Exposure of Fluoride with Streptozotocin-Induced Diabetes Aggravates Testicular Damage and Spermatozoa Parameters in Mice

Diabetes mellitus is the most common chronic disease worldwide that causes numerous complications, including male infertility. The prevalence of DM is 451 million people and estimated that would increase to 693 million in 2045. Fluorosis caused by drinking water contaminated with inorganic fluoride...

Full description

Saved in:
Bibliographic Details
Main Authors: Manuel Sánchez-Gutiérrez, Evelia Martínez-Loredo, Eduardo Osiris Madrigal-Santillán, Gabriel Betanzos-Cabrera, Araceli Hernández-Zavala, María Angélica Mojica-Villegas, Jeannett Alejandra Izquierdo-Vega
Format: Article
Language:English
Published: Wiley 2019-01-01
Series:Journal of Toxicology
Online Access:http://dx.doi.org/10.1155/2019/5269380
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1849412803526393856
author Manuel Sánchez-Gutiérrez
Evelia Martínez-Loredo
Eduardo Osiris Madrigal-Santillán
Gabriel Betanzos-Cabrera
Araceli Hernández-Zavala
María Angélica Mojica-Villegas
Jeannett Alejandra Izquierdo-Vega
author_facet Manuel Sánchez-Gutiérrez
Evelia Martínez-Loredo
Eduardo Osiris Madrigal-Santillán
Gabriel Betanzos-Cabrera
Araceli Hernández-Zavala
María Angélica Mojica-Villegas
Jeannett Alejandra Izquierdo-Vega
author_sort Manuel Sánchez-Gutiérrez
collection DOAJ
description Diabetes mellitus is the most common chronic disease worldwide that causes numerous complications, including male infertility. The prevalence of DM is 451 million people and estimated that would increase to 693 million in 2045. Fluorosis caused by drinking water contaminated with inorganic fluoride is a public health problem in many areas around the world. Previous studies have shown that fluoride exposure damages the male reproductive function. This study aimed to evaluate the fluoride sub-chronic exposure on the spermatozoa function in streptozotocin (STZ)-induced diabetic mice. After confirming diabetes by measuring blood glucose levels, the male mice received 45.2 ppm of fluoride added or deionized water. We evaluated several parameters in diabetic mice exposed to fluoride: standard quality analysis, the mitochondrial transmembrane potential (ψm), the caspase activity in spermatozoa, urinary fluoride excretion, and histological evaluation in the testes. After 60 days of fluoride-exposure, diabetic mice, significantly decreased sperm quality (motility, viability, and concentration). Spermatozoa from fluoride-exposure in diabetic mice presented a significant decrease in ψm and a significant increase in activity caspase 3/7. Urinary fluoride excretion was decreased in diabetic mice exposed to fluoride. Subchronic fluoride exposure of mice with STZ-induced diabetes aggravated testicular damage and the spermatozoa function.
format Article
id doaj-art-549aa44de01d4cd5b558e0e5afe5b5b9
institution Kabale University
issn 1687-8191
1687-8205
language English
publishDate 2019-01-01
publisher Wiley
record_format Article
series Journal of Toxicology
spelling doaj-art-549aa44de01d4cd5b558e0e5afe5b5b92025-08-20T03:34:20ZengWileyJournal of Toxicology1687-81911687-82052019-01-01201910.1155/2019/52693805269380Exposure of Fluoride with Streptozotocin-Induced Diabetes Aggravates Testicular Damage and Spermatozoa Parameters in MiceManuel Sánchez-Gutiérrez0Evelia Martínez-Loredo1Eduardo Osiris Madrigal-Santillán2Gabriel Betanzos-Cabrera3Araceli Hernández-Zavala4María Angélica Mojica-Villegas5Jeannett Alejandra Izquierdo-Vega6Área Académica de Medicina, Instituto de Ciencias de la Salud, Universidad Autonoma del Estado de Hidalgo, Carretera Actopan-Tilcuautla, Ex-hacienda La Concepción S/N, San Agustín Tlaxiaca 42160, Hidalgo, MexicoÁrea Académica de Medicina, Instituto de Ciencias de la Salud, Universidad Autonoma del Estado de Hidalgo, Carretera Actopan-Tilcuautla, Ex-hacienda La Concepción S/N, San Agustín Tlaxiaca 42160, Hidalgo, MexicoÁrea Académica de Medicina, Instituto de Ciencias de la Salud, Universidad Autonoma del Estado de Hidalgo, Carretera Actopan-Tilcuautla, Ex-hacienda La Concepción S/N, San Agustín Tlaxiaca 42160, Hidalgo, MexicoTecnológico de Monterrey, Escuela de Ingeniería y Ciencias, Campus Querétaro, Epigmenio González 500, 76130 Querétaro, MexicoSección