Wnt5a promotes VM formation by modulating the stemness and EMT progression of prostate cancer cell

The incidence of prostate cancer (PCa) is increasing annually, making it the leading cause of tumor-related mortality in males. The available treatment options for metastatic PCa are limited. Vasculogenic mimicry (VM), an emerging phenomenon involving aggressive tumor cells, has a significant impact...

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Main Authors: Bide Liu, Xun Li, Shuheng Wang, Hongliang Jia, Xiaoan Zhang, Qiang Dong, Jiuzhi Li
Format: Article
Language:English
Published: Elsevier 2025-01-01
Series:Translational Oncology
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Online Access:http://www.sciencedirect.com/science/article/pii/S1936523324002821
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author Bide Liu
Xun Li
Shuheng Wang
Hongliang Jia
Xiaoan Zhang
Qiang Dong
Jiuzhi Li
author_facet Bide Liu
Xun Li
Shuheng Wang
Hongliang Jia
Xiaoan Zhang
Qiang Dong
Jiuzhi Li
author_sort Bide Liu
collection DOAJ
description The incidence of prostate cancer (PCa) is increasing annually, making it the leading cause of tumor-related mortality in males. The available treatment options for metastatic PCa are limited. Vasculogenic mimicry (VM), an emerging phenomenon involving aggressive tumor cells, has a significant impact on patient survival. Misregulation of Wnt5a expression is commonly observed during cancer progression. However, there is a lack of comprehensive studies investigating the effects of Wnt5a on tumor VM formation. In this study, we demonstrate that alterations in wnt5a expression, either through gain or loss, have a significant influence on the formation of VM in tumor cells mediated by cell stemness and EMT progression. Further research has demonstrated that Wnt5a regulates the formation of VM through the PI3K/JNK signaling pathway. These experimental findings offer a novel avenue for the clinical management of prostate cancer.
format Article
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institution OA Journals
issn 1936-5233
language English
publishDate 2025-01-01
publisher Elsevier
record_format Article
series Translational Oncology
spelling doaj-art-56cb46699ea84fb18ea4062a034826592025-08-20T02:30:39ZengElsevierTranslational Oncology1936-52332025-01-015110215510.1016/j.tranon.2024.102155Wnt5a promotes VM formation by modulating the stemness and EMT progression of prostate cancer cellBide Liu0Xun Li1Shuheng Wang2Hongliang Jia3Xiaoan Zhang4Qiang Dong5Jiuzhi Li6Department of Urology, People's Hospital of Xinjiang Uygur Autonomous Region, Urumqi, 830001, China; Laboratory of Urology, People's Hospital of Xinjiang Uygur Autonomous Region, Urumqi, 830001, ChinaDepartment of Urology, People's Hospital of Xinjiang Uygur Autonomous Region, Urumqi, 830001, China; Laboratory of Urology, People's Hospital of Xinjiang Uygur Autonomous Region, Urumqi, 830001, ChinaDepartment of Urology, People's Hospital of Xinjiang Uygur Autonomous Region, Urumqi, 830001, China; Laboratory of Urology, People's Hospital of Xinjiang Uygur Autonomous Region, Urumqi, 830001, ChinaDepartment of Urology, People's Hospital of Xinjiang Uygur Autonomous Region, Urumqi, 830001, China; Laboratory of Urology, People's Hospital of Xinjiang Uygur Autonomous Region, Urumqi, 830001, ChinaDepartment of Urology, People's Hospital of Xinjiang Uygur Autonomous Region, Urumqi, 830001, China; Laboratory of Urology, People's Hospital of Xinjiang Uygur Autonomous Region, Urumqi, 830001, ChinaDepartment of Urology, People's Hospital of Xinjiang Uygur Autonomous Region, Urumqi, 830001, China; Laboratory of Urology, People's Hospital of Xinjiang Uygur Autonomous Region, Urumqi, 830001, ChinaDepartment of Urology, People's Hospital of Xinjiang Uygur Autonomous Region, Urumqi, 830001, China; Laboratory of Urology, People's Hospital of Xinjiang Uygur Autonomous Region, Urumqi, 830001, China; Corresponding author at: Department of Urology, People's Hospital of Xinjiang Uygur Autonomous Region, Urumqi, 830001, China.The incidence of prostate cancer (PCa) is increasing annually, making it the leading cause of tumor-related mortality in males. The available treatment options for metastatic PCa are limited. Vasculogenic mimicry (VM), an emerging phenomenon involving aggressive tumor cells, has a significant impact on patient survival. Misregulation of Wnt5a expression is commonly observed during cancer progression. However, there is a lack of comprehensive studies investigating the effects of Wnt5a on tumor VM formation. In this study, we demonstrate that alterations in wnt5a expression, either through gain or loss, have a significant influence on the formation of VM in tumor cells mediated by cell stemness and EMT progression. Further research has demonstrated that Wnt5a regulates the formation of VM through the PI3K/JNK signaling pathway. These experimental findings offer a novel avenue for the clinical management of prostate cancer.http://www.sciencedirect.com/science/article/pii/S1936523324002821Cell stemnessEMTJNK signalingProstate cancerVasculogenic mimicryWnt5a
spellingShingle Bide Liu
Xun Li
Shuheng Wang
Hongliang Jia
Xiaoan Zhang
Qiang Dong
Jiuzhi Li
Wnt5a promotes VM formation by modulating the stemness and EMT progression of prostate cancer cell
Translational Oncology
Cell stemness
EMT
JNK signaling
Prostate cancer
Vasculogenic mimicry
Wnt5a
title Wnt5a promotes VM formation by modulating the stemness and EMT progression of prostate cancer cell
title_full Wnt5a promotes VM formation by modulating the stemness and EMT progression of prostate cancer cell
title_fullStr Wnt5a promotes VM formation by modulating the stemness and EMT progression of prostate cancer cell
title_full_unstemmed Wnt5a promotes VM formation by modulating the stemness and EMT progression of prostate cancer cell
title_short Wnt5a promotes VM formation by modulating the stemness and EMT progression of prostate cancer cell
title_sort wnt5a promotes vm formation by modulating the stemness and emt progression of prostate cancer cell
topic Cell stemness
EMT
JNK signaling
Prostate cancer
Vasculogenic mimicry
Wnt5a
url http://www.sciencedirect.com/science/article/pii/S1936523324002821
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