The Hypoxia-Mimetic Agent Cobalt Chloride Differently Affects Human Mesenchymal Stem Cells in Their Chondrogenic Potential

Adult stem cells are a promising cell source for cartilage regeneration. They resided in a special microenvironment known as the stem-cell niche, characterized by the presence of low oxygen concentration. Cobalt chloride (CoCl2) imitates hypoxia in vitro by stabilizing hypoxia-inducible factor-alpha...

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Main Authors: Gabriella Teti, Stefano Focaroli, Viviana Salvatore, Eleonora Mazzotti, Laura Ingra’, Antonio Mazzotti, Mirella Falconi
Format: Article
Language:English
Published: Wiley 2018-01-01
Series:Stem Cells International
Online Access:http://dx.doi.org/10.1155/2018/3237253
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author Gabriella Teti
Stefano Focaroli
Viviana Salvatore
Eleonora Mazzotti
Laura Ingra’
Antonio Mazzotti
Mirella Falconi
author_facet Gabriella Teti
Stefano Focaroli
Viviana Salvatore
Eleonora Mazzotti
Laura Ingra’
Antonio Mazzotti
Mirella Falconi
author_sort Gabriella Teti
collection DOAJ
description Adult stem cells are a promising cell source for cartilage regeneration. They resided in a special microenvironment known as the stem-cell niche, characterized by the presence of low oxygen concentration. Cobalt chloride (CoCl2) imitates hypoxia in vitro by stabilizing hypoxia-inducible factor-alpha (HIF-1α), which is the master regulator in the cellular adaptive response to hypoxia. In this study, the influence of CoCl2 on the chondrogenic potential of human MSCs, isolated from dental pulp, umbilical cord, and adipose tissue, was investigated. Cells were treated with concentrations of CoCl2 ranging from 50 to 400 μM. Cell viability, HIF-1α protein synthesis, and the expression of the chondrogenic markers were analyzed. The results showed that the CoCl2 supplementation had no effect on cell viability, while the upregulation of chondrogenic markers such as SOX9, COL2A1, VCAN, and ACAN was dependent on the cellular source. This study shows that hypoxia, induced by CoCl2 treatment, can differently influence the behavior of MSCs, isolated from different sources, in their chondrogenic potential. These findings should be taken into consideration in the treatment of cartilage repair and regeneration based on stem cell therapies.
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institution Kabale University
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publishDate 2018-01-01
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series Stem Cells International
spelling doaj-art-5fe647e2f5a449b1bef36fa6a6c66c392025-08-20T03:34:53ZengWileyStem Cells International1687-966X1687-96782018-01-01201810.1155/2018/32372533237253The Hypoxia-Mimetic Agent Cobalt Chloride Differently Affects Human Mesenchymal Stem Cells in Their Chondrogenic PotentialGabriella Teti0Stefano Focaroli1Viviana Salvatore2Eleonora Mazzotti3Laura Ingra’4Antonio Mazzotti5Mirella Falconi6Department of Biomedical and Neuromotor Sciences, University of Bologna, Bologna, ItalyDepartment of Biomedical and Neuromotor Sciences, University of Bologna, Bologna, ItalyAn2H Discovery Limited, National Institute of Cellular Biotechnology (NICB), Dublin City University Campus, Dublin, IrelandFaculty of Comparative Biomedical Sciences, University of Teramo, Teramo, ItalyDepartment of Biomedical and Neuromotor Sciences, University of Bologna, Bologna, ItalyFirst Orthopaedic and Traumatologic Clinic, Istituto Ortopedico Rizzoli, University of Bologna, Bologna, ItalyDepartment of Biomedical and Neuromotor Sciences, University of Bologna, Bologna, ItalyAdult stem cells are a promising cell source for cartilage regeneration. They resided in a special microenvironment known as the stem-cell niche, characterized by the presence of low oxygen concentration. Cobalt chloride (CoCl2) imitates hypoxia in vitro by stabilizing hypoxia-inducible factor-alpha (HIF-1α), which is the master regulator in the cellular adaptive response to hypoxia. In this study, the influence of CoCl2 on the chondrogenic potential of human MSCs, isolated from dental pulp, umbilical cord, and adipose tissue, was investigated. Cells were treated with concentrations of CoCl2 ranging from 50 to 400 μM. Cell viability, HIF-1α protein synthesis, and the expression of the chondrogenic markers were analyzed. The results showed that the CoCl2 supplementation had no effect on cell viability, while the upregulation of chondrogenic markers such as SOX9, COL2A1, VCAN, and ACAN was dependent on the cellular source. This study shows that hypoxia, induced by CoCl2 treatment, can differently influence the behavior of MSCs, isolated from different sources, in their chondrogenic potential. These findings should be taken into consideration in the treatment of cartilage repair and regeneration based on stem cell therapies.http://dx.doi.org/10.1155/2018/3237253
spellingShingle Gabriella Teti
Stefano Focaroli
Viviana Salvatore
Eleonora Mazzotti
Laura Ingra’
Antonio Mazzotti
Mirella Falconi
The Hypoxia-Mimetic Agent Cobalt Chloride Differently Affects Human Mesenchymal Stem Cells in Their Chondrogenic Potential
Stem Cells International
title The Hypoxia-Mimetic Agent Cobalt Chloride Differently Affects Human Mesenchymal Stem Cells in Their Chondrogenic Potential
title_full The Hypoxia-Mimetic Agent Cobalt Chloride Differently Affects Human Mesenchymal Stem Cells in Their Chondrogenic Potential
title_fullStr The Hypoxia-Mimetic Agent Cobalt Chloride Differently Affects Human Mesenchymal Stem Cells in Their Chondrogenic Potential
title_full_unstemmed The Hypoxia-Mimetic Agent Cobalt Chloride Differently Affects Human Mesenchymal Stem Cells in Their Chondrogenic Potential
title_short The Hypoxia-Mimetic Agent Cobalt Chloride Differently Affects Human Mesenchymal Stem Cells in Their Chondrogenic Potential
title_sort hypoxia mimetic agent cobalt chloride differently affects human mesenchymal stem cells in their chondrogenic potential
url http://dx.doi.org/10.1155/2018/3237253
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