de Investigación y Posgrado, Escuela Superior de Medicina del Instituto Politécnico Nacional, Plan de San Luis y Díaz Mirón Col, Casco de Santo Tomas, Delegación Miguel Hidalgo, 11340 Ciudad de México, MexicoLaboratorio de Toxicología de la Reproducción-Fertilidad, Escuela Nacional de Ciencias Biológicas, Instituto Politécnico Nacional, Av Wilfrido Massieu S/N, Colonia Adolfo López Mateos, Ciudad de México 07738, MexicoÁrea Académica de Medicina, Instituto de Ciencias de la Salud, Universidad Autonoma del Estado de Hidalgo, Carretera Actopan-Tilcuautla, Ex-hacienda La Concepción S/N, San Agustín Tlaxiaca 42160, Hidalgo, MexicoDiabetes mellitus is the most common chronic disease worldwide that causes numerous complications, including male infertility. The prevalence of DM is 451 million people and estimated that would increase to 693 million in 2045. Fluorosis caused by drinking water contaminated with inorganic fluoride is a public health problem in many areas around the world. Previous studies have shown that fluoride exposure damages the male reproductive function. This study aimed to evaluate the fluoride sub-chronic exposure on the spermatozoa function in streptozotocin (STZ)-induced diabetic mice. After confirming diabetes by measuring blood glucose levels, the male mice received 45.2 ppm of fluoride added or deionized water. We evaluated several parameters in diabetic mice exposed to fluoride: standard quality analysis, the mitochondrial transmembrane potential (ψm), the caspase activity in spermatozoa, urinary fluoride excretion, and histological evaluation in the testes. After 60 days of fluoride-exposure, diabetic mice, significantly decreased sperm quality (motility, viability, and concentration). Spermatozoa from fluoride-exposure in diabetic mice presented a significant decrease in ψm and a significant increase in activity caspase 3/7. Urinary fluoride excretion was decreased in diabetic mice exposed to fluoride. Subchronic fluoride exposure of mice with STZ-induced diabetes aggravated testicular damage and the spermatozoa function.http://dx.doi.org/10.1155/2019/5269380
spellingShingle Manuel Sánchez-Gutiérrez
Evelia Martínez-Loredo
Eduardo Osiris Madrigal-Santillán
Gabriel Betanzos-Cabrera
Araceli Hernández-Zavala
María Angélica Mojica-Villegas
Jeannett Alejandra Izquierdo-Vega
Exposure of Fluoride with Streptozotocin-Induced Diabetes Aggravates Testicular Damage and Spermatozoa Parameters in Mice
Journal of Toxicology
title Exposure of Fluoride with Streptozotocin-Induced Diabetes Aggravates Testicular Damage and Spermatozoa Parameters in Mice
title_full Exposure of Fluoride with Streptozotocin-Induced Diabetes Aggravates Testicular Damage and Spermatozoa Parameters in Mice
title_fullStr Exposure of Fluoride with Streptozotocin-Induced Diabetes Aggravates Testicular Damage and Spermatozoa Parameters in Mice
title_full_unstemmed Exposure of Fluoride with Streptozotocin-Induced Diabetes Aggravates Testicular Damage and Spermatozoa Parameters in Mice
title_short Exposure of Fluoride with Streptozotocin-Induced Diabetes Aggravates Testicular Damage and Spermatozoa Parameters in Mice
title_sort exposure of fluoride with streptozotocin induced diabetes aggravates testicular damage and spermatozoa parameters in mice
url http://dx.doi.org/10.1155/2019/5269380
work_keys_str_mv AT manuelsanchezgutierrez exposureoffluoridewithstreptozotocininduceddiabetesaggravatestesticulardamageandspermatozoaparametersinmice
AT eveliamartinezloredo exposureoffluoridewithstreptozotocininduceddiabetesaggravatestesticulardamageandspermatozoaparametersinmice
AT eduardoosirismadrigalsantillan exposureoffluoridewithstreptozotocininduceddiabetesaggravatestesticulardamageandspermatozoaparametersinmice
AT gabrielbetanzoscabrera exposureoffluoridewithstreptozotocininduceddiabetesaggravatestesticulardamageandspermatozoaparametersinmice
AT aracelihernandezzavala exposureoffluoridewithstreptozotocininduceddiabetesaggravatestesticulardamageandspermatozoaparametersinmice
AT mariaangelicamojicavillegas exposureoffluoridewithstreptozotocininduceddiabetesaggravatestesticulardamageandspermatozoaparametersinmice
AT jeannettalejandraizquierdovega exposureoffluoridewithstreptozotocininduceddiabetesaggravatestesticulardamageandspermatozoaparametersinmice