessity of new sex-specific prevention and treatment of cardiovascular risk approaches.
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spelling doaj-art-60d098fc5d23483ca8f477dc672787e12025-08-20T01:50:38ZengNational Academy of Sciences of Ukraine, Palladin Institute of BiochemistryThe Ukrainian Biochemical Journal2409-49432413-50032024-08-01964334310.15407/ubj96.04.033Sex differences in respiration and redox homeostasis of heart mitochondria in rats on high-fructose dietO. Ivanova0https://orcid.org/0000-0002-2807-8729N. Gorbenko1https://orcid.org/0000-0001-6598-730XO. Borikov2https://orcid.org/0000-0001-5776-566XT. Kiprych3https://orcid.org/0000-0002-5555-6175K. Taran4SI “V. Danilevsky Institute for Endocrine Pathology Problems of the NAMS of Ukraine”, Kharkiv, UkraineSI “V. Danilevsky Institute for Endocrine Pathology Problems of the NAMS of Ukraine”, Kharkiv, UkraineV. N. Karazin Kharkiv National University, Kharkiv, UkraineSI “V. Danilevsky Institute for Endocrine Pathology Problems of the NAMS of Ukraine”, Kharkiv, UkraineSI “V. Danilevsky Institute for Endocrine Pathology Problems of the NAMS of Ukraine”, Kharkiv, UkraineSex hormones play a leading role in the sexual dimorphism of mitochondrial dysfunction and oxidative stress that are associated with Metabolic Syndrome (MetS) and considered as possible causes of cardiovascular disease. The aim of the work was to determine mitochondrial respiration and redox homeostasis in the heart mitochondria of high-fructose diet-fed (НFD) rats depending on sex. MetS was induced in Wistar rats by 8 weeks intake of fructose (200 g/l) with drinking water. The experiment was performed on 30 rats divided into five groups: control males, control females, HFD-fed males, HFD- fed females with intact ovaries, ovariectomized HFD-fed females. Heart mitochondria were isolated and indicators of redox homeostasis as well as mitochondrial oxygen consumption rate were determined. Heart mitochondria of intact female rats were characterized by a lower intensity of lipid peroxidation, a higher activity of antioxidant defense system and state 3 respiration in comparison with control males. HFD was shown to induce more expressed oxidative stress due to significant inhibition of enzymatic and non-enzymatic components of antioxidant defese and more pronounced dysregulation of mitochondrial respiration in the heart mitochondria of ovariectomized females as compared to males. This data may partially explain the greater cardiovascular risk in women with low estrogen sufficiency and justify the necessity of new sex-specific prevention and treatment of cardiovascular risk approaches.http://ukrbiochemjournal.org/wp-content/uploads/2024/07/Ivanova_96_4.pdfantioxidant defense systemheart mitochondriamitochondrial respirationoxidative stressratssex differences
spellingShingle O. Ivanova
N. Gorbenko
O. Borikov
T. Kiprych
K. Taran
Sex differences in respiration and redox homeostasis of heart mitochondria in rats on high-fructose diet
The Ukrainian Biochemical Journal
antioxidant defense system
heart mitochondria
mitochondrial respiration
oxidative stress
rats
sex differences
title Sex differences in respiration and redox homeostasis of heart mitochondria in rats on high-fructose diet
title_full Sex differences in respiration and redox homeostasis of heart mitochondria in rats on high-fructose diet
title_fullStr Sex differences in respiration and redox homeostasis of heart mitochondria in rats on high-fructose diet
title_full_unstemmed Sex differences in respiration and redox homeostasis of heart mitochondria in rats on high-fructose diet
title_short Sex differences in respiration and redox homeostasis of heart mitochondria in rats on high-fructose diet
title_sort sex differences in respiration and redox homeostasis of heart mitochondria in rats on high fructose diet
topic antioxidant defense system
heart mitochondria
mitochondrial respiration
oxidative stress
rats
sex differences
url http://ukrbiochemjournal.org/wp-content/uploads/2024/07/Ivanova_96_4.pdf
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AT ngorbenko sexdifferencesinrespirationandredoxhomeostasisofheartmitochondriainratsonhighfructosediet
AT oborikov sexdifferencesinrespirationandredoxhomeostasisofheartmitochondriainratsonhighfructosediet
AT tkiprych sexdifferencesinrespirationandredoxhomeostasisofheartmitochondriainratsonhighfructosediet
AT ktaran sexdifferencesinrespirationandredoxhomeostasisofheartmitochondriainratsonhighfructosediet