Short term doxycycline treatment induces sustained improvement in myocardial infarction border zone contractility.

Decreased contractility in the non-ischemic border zone surrounding a MI is in part due to degradation of cardiomyocyte sarcomeric components by intracellular matrix metalloproteinase-2 (MMP-2). We recently reported that MMP-2 levels were increased in the border zone after a MI and that treatment wi...

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Main Authors: Kimberly Spaulding, Kiyoaki Takaba, Alexander Collins, Farshid Faraji, Guanying Wang, Esteban Aguayo, Liang Ge, David Saloner, Arthur W Wallace, Anthony J Baker, David H Lovett, Mark B Ratcliffe
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2018-01-01
Series:PLoS ONE
Online Access:https://doi.org/10.1371/journal.pone.0192720
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author Kimberly Spaulding
Kiyoaki Takaba
Alexander Collins
Farshid Faraji
Guanying Wang
Esteban Aguayo
Liang Ge
David Saloner
Arthur W Wallace
Anthony J Baker
David H Lovett
Mark B Ratcliffe
author_facet Kimberly Spaulding
Kiyoaki Takaba
Alexander Collins
Farshid Faraji
Guanying Wang
Esteban Aguayo
Liang Ge
David Saloner
Arthur W Wallace
Anthony J Baker
David H Lovett
Mark B Ratcliffe
author_sort Kimberly Spaulding
collection DOAJ
description Decreased contractility in the non-ischemic border zone surrounding a MI is in part due to degradation of cardiomyocyte sarcomeric components by intracellular matrix metalloproteinase-2 (MMP-2). We recently reported that MMP-2 levels were increased in the border zone after a MI and that treatment with doxycycline for two weeks after MI was associated with normalization of MMP-2 levels and improvement in ex-vivo contractile protein developed force in the myocardial border zone. The purpose of the current study was to determine if there is a sustained effect of short term treatment with doxycycline (Dox) on border zone function in a large animal model of antero-apical myocardial infarction (MI). Antero-apical MI was created in 14 sheep. Seven sheep received doxycycline 0.8 mg/kg/hr IV for two weeks. Cardiac MRI was performed two weeks before, and then two and six weeks after MI. Two sheep died prior to MRI at six weeks from surgical/anesthesia-related causes. The remaining 12 sheep completed the protocol. Doxycycline induced a sustained reduction in intracellular MMP-2 by Western blot (3649±643 MI+Dox vs 9236±114 MI relative intensity; p = 0.0009), an improvement in ex-vivo contractility (65.3±2.0 MI+Dox vs 39.7±0.8 MI mN/mm2; p<0.0001) and an increase in ventricular wall thickness at end-systole 1.0 cm from the infarct edge (12.4±0.6 MI+Dox vs 10.0±0.5 MI mm; p = 0.0095). Administration of doxycycline for a limited two week period is associated with a sustained improvement in ex-vivo contractility and an increase in wall thickness at end-systole in the border zone six weeks after MI. These findings were associated with a reduction in intracellular MMP-2 activity.
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spelling doaj-art-56f8bd079baf4cbf975c5ead44a216f42025-08-20T02:36:18ZengPublic Library of Science (PLoS)PLoS ONE1932-62032018-01-01132e019272010.1371/journal.pone.0192720Short term doxycycline treatment induces sustained improvement in myocardial infarction border zone contractility.Kimberly SpauldingKiyoaki TakabaAlexander CollinsFarshid FarajiGuanying WangEsteban AguayoLiang GeDavid SalonerArthur W WallaceAnthony J BakerDavid H LovettMark B RatcliffeDecreased contractility in the non-ischemic border zone surrounding a MI is in part due to degradation of cardiomyocyte sarcomeric components by intracellular matrix metalloproteinase-2 (MMP-2). We recently reported that MMP-2 levels were increased in the border zone after a MI and that treatment with doxycycline for two weeks after MI was associated with normalization of MMP-2 levels and improvement in ex-vivo contractile protein developed force in the myocardial border zone. The purpose of the current study was to determine if there is a sustained effect of short term treatment with doxycycline (Dox) on border zone function in a large animal model of antero-apical myocardial infarction (MI). Antero-apical MI was created in 14 sheep. Seven sheep received doxycycline 0.8 mg/kg/hr IV for two weeks. Cardiac MRI was performed two weeks before, and then two and six weeks after MI. Two sheep died prior to MRI at six weeks from surgical/anesthesia-related causes. The remaining 12 sheep completed the protocol. Doxycycline induced a sustained reduction in intracellular MMP-2 by Western blot (3649±643 MI+Dox vs 9236±114 MI relative intensity; p = 0.0009), an improvement in ex-vivo contractility (65.3±2.0 MI+Dox vs 39.7±0.8 MI mN/mm2; p<0.0001) and an increase in ventricular wall thickness at end-systole 1.0 cm from the infarct edge (12.4±0.6 MI+Dox vs 10.0±0.5 MI mm; p = 0.0095). Administration of doxycycline for a limited two week period is associated with a sustained improvement in ex-vivo contractility and an increase in wall thickness at end-systole in the border zone six weeks after MI. These findings were associated with a reduction in intracellular MMP-2 activity.https://doi.org/10.1371/journal.pone.0192720
spellingShingle Kimberly Spaulding
Kiyoaki Takaba
Alexander Collins
Farshid Faraji
Guanying Wang
Esteban Aguayo
Liang Ge
David Saloner
Arthur W Wallace
Anthony J Baker
David H Lovett
Mark B Ratcliffe
Short term doxycycline treatment induces sustained improvement in myocardial infarction border zone contractility.
PLoS ONE
title Short term doxycycline treatment induces sustained improvement in myocardial infarction border zone contractility.
title_full Short term doxycycline treatment induces sustained improvement in myocardial infarction border zone contractility.
title_fullStr Short term doxycycline treatment induces sustained improvement in myocardial infarction border zone contractility.
title_full_unstemmed Short term doxycycline treatment induces sustained improvement in myocardial infarction border zone contractility.
title_short Short term doxycycline treatment induces sustained improvement in myocardial infarction border zone contractility.
title_sort short term doxycycline treatment induces sustained improvement in myocardial infarction border zone contractility
url https://doi.org/10.1371/journal.pone.0192720
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