Overexpression of the Oncogenic Variant (KLF6-SV1) in Young NPC Patients and Correlation with Lack of E-Cadherin

Purpose. The transcription factor Krüppel-like factor 6 (KLF6) regulates various cellular functions, such as metabolism, cell proliferation, and differentiation. KLF6 plays a key role in the development and progression of multiple human cancers. Methods. Fifty primary biopsies and 10 normal nasophar...

Full description

Saved in:
Bibliographic Details
Main Authors: Saoussen Debouki-Joudi, Sonia Mhirsi, Nihel Ammous-Boukhris, Nehla Mokni-Baizig, Hayet Mhamdi, Said Gritli, Raja Mokdad Gargouri, Mohamed Nejib Marzouki
Format: Article
Language:English
Published: Wiley 2018-01-01
Series:Analytical Cellular Pathology
Online Access:http://dx.doi.org/10.1155/2018/9654067
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850226294874177536
author Saoussen Debouki-Joudi
Sonia Mhirsi
Nihel Ammous-Boukhris
Nehla Mokni-Baizig
Hayet Mhamdi
Said Gritli
Raja Mokdad Gargouri
Mohamed Nejib Marzouki
author_facet Saoussen Debouki-Joudi
Sonia Mhirsi
Nihel Ammous-Boukhris
Nehla Mokni-Baizig
Hayet Mhamdi
Said Gritli
Raja Mokdad Gargouri
Mohamed Nejib Marzouki
author_sort Saoussen Debouki-Joudi
collection DOAJ
description Purpose. The transcription factor Krüppel-like factor 6 (KLF6) regulates various cellular functions, such as metabolism, cell proliferation, and differentiation. KLF6 plays a key role in the development and progression of multiple human cancers. Methods. Fifty primary biopsies and 10 normal nasopharyngeal mucosae were used to analyze by RT-QPCR the expression and the copy number of wtKLF6 and the spliced variants (KLF6-SV1, KLF6-SV2, and KLF6-SV3) in Tunisian patients with nasopharyngeal carcinoma. The expression analysis of E-cadherin and cyclin D1 was conducted by RT-QPCR and Western blot, respectively. Results. The wtKLF6 was significantly downexpressed in tumors compared to normal tissues (p=0.0015), whereas KLF6-SV1 and KLF6-SV2 were overexpressed in tumors compared to wtKLF6 and KLF6-SV3 (p<0.0001). Copy number variation was reduced in tumors compared to normal tissues (p=0.0071). Interestingly, KLF6-SV1 is associated with the juvenile form (p=0.0003) which is more aggressive than the adult form of NPC. Furthermore, the oncogenic variant KLF6-SV1 was overexpressed in tumors lacking the expression of E-cadherin (p=0.0022) suggesting its role in metastasis and tumor progression. The wtKLF6 is associated negatively with cyclin D1 in tumor tissues (p=0.048). Conclusion. The wtKLF6 was downexpressed in contrast with the oncogenic variants. Overexpression of KLF6-SV1 is associated with young patients, and loss of E-cadherin suggests that this variant correlated with the aggressiveness of NPC.
format Article
id doaj-art-551f8e94fcac45abb362c26b005b2177
institution OA Journals
issn 2210-7177
2210-7185
language English
publishDate 2018-01-01
publisher Wiley
record_format Article
series Analytical Cellular Pathology
spelling doaj-art-551f8e94fcac45abb362c26b005b21772025-08-20T02:05:07ZengWileyAnalytical Cellular Pathology2210-71772210-71852018-01-01201810.1155/2018/96540679654067Overexpression of the Oncogenic Variant (KLF6-SV1) in Young NPC Patients and Correlation with Lack of E-CadherinSaoussen Debouki-Joudi0Sonia Mhirsi1Nihel Ammous-Boukhris2Nehla Mokni-Baizig3Hayet Mhamdi4Said Gritli5Raja Mokdad Gargouri6Mohamed Nejib Marzouki7Laboratoire d'Ingénierie des Protéines et des Molécules Bioactives, Institut National des Sciences Appliquées et de Technologie (INSAT), Tunis, TunisiaLaboratoire d'Ingénierie des Protéines et des Molécules Bioactives, Institut National des Sciences Appliquées et de Technologie (INSAT), Tunis, TunisiaLaboratoire de Biotechnologie Moléculaire des Eucaryotes, Centre de Biotechnologie de Sfax, Université de Sfax, Sfax, TunisiaInstitut de Carcinologie Salah Azaiz, Tunis, TunisiaInstitut de Carcinologie Salah