Pi, H., Fu, Q., Liu, J., Zhou, H., & Wang, Y. Excessive apoptosis, glycolysis, and abnormal levels of gluconeogenase in rheumatoid arthritis involves in the dysregulation of glucose metabolism: An animal model study. Taylor & Francis Group.
Chicago Style (17th ed.) CitationPi, Hui, Qiang Fu, Juan Liu, Haotong Zhou, and Youlian Wang. Excessive Apoptosis, Glycolysis, and Abnormal Levels of Gluconeogenase in Rheumatoid Arthritis Involves in the Dysregulation of Glucose Metabolism: An Animal Model Study. Taylor & Francis Group.
MLA (9th ed.) CitationPi, Hui, et al. Excessive Apoptosis, Glycolysis, and Abnormal Levels of Gluconeogenase in Rheumatoid Arthritis Involves in the Dysregulation of Glucose Metabolism: An Animal Model Study. Taylor & Francis Group.
Warning: These citations may not always be 100% accurate.