Excessive apoptosis, glycolysis, and abnormal levels of gluconeogenase in rheumatoid arthritis involves in the dysregulation of glucose metabolism: an animal model study

Excessive apoptosis, glycolysis, and abnormal levels of gluconeogenase in rheumatoid arthritis involves in the dysregulation of glucose metabolism: an animal model study

Rheumatoid arthritis (RA) has been associated with an elevated risk of developing disorders related to glucose metabolism, including decreased insulin secretion, impaired glucose tolerance, and type 2 diabetes mellitus. The previse mechanisms underlying this association remain incompletely elucidate...

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Bibliographic Details
Main Authors: Hui Pi, Qiang Fu, Juan Liu, Haotong Zhou, Youlian Wang
Format: Article
Language:English
Published: Taylor & Francis Group 2025-12-01
Series:Autoimmunity
Subjects:
Cell apoptosis
insulin
glucose metabolism
rheumatoid arthritis
β cell function
Online Access:https://www.tandfonline.com/doi/10.1080/08916934.2025.2499730
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