Identification of Candidate Tolerogenic CD8+ T Cell Epitopes for Therapy of Type 1 Diabetes in the NOD Mouse Model

Type 1 diabetes is an autoimmune disease in which insulin-producing pancreatic islet β cells are the target of self-reactive B and T cells. T cells reactive with epitopes derived from insulin and/or IGRP are critical for the initiation and maintenance of disease, but T cells reactive with other isle...

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Main Authors: Cailin Yu, Jeremy C. Burns, William H. Robinson, Paul J. Utz, Peggy P. Ho, Lawrence Steinman, Alan B. Frey
Format: Article
Language:English
Published: Wiley 2016-01-01
Series:Journal of Diabetes Research
Online Access:http://dx.doi.org/10.1155/2016/9083103
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author Cailin Yu
Jeremy C. Burns
William H. Robinson
Paul J. Utz
Peggy P. Ho
Lawrence Steinman
Alan B. Frey
author_facet Cailin Yu
Jeremy C. Burns
William H. Robinson
Paul J. Utz
Peggy P. Ho
Lawrence Steinman
Alan B. Frey
author_sort Cailin Yu
collection DOAJ
description Type 1 diabetes is an autoimmune disease in which insulin-producing pancreatic islet β cells are the target of self-reactive B and T cells. T cells reactive with epitopes derived from insulin and/or IGRP are critical for the initiation and maintenance of disease, but T cells reactive with other islet antigens likely have an essential role in disease progression. We sought to identify candidate CD8+ T cell epitopes that are pathogenic in type 1 diabetes. Proteins that elicit autoantibodies in human type 1 diabetes were analyzed by predictive algorithms for candidate epitopes. Using several different tolerizing regimes using synthetic peptides, two new predicted tolerogenic CD8+ T cell epitopes were identified in the murine homolog of the major human islet autoantigen zinc transporter ZnT8 (aa 158–166 and 282–290) and one in a non-β cell protein, dopamine β-hydroxylase (aa 233–241). Tolerizing vaccination of NOD mice with a cDNA plasmid expressing full-length proinsulin prevented diabetes, whereas plasmids encoding ZnT8 and DβH did not. However, tolerizing vaccination of NOD mice with the proinsulin plasmid in combination with plasmids expressing ZnT8 and DβH decreased insulitis and enhanced prevention of disease compared to vaccination with the plasmid encoding proinsulin alone.
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issn 2314-6745
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publishDate 2016-01-01
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series Journal of Diabetes Research
spelling doaj-art-5a4a980daeee4972aa73e3daa632d7232025-08-20T02:05:28ZengWileyJournal of Diabetes Research2314-67452314-67532016-01-01201610.1155/2016/90831039083103Identification of Candidate Tolerogenic CD8+ T Cell Epitopes for Therapy of Type 1 Diabetes in the NOD Mouse ModelCailin Yu0Jeremy C. Burns1William H. Robinson2Paul J. Utz3Peggy P. Ho4Lawrence Steinman5Alan B. Frey6Department of Cell Biology, New York University Langone School of Medicine, 550 First Avenue, New York, NY 10016, USADepartment of Cell Biology, New York University Langone School of Medicine, 550 First Avenue, New York, NY 10016, USADivision of Immunology and Rheumatology, Department of Medicine, Stanford University School of Medicine, Stanford, CA 94305, USADivision of Immunology and Rheumatology, Department of Medicine, Stanford University School of Medicine, Stanford, CA 94305, USADepartment of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA 94305, USADepartment of Neurology and Neurological Sciences, Stanford University School of Medicine, Stanford, CA 94305, USADepartment of Cell Biology, New York University Langone School of Medicine, 550 First Avenue, New York, NY 10016, USAType 1 diabetes is an autoimmune disease in which insulin-producing pancreatic islet β cells are the target of self-reactive B and T cells. T cells reactive with epitopes derived from insulin and/or IGRP are critical for the initiation and maintenance of disease, but T cells reactive with other islet antigens likely have an essential role in disease progression. We sought to identify candidate CD8+ T cell epitopes that are pathogenic in type 1 diabetes. Proteins that elicit autoantibodies in human type 1 diabetes were analyzed by predictive algorithms for candidate epitopes. Using several different tolerizing regimes using synthetic peptides, two new predicted tolerogenic CD8+ T cell epitopes were identified in the murine homolog of the major human islet autoantigen zinc transporter ZnT8 (aa 158–166 and 282–290) and one in a non-β cell protein, dopamine β-hydroxylase (aa 233–241). Tolerizing vaccination of NOD mice with a cDNA plasmid expressing full-length proinsulin prevented diabetes, whereas plasmids encoding ZnT8 and DβH did not. However, tolerizing vaccination of NOD mice with the proinsulin plasmid in combination with plasmids expressing ZnT8 and DβH decreased insulitis and enhanced prevention of disease compared to vaccination with the plasmid encoding proinsulin alone.http://dx.doi.org/10.1155/2016/9083103
spellingShingle Cailin Yu
Jeremy C. Burns
William H. Robinson
Paul J. Utz
Peggy P. Ho
Lawrence Steinman
Alan B. Frey
Identification of Candidate Tolerogenic CD8+ T Cell Epitopes for Therapy of Type 1 Diabetes in the NOD Mouse Model
Journal of Diabetes Research
title Identification of Candidate Tolerogenic CD8+ T Cell Epitopes for Therapy of Type 1 Diabetes in the NOD Mouse Model
title_full Identification of Candidate Tolerogenic CD8+ T Cell Epitopes for Therapy of Type 1 Diabetes in the NOD Mouse Model
title_fullStr Identification of Candidate Tolerogenic CD8+ T Cell Epitopes for Therapy of Type 1 Diabetes in the NOD Mouse Model
title_full_unstemmed Identification of Candidate Tolerogenic CD8+ T Cell Epitopes for Therapy of Type 1 Diabetes in the NOD Mouse Model
title_short Identification of Candidate Tolerogenic CD8+ T Cell Epitopes for Therapy of Type 1 Diabetes in the NOD Mouse Model
title_sort identification of candidate tolerogenic cd8 t cell epitopes for therapy of type 1 diabetes in the nod mouse model
url http://dx.doi.org/10.1155/2016/9083103
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