Aminophosphinates against Helicobacter pylori ureolysis-Biochemical and whole-cell inhibition characteristics.

Urease is an important virulence factor from Helicobacter pylori that enables bacterial colonization of human gastric mucosa. Specific inhibition of urease activity can be regarded as a promising adjuvant strategy for eradication of this pathogen. A group of organophosphorus inhibitors of urease, na...

Full description

Saved in:
Bibliographic Details
Main Authors: Katarzyna Macegoniuk, Ewa Grela, Monika Biernat, Mateusz Psurski, Grażyna Gościniak, Anna Dziełak, Artur Mucha, Joanna Wietrzyk, Łukasz Berlicki, Agnieszka Grabowiecka
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2017-01-01
Series:PLoS ONE
Online Access:https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0182437&type=printable
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849765114712948736
author Katarzyna Macegoniuk
Ewa Grela
Monika Biernat
Mateusz Psurski
Grażyna Gościniak
Anna Dziełak
Artur Mucha
Joanna Wietrzyk
Łukasz Berlicki
Agnieszka Grabowiecka
author_facet Katarzyna Macegoniuk
Ewa Grela
Monika Biernat
Mateusz Psurski
Grażyna Gościniak
Anna Dziełak
Artur Mucha
Joanna Wietrzyk
Łukasz Berlicki
Agnieszka Grabowiecka
author_sort Katarzyna Macegoniuk
collection DOAJ
description Urease is an important virulence factor from Helicobacter pylori that enables bacterial colonization of human gastric mucosa. Specific inhibition of urease activity can be regarded as a promising adjuvant strategy for eradication of this pathogen. A group of organophosphorus inhibitors of urease, namely, aminophosphinic acid and aminophosphonic acid derivatives, were evaluated in vitro against H. pylori urease. The kinetic characteristics of recombinant enzyme activity demonstrated a competitive reversible mode of inhibition with Ki values ranging from 0.294 to 878 μM. N-n-Hexylaminomethyl-P-aminomethylphosphinic acid and N-methylaminomethyl-P-hydroxymethylphosphinic acid were the most effective inhibitors (Ki = 0.294 μM and 1.032 μM, respectively, compared to Ki = 23 μM for the established urease inhibitor acetohydroxamic acid). The biological relevance of the inhibitors was verified in vitro against a ureolytically active Escherichia coli Rosetta host that expressed H. pylori urease and against a reference strain, H. pylori J99 (CagA+/VacA+). The majority of the studied compounds exhibited urease-inhibiting activity in these whole-cell systems. Bis(N-methylaminomethyl)phosphinic acid was found to be the most effective inhibitor in the susceptibility profile studies of H. pylori J99. The cytotoxicity of nine structurally varied inhibitors was evaluated against four normal human cell lines and was found to be negligible.
format Article
id doaj-art-5b7119fac3d34381a359a7892f20e1ec
institution DOAJ
issn 1932-6203
language English
publishDate 2017-01-01
publisher Public Library of Science (PLoS)
record_format Article
series PLoS ONE
spelling doaj-art-5b7119fac3d34381a359a7892f20e1ec2025-08-20T03:04:57ZengPublic Library of Science (PLoS)PLoS ONE1932-62032017-01-01128e018243710.1371/journal.pone.0182437Aminophosphinates against Helicobacter pylori ureolysis-Biochemical and whole-cell inhibition characteristics.Katarzyna MacegoniukEwa GrelaMonika BiernatMateusz PsurskiGrażyna GościniakAnna DziełakArtur MuchaJoanna WietrzykŁukasz BerlickiAgnieszka GrabowieckaUrease is an important virulence factor from Helicobacter pylori that enables bacterial colonization of human gastric mucosa. Specific inhibition of urease activity can be regarded as a promising adjuvant strategy for eradication of this pathogen. A group of organophosphorus inhibitors of urease, namely, aminophosphinic acid and aminophosphonic acid derivatives, were evaluated in vitro against H. pylori urease. The kinetic characteristics of recombinant enzyme activity demonstrated a competitive reversible mode of inhibition with Ki values ranging from 0.294 to 878 μM. N-n-Hexylaminomethyl-P-aminomethylphosphinic acid and N-methylaminomethyl-P-hydroxymethylphosphinic acid were the most effective inhibitors (Ki = 0.294 μM and 1.032 μM, respectively, compared to Ki = 23 μM for the established urease inhibitor acetohydroxamic acid). The biological relevance of the inhibitors was verified in vitro against a ureolytically active Escherichia coli Rosetta host that expressed H. pylori urease and against a reference strain, H. pylori J99 (CagA+/VacA+). The majority of the studied compounds exhibited urease-inhibiting activity in these whole-cell systems. Bis(N-methylaminomethyl)phosphinic acid was found to be the most effective inhibitor in the susceptibility profile studies of H. pylori J99. The cytotoxicity of nine structurally varied inhibitors was evaluated against four normal human cell lines and was found to be negligible.https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0182437&type=printable
spellingShingle Katarzyna Macegoniuk
Ewa Grela
Monika Biernat
Mateusz Psurski
Grażyna Gościniak
Anna Dziełak
Artur Mucha
Joanna Wietrzyk
Łukasz Berlicki
Agnieszka Grabowiecka
Aminophosphinates against Helicobacter pylori ureolysis-Biochemical and whole-cell inhibition characteristics.
PLoS ONE
title Aminophosphinates against Helicobacter pylori ureolysis-Biochemical and whole-cell inhibition characteristics.
title_full Aminophosphinates against Helicobacter pylori ureolysis-Biochemical and whole-cell inhibition characteristics.
title_fullStr Aminophosphinates against Helicobacter pylori ureolysis-Biochemical and whole-cell inhibition characteristics.
title_full_unstemmed Aminophosphinates against Helicobacter pylori ureolysis-Biochemical and whole-cell inhibition characteristics.
title_short Aminophosphinates against Helicobacter pylori ureolysis-Biochemical and whole-cell inhibition characteristics.
title_sort aminophosphinates against helicobacter pylori ureolysis biochemical and whole cell inhibition characteristics
url https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0182437&type=printable
work_keys_str_mv AT katarzynamacegoniuk aminophosphinatesagainsthelicobacterpyloriureolysisbiochemicalandwholecellinhibitioncharacteristics
AT ewagrela aminophosphinatesagainsthelicobacterpyloriureolysisbiochemicalandwholecellinhibitioncharacteristics
AT monikabiernat aminophosphinatesagainsthelicobacterpyloriureolysisbiochemicalandwholecellinhibitioncharacteristics
AT mateuszpsurski aminophosphinatesagainsthelicobacterpyloriureolysisbiochemicalandwholecellinhibitioncharacteristics
AT grazynagosciniak aminophosphinatesagainsthelicobacterpyloriureolysisbiochemicalandwholecellinhibitioncharacteristics
AT annadziełak aminophosphinatesagainsthelicobacterpyloriureolysisbiochemicalandwholecellinhibitioncharacteristics
AT arturmucha aminophosphinatesagainsthelicobacterpyloriureolysisbiochemicalandwholecellinhibitioncharacteristics
AT joannawietrzyk aminophosphinatesagainsthelicobacterpyloriureolysisbiochemicalandwholecellinhibitioncharacteristics
AT łukaszberlicki aminophosphinatesagainsthelicobacterpyloriureolysisbiochemicalandwholecellinhibitioncharacteristics
AT agnieszkagrabowiecka aminophosphinatesagainsthelicobacterpyloriureolysisbiochemicalandwholecellinhibitioncharacteristics