Gonorrhea – an evolving disease of the new millennium

Etiology, transmission and protection: Neisseria gonorrhoeae (the gonococcus) is the etiological agent for the strictly human sexually transmitted disease gonorrhea. Infections lead to limited immunity, therefore individuals can become repeatedly infected. Pathology/symptomatology: Gonorrhea is gene...

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Main Authors: Stuart A. Hill, Thao L. Masters, Jenny Wachter
Format: Article
Language:English
Published: Shared Science Publishers OG 2016-09-01
Series:Microbial Cell
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Online Access:http://microbialcell.com/researcharticles/gonorrhea-an-evolving-disease-of-the-new-millennium/
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author Stuart A. Hill
Thao L. Masters
Jenny Wachter
author_facet Stuart A. Hill
Thao L. Masters
Jenny Wachter
author_sort Stuart A. Hill
collection DOAJ
description Etiology, transmission and protection: Neisseria gonorrhoeae (the gonococcus) is the etiological agent for the strictly human sexually transmitted disease gonorrhea. Infections lead to limited immunity, therefore individuals can become repeatedly infected. Pathology/symptomatology: Gonorrhea is generally a non-complicated mucosal infection with a pustular discharge. More severe sequellae include salpingitis and pelvic inflammatory disease which may lead to sterility and/or ectopic pregnancy. Occasionally, the organism can disseminate as a bloodstream infection. Epidemiology, incidence and prevalence: Gonorrhea is a global disease infecting approximately 60 million people annually. In the United States there are approximately 300, 000 cases each year, with an incidence of approximately 100 cases per 100,000 population. Treatment and curability: Gonorrhea is susceptible to an array of antibiotics. Antibiotic resistance is becoming a major problem and there are fears that the gonococcus will become the next “superbug” as the antibiotic arsenal diminishes. Currently, third generation extended-spectrum cephalosporins are being prescribed. Molecular mechanisms of infection: Gonococci elaborate numerous strategies to thwart the immune system. The organism engages in extensive phase (on/off switching) and antigenic variation of several surface antigens. The organism expresses IgA protease which cleaves mucosal antibody. The organism can become serum resistant due to its ability to sialylate lipooligosaccharide in conjunction with its ability to subvert complement activation. The gonococcus can survive within neutrophils as well as in several other lymphocytic cells. The organism manipulates the immune response such that no immune memory is generated which leads to a lack of protective immunity.
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spelling doaj-art-5d7e383f1a784e2bae5d2c3fee299ea72025-08-20T02:52:37ZengShared Science Publishers OGMicrobial Cell2311-26382016-09-013937138910.15698/mic2016.09.524Gonorrhea – an evolving disease of the new millenniumStuart A. Hill0Thao L. Masters1Jenny Wachter2Department of Biological Sciences, Northern Illinois University, DeKalb, IL 60115 USA.Department of Biological Sciences, Northern Illinois University, DeKalb, IL 60115 USA.Department of Biological Sciences, Northern Illinois University, DeKalb, IL 60115 USA.Etiology, transmission and protection: Neisseria gonorrhoeae (the gonococcus) is the etiological agent for the strictly human sexually transmitted disease gonorrhea. Infections lead to limited immunity, therefore individuals can become repeatedly infected. Pathology/symptomatology: Gonorrhea is generally a non-complicated mucosal infection with a pustular discharge. More severe sequellae include salpingitis and pelvic inflammatory disease which may lead to sterility and/or ectopic pregnancy. Occasionally, the organism can disseminate as a bloodstream infection. Epidemiology, incidence and prevalence: Gonorrhea is a global disease infecting approximately 60 million people annually. In the United States there are approximately 300, 000 cases each year, with an incidence of approximately 100 cases per 100,000 population. Treatment and curability: Gonorrhea is susceptible to an array of antibiotics. Antibiotic resistance is becoming a major problem and there are fears that the gonococcus will become the next “superbug” as the antibiotic arsenal diminishes. Currently, third generation extended-spectrum cephalosporins are being prescribed. Molecular mechanisms of infection: Gonococci elaborate numerous strategies to thwart the immune system. The organism engages in extensive phase (on/off switching) and antigenic variation of several surface antigens. The organism expresses IgA protease which cleaves mucosal antibody. The organism can become serum resistant due to its ability to sialylate lipooligosaccharide in conjunction with its ability to subvert complement activation. The gonococcus can survive within neutrophils as well as in several other lymphocytic cells. The organism manipulates the immune response such that no immune memory is generated which leads to a lack of protective immunity.http://microbialcell.com/researcharticles/gonorrhea-an-evolving-disease-of-the-new-millennium/pathogenesisantigenic variationimmune manipulationantibiotic resistancepanmictic
spellingShingle Stuart A. Hill
Thao L. Masters
Jenny Wachter
Gonorrhea – an evolving disease of the new millennium
Microbial Cell
pathogenesis
antigenic variation
immune manipulation
antibiotic resistance
panmictic
title Gonorrhea – an evolving disease of the new millennium
title_full Gonorrhea – an evolving disease of the new millennium
title_fullStr Gonorrhea – an evolving disease of the new millennium
title_full_unstemmed Gonorrhea – an evolving disease of the new millennium
title_short Gonorrhea – an evolving disease of the new millennium
title_sort gonorrhea an evolving disease of the new millennium
topic pathogenesis
antigenic variation
immune manipulation
antibiotic resistance
panmictic
url http://microbialcell.com/researcharticles/gonorrhea-an-evolving-disease-of-the-new-millennium/
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AT thaolmasters gonorrheaanevolvingdiseaseofthenewmillennium
AT jennywachter gonorrheaanevolvingdiseaseofthenewmillennium