Beneficial or Harmful Role of Macrophages in Guillain-Barré Syndrome and Experimental Autoimmune Neuritis

Guillain-Barré syndrome (GBS), an immune-mediated demyelinating peripheral neuropathy, is characterized by acute weakness of the extremities and areflexia or hyporeflexia. Experimental autoimmune neuritis (EAN) is a common animal model for GBS, which represents a CD4+ T cell-mediated inflammatory au...

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Main Authors: Donghui Shen, Fengna Chu, Yue Lang, Yunlong Geng, Xiangyu Zheng, Jie Zhu, Kangding Liu
Format: Article
Language:English
Published: Wiley 2018-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/2018/4286364
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author Donghui Shen
Fengna Chu
Yue Lang
Yunlong Geng
Xiangyu Zheng
Jie Zhu
Kangding Liu
author_facet Donghui Shen
Fengna Chu
Yue Lang
Yunlong Geng
Xiangyu Zheng
Jie Zhu
Kangding Liu
author_sort Donghui Shen
collection DOAJ
description Guillain-Barré syndrome (GBS), an immune-mediated demyelinating peripheral neuropathy, is characterized by acute weakness of the extremities and areflexia or hyporeflexia. Experimental autoimmune neuritis (EAN) is a common animal model for GBS, which represents a CD4+ T cell-mediated inflammatory autoimmune demyelination of the peripheral nervous system (PNS), and is used to investigate the pathogenic mechanism of GBS. It has been found that macrophages play a critical role in the pathogenesis of both GBS and EAN. Macrophages have been primarily classified into two major phenotypes: proinflammatory macrophages (M1) and anti-inflammatory macrophages (M2). The two different macrophage subsets M1 and M2 may play a decisive role in initiation and development of GBS and EAN. However, recently, it has been indicated that the roles of macrophages in immune regulation and autoimmune diseases are more complex than those suggested by a simple M1-M2 dichotomy. Macrophages might exert either inflammatory or anti-inflammatory effect by secreting pro- or anti-inflammatory cytokines, and either inducing the activation of T cells to mediate immune response, resulting in inflammation and demyelination in the PNS, or promoting disease recovery. In this review, we summarize the dual roles of macrophages in GBS and EAN and explore the mechanism of macrophage polarization to provide a potential therapeutic approach for GBS in the future.
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spelling doaj-art-540fadc78eb34198aa95d52073ca0f7b2025-08-20T03:36:25ZengWileyMediators of Inflammation0962-93511466-18612018-01-01201810.1155/2018/42863644286364Beneficial or Harmful Role of Macrophages in Guillain-Barré Syndrome and Experimental Autoimmune NeuritisDonghui Shen0Fengna Chu1Yue Lang2Yunlong Geng3Xiangyu Zheng4Jie Zhu5Kangding Liu6Neuroscience Center, Department of Neurology, The First Hospital of Jilin University, Jilin University, Changchun 130021, ChinaNeuroscience Center, Department of Neurology, The First Hospital of Jilin University, Jilin University, Changchun 130021, ChinaNeuroscience Center, Department of Neurology, The First Hospital of Jilin University, Jilin University, Changchun 130021, ChinaNeuroscience Center, Department of Neurology, The First Hospital of Jilin University, Jilin University, Changchun 130021, ChinaNeuroscience Center, Department of Neurology, The First Hospital of Jilin University, Jilin University, Changchun 130021, ChinaNeuroscience Center, Department of Neurology, The First Hospital of Jilin University, Jilin University, Changchun 130021, ChinaNeuroscience Center, Department of Neurology, The First Hospital of Jilin University, Jilin University, Changchun 130021, ChinaGuillain-Barré syndrome (GBS), an immune-mediated demyelinating peripheral neuropathy, is characterized by acute weakness of the extremities and areflexia or hyporeflexia. Experimental autoimmune neuritis (EAN) is a common animal model for GBS, which represents a CD4+ T cell-mediated inflammatory autoimmune demyelination of the peripheral nervous system (PNS), and is used to investigate the pathogenic mechanism of GBS. It has been found that macrophages play a critical role in the pathogenesis of both GBS and EAN. Macrophages have been primarily classified into two major phenotypes: proinflammatory macrophages (M1) and anti-inflammatory macrophages (M2). The two different macrophage subsets M1 and M2 may play a decisive role in initiation and development of GBS and EAN. However, recently, it has been indicated that the roles of macrophages in immune regulation and autoimmune diseases are more complex than those suggested by a simple M1-M2 dichotomy. Macrophages might exert either inflammatory or anti-inflammatory effect by secreting pro- or anti-inflammatory cytokines, and either inducing the activation of T cells to mediate immune response, resulting in inflammation and demyelination in the PNS, or promoting disease recovery. In this review, we summarize the dual roles of macrophages in GBS and EAN and explore the mechanism of macrophage polarization to provide a potential therapeutic approach for GBS in the future.http://dx.doi.org/10.1155/2018/4286364
spellingShingle Donghui Shen
Fengna Chu
Yue Lang
Yunlong Geng
Xiangyu Zheng
Jie Zhu
Kangding Liu
Beneficial or Harmful Role of Macrophages in Guillain-Barré Syndrome and Experimental Autoimmune Neuritis
Mediators of Inflammation
title Beneficial or Harmful Role of Macrophages in Guillain-Barré Syndrome and Experimental Autoimmune Neuritis
title_full Beneficial or Harmful Role of Macrophages in Guillain-Barré Syndrome and Experimental Autoimmune Neuritis
title_fullStr Beneficial or Harmful Role of Macrophages in Guillain-Barré Syndrome and Experimental Autoimmune Neuritis
title_full_unstemmed Beneficial or Harmful Role of Macrophages in Guillain-Barré Syndrome and Experimental Autoimmune Neuritis
title_short Beneficial or Harmful Role of Macrophages in Guillain-Barré Syndrome and Experimental Autoimmune Neuritis
title_sort beneficial or harmful role of macrophages in guillain barre syndrome and experimental autoimmune neuritis
url http://dx.doi.org/10.1155/2018/4286364
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