Azaiz, Tunis, TunisiaInstitut de Carcinologie Salah Azaiz, Tunis, TunisiaLaboratoire de Biotechnologie Moléculaire des Eucaryotes, Centre de Biotechnologie de Sfax, Université de Sfax, Sfax, TunisiaLaboratoire d'Ingénierie des Protéines et des Molécules Bioactives, Institut National des Sciences Appliquées et de Technologie (INSAT), Tunis, TunisiaPurpose. The transcription factor Krüppel-like factor 6 (KLF6) regulates various cellular functions, such as metabolism, cell proliferation, and differentiation. KLF6 plays a key role in the development and progression of multiple human cancers. Methods. Fifty primary biopsies and 10 normal nasopharyngeal mucosae were used to analyze by RT-QPCR the expression and the copy number of wtKLF6 and the spliced variants (KLF6-SV1, KLF6-SV2, and KLF6-SV3) in Tunisian patients with nasopharyngeal carcinoma. The expression analysis of E-cadherin and cyclin D1 was conducted by RT-QPCR and Western blot, respectively. Results. The wtKLF6 was significantly downexpressed in tumors compared to normal tissues (p=0.0015), whereas KLF6-SV1 and KLF6-SV2 were overexpressed in tumors compared to wtKLF6 and KLF6-SV3 (p<0.0001). Copy number variation was reduced in tumors compared to normal tissues (p=0.0071). Interestingly, KLF6-SV1 is associated with the juvenile form (p=0.0003) which is more aggressive than the adult form of NPC. Furthermore, the oncogenic variant KLF6-SV1 was overexpressed in tumors lacking the expression of E-cadherin (p=0.0022) suggesting its role in metastasis and tumor progression. The wtKLF6 is associated negatively with cyclin D1 in tumor tissues (p=0.048). Conclusion. The wtKLF6 was downexpressed in contrast with the oncogenic variants. Overexpression of KLF6-SV1 is associated with young patients, and loss of E-cadherin suggests that this variant correlated with the aggressiveness of NPC.http://dx.doi.org/10.1155/2018/9654067
spellingShingle Saoussen Debouki-Joudi
Sonia Mhirsi
Nihel Ammous-Boukhris
Nehla Mokni-Baizig
Hayet Mhamdi
Said Gritli
Raja Mokdad Gargouri
Mohamed Nejib Marzouki
Overexpression of the Oncogenic Variant (KLF6-SV1) in Young NPC Patients and Correlation with Lack of E-Cadherin
Analytical Cellular Pathology
title Overexpression of the Oncogenic Variant (KLF6-SV1) in Young NPC Patients and Correlation with Lack of E-Cadherin
title_full Overexpression of the Oncogenic Variant (KLF6-SV1) in Young NPC Patients and Correlation with Lack of E-Cadherin
title_fullStr Overexpression of the Oncogenic Variant (KLF6-SV1) in Young NPC Patients and Correlation with Lack of E-Cadherin
title_full_unstemmed Overexpression of the Oncogenic Variant (KLF6-SV1) in Young NPC Patients and Correlation with Lack of E-Cadherin
title_short Overexpression of the Oncogenic Variant (KLF6-SV1) in Young NPC Patients and Correlation with Lack of E-Cadherin
title_sort overexpression of the oncogenic variant klf6 sv1 in young npc patients and correlation with lack of e cadherin
url http://dx.doi.org/10.1155/2018/9654067
work_keys_str_mv AT saoussendeboukijoudi overexpressionoftheoncogenicvariantklf6sv1inyoungnpcpatientsandcorrelationwithlackofecadherin
AT soniamhirsi overexpressionoftheoncogenicvariantklf6sv1inyoungnpcpatientsandcorrelationwithlackofecadherin
AT nihelammousboukhris overexpressionoftheoncogenicvariantklf6sv1inyoungnpcpatientsandcorrelationwithlackofecadherin
AT nehlamoknibaizig overexpressionoftheoncogenicvariantklf6sv1inyoungnpcpatientsandcorrelationwithlackofecadherin
AT hayetmhamdi overexpressionoftheoncogenicvariantklf6sv1inyoungnpcpatientsandcorrelationwithlackofecadherin
AT saidgritli overexpressionoftheoncogenicvariantklf6sv1inyoungnpcpatientsandcorrelationwithlackofecadherin
AT rajamokdadgargouri overexpressionoftheoncogenicvariantklf6sv1inyoungnpcpatientsandcorrelationwithlackofecadherin
AT mohamednejibmarzouki overexpressionoftheoncogenicvariantklf6sv1inyoungnpcpatientsandcorrelationwithlackofecadherin