Effects of SHINBARO2 on Rat Models of Lumbar Spinal Stenosis

Lumbar spinal stenosis (LSS) is a major cause of chronic low back pain; however, only a few therapies which have been used in clinics still have limited effects on functional recovery. SHINBARO2 is a refined traditional formulation for inflamed lesions and relieve pain of muscular skeletal disease....

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Main Authors: So Hyun Park, Ji-Young Hong, Won Kyung Kim, Joon-Shik Shin, Jinho Lee, In-Hyuk Ha, Hwa-Jin Chung, Sang Kook Lee
Format: Article
Language:English
Published: Wiley 2019-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/2019/7651470
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author So Hyun Park
Ji-Young Hong
Won Kyung Kim
Joon-Shik Shin
Jinho Lee
In-Hyuk Ha
Hwa-Jin Chung
Sang Kook Lee
author_facet So Hyun Park
Ji-Young Hong
Won Kyung Kim
Joon-Shik Shin
Jinho Lee
In-Hyuk Ha
Hwa-Jin Chung
Sang Kook Lee
author_sort So Hyun Park
collection DOAJ
description Lumbar spinal stenosis (LSS) is a major cause of chronic low back pain; however, only a few therapies which have been used in clinics still have limited effects on functional recovery. SHINBARO2 is a refined traditional formulation for inflamed lesions and relieve pain of muscular skeletal disease. This study aimed at investigating the effects of SHINBARO2 on LSS and at determining its underlying molecular mechanism in rat models. The LSS rat models were set up by surgical operations in 6-week-old male Sprague-Dawley rats. SHINBARO2 was orally or intraperitoneally administered for 14 days. The motor and sensory ability of rats were evaluated using the activity cage and hot plate method. On the termination day, total vertebrae including the disc and spinal cord were excised for ex vivo study. SHINBARO2 improved locomotor functions and pain sensitivity in LSS rat models. Mechanism study suggested that SHINBARO2 inhibited the production of nitric oxide and prostaglandin E2 in tissues from LSS-induced rats. SHINBARO2 also suppressed the expression of proinflammatory cytokines including tumor necrosis factor-α and interleukin-1β. The activation of NF-κB by LSS surgery was effectively reduced by SHINBARO2, which coincided with the inhibition of IκB degradation. In addition, brain-derived neurotrophic factor (BDNF), a potent promoter of neurite growth, and its downstream ERK signaling were also regulated by SHINBARO2. These findings suggest that the effect of SHINBARO2 might be associated in part with the anti-inflammation and pain control in LSS rat models.
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spelling doaj-art-5ea67ef3f4264fe58e61a8b79da4e4872025-08-20T03:34:33ZengWileyMediators of Inflammation0962-93511466-18612019-01-01201910.1155/2019/76514707651470Effects of SHINBARO2 on Rat Models of Lumbar Spinal StenosisSo Hyun Park0Ji-Young Hong1Won Kyung Kim2Joon-Shik Shin3Jinho Lee4In-Hyuk Ha5Hwa-Jin Chung6Sang Kook Lee7College of Pharmacy, Natural Products Research Institute, Seoul National University, Seoul 08826, Republic of KoreaCollege of Pharmacy, Natural Products Research Institute, Seoul National University, Seoul 08826, Republic of KoreaCollege of Pharmacy, Natural Products Research Institute, Seoul National University, Seoul 08826, Republic of KoreaJaseng Spine and Joint Research Institute, Jaseng Medical Foundation, Seoul 06110, Republic of KoreaJaseng Spine and Joint Research Institute, Jaseng Medical Foundation, Seoul 06110, Republic of KoreaJaseng Spine and Joint Research Institute, Jaseng Medical Foundation, Seoul 06110, Republic of KoreaJaseng Spine and Joint Research Institute, Jaseng Medical Foundation, Seoul 06110, Republic of KoreaCollege of Pharmacy, Natural Products Research Institute, Seoul National University, Seoul 08826, Republic of KoreaLumbar spinal stenosis (LSS) is a major cause of chronic low back pain; however, only a few therapies which have been used in clinics still have limited effects on functional recovery. SHINBARO2 is a refined traditional formulation for inflamed lesions and relieve pain of muscular skeletal disease. This study aimed at investigating the effects of SHINBARO2 on LSS and at determining its underlying molecular mechanism in rat models. The LSS rat models were set up by surgical operations in 6-week-old male Sprague-Dawley rats. SHINBARO2 was orally or intraperitoneally administered for 14 days. The motor and sensory ability of rats were evaluated using the activity cage and hot plate method. On the termination day, total vertebrae including the disc and spinal cord were excised for ex vivo study. SHINBARO2 improved locomotor functions and pain sensitivity in LSS rat models. Mechanism study suggested that SHINBARO2 inhibited the production of nitric oxide and prostaglandin E2 in tissues from LSS-induced rats. SHINBARO2 also suppressed the expression of proinflammatory cytokines including tumor necrosis factor-α and interleukin-1β. The activation of NF-κB by LSS surgery was effectively reduced by SHINBARO2, which coincided with the inhibition of IκB degradation. In addition, brain-derived neurotrophic factor (BDNF), a potent promoter of neurite growth, and its downstream ERK signaling were also regulated by SHINBARO2. These findings suggest that the effect of SHINBARO2 might be associated in part with the anti-inflammation and pain control in LSS rat models.http://dx.doi.org/10.1155/2019/7651470
spellingShingle So Hyun Park
Ji-Young Hong
Won Kyung Kim
Joon-Shik Shin
Jinho Lee
In-Hyuk Ha
Hwa-Jin Chung
Sang Kook Lee
Effects of SHINBARO2 on Rat Models of Lumbar Spinal Stenosis
Mediators of Inflammation
title Effects of SHINBARO2 on Rat Models of Lumbar Spinal Stenosis
title_full Effects of SHINBARO2 on Rat Models of Lumbar Spinal Stenosis
title_fullStr Effects of SHINBARO2 on Rat Models of Lumbar Spinal Stenosis
title_full_unstemmed Effects of SHINBARO2 on Rat Models of Lumbar Spinal Stenosis
title_short Effects of SHINBARO2 on Rat Models of Lumbar Spinal Stenosis
title_sort effects of shinbaro2 on rat models of lumbar spinal stenosis
url http://dx.doi.org/10.1155/2019/7651470
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