Angiogenesis-Related Cytokines, RANKL, and Osteoprotegerin in Multiple Myeloma Patients in relation to Clinical Features and Response to Treatment

An essential cytokine system for the osteoclast biology in multiple myeloma (MM) consists of the receptor of activator of NF-κB ligand (RANKL), its receptor (RANK), and the soluble decoy receptor, osteoprotegerin (OPG). Myeloma cells cause imbalance in OPG/RANKL interactions. We measured serum level...

Full description

Saved in:
Bibliographic Details
Main Authors: K. Sfiridaki, C. A. Pappa, G. Tsirakis, P. Kanellou, M. Kaparou, M. Stratinaki, G. Sakellaris, G. Kontakis, M. G. Alexandrakis
Format: Article
Language:English
Published: Wiley 2011-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/2011/867576
Tags: Add Tag
No Tags, Be the first to tag this record!
_version_ 1850224836490559488
author K. Sfiridaki
C. A. Pappa
G. Tsirakis
P. Kanellou
M. Kaparou
M. Stratinaki
G. Sakellaris
G. Kontakis
M. G. Alexandrakis
author_facet K. Sfiridaki
C. A. Pappa
G. Tsirakis
P. Kanellou
M. Kaparou
M. Stratinaki
G. Sakellaris
G. Kontakis
M. G. Alexandrakis
author_sort K. Sfiridaki
collection DOAJ
description An essential cytokine system for the osteoclast biology in multiple myeloma (MM) consists of the receptor of activator of NF-κB ligand (RANKL), its receptor (RANK), and the soluble decoy receptor, osteoprotegerin (OPG). Myeloma cells cause imbalance in OPG/RANKL interactions. We measured serum levels of OPG, soluble (s) RANKL, sRANKL/OPG ratio, markers of disease activity [LDH, CRP, interleukin-6 (IL-6), β2-microglobulin (B2M)], and angiogenic factors [hepatocyte growth factor (HGF), vascular endothelial growth factor (VEGF)], in 54 newly diagnosed MM patients and in 25 of them in plateau phase. All the above values were higher in MM patients compared to controls and decreased in plateau phase. sRANKL and RANKL/OPG were higher with advancing disease stage and skeletal grade. Significant correlations were found among RANKL and RANKL/OPG with HGF, LDH, VEGF, IL-6, and B2M. In conclusion, RANKL and OPG play significant roles in MM pathophysiology, as regulators of bone turnover and mediators of angiogenesis.
format Article
id doaj-art-5373bef82c2f4ff2ad00997785c50c1c
institution OA Journals
issn 0962-9351
1466-1861
language English
publishDate 2011-01-01
publisher Wiley
record_format Article
series Mediators of Inflammation
spelling doaj-art-5373bef82c2f4ff2ad00997785c50c1c2025-08-20T02:05:31ZengWileyMediators of Inflammation0962-93511466-18612011-01-01201110.1155/2011/867576867576Angiogenesis-Related Cytokines, RANKL, and Osteoprotegerin in Multiple Myeloma Patients in relation to Clinical Features and Response to TreatmentK. Sfiridaki0C. A. Pappa1G. Tsirakis2P. Kanellou3M. Kaparou4M. Stratinaki5G. Sakellaris6G. Kontakis7M. G. Alexandrakis8Blood Bank, Venizelion General Hospital of Heraklion, 71409 Crete, GreeceBlood Bank, Venizelion General Hospital of Heraklion, 71409 Crete, GreeceDepartment of Haematology, University Hospital of Heraklion, 70013 Crete, GreeceDepartment of Haematology, University Hospital of Heraklion, 70013 Crete, GreeceDepartment of Haematology, University Hospital of Heraklion, 70013 Crete, GreeceBlood Bank, Venizelion General Hospital of Heraklion, 71409 Crete, GreeceDepartment of Surgery, University Hospital of Heraklion, 70013 Crete, GreeceDepartment of Surgery, University Hospital of Heraklion, 70013 Crete, GreeceDepartment of Haematology, University Hospital of Heraklion, 70013 Crete, GreeceAn essential cytokine system for the osteoclast biology in multiple myeloma (MM) consists of the receptor of activator of NF-κB ligand (RANKL), its receptor (RANK), and the soluble decoy receptor, osteoprotegerin (OPG). Myeloma cells cause imbalance in OPG/RANKL interactions. We measured serum levels of OPG, soluble (s) RANKL, sRANKL/OPG ratio, markers of disease activity [LDH, CRP, interleukin-6 (IL-6), β2-microglobulin (B2M)], and angiogenic factors [hepatocyte growth factor (HGF), vascular endothelial growth factor (VEGF)], in 54 newly diagnosed MM patients and in 25 of them in plateau phase. All the above values were higher in MM patients compared to controls and decreased in plateau phase. sRANKL and RANKL/OPG were higher with advancing disease stage and skeletal grade. Significant correlations were found among RANKL and RANKL/OPG with HGF, LDH, VEGF, IL-6, and B2M. In conclusion, RANKL and OPG play significant roles in MM pathophysiology, as regulators of bone turnover and mediators of angiogenesis.http://dx.doi.org/10.1155/2011/867576
spellingShingle K. Sfiridaki
C. A. Pappa
G. Tsirakis
P. Kanellou
M. Kaparou
M. Stratinaki
G. Sakellaris
G. Kontakis
M. G. Alexandrakis
Angiogenesis-Related Cytokines, RANKL, and Osteoprotegerin in Multiple Myeloma Patients in relation to Clinical Features and Response to Treatment
Mediators of Inflammation
title Angiogenesis-Related Cytokines, RANKL, and Osteoprotegerin in Multiple Myeloma Patients in relation to Clinical Features and Response to Treatment
title_full Angiogenesis-Related Cytokines, RANKL, and Osteoprotegerin in Multiple Myeloma Patients in relation to Clinical Features and Response to Treatment
title_fullStr Angiogenesis-Related Cytokines, RANKL, and Osteoprotegerin in Multiple Myeloma Patients in relation to Clinical Features and Response to Treatment
title_full_unstemmed Angiogenesis-Related Cytokines, RANKL, and Osteoprotegerin in Multiple Myeloma Patients in relation to Clinical Features and Response to Treatment
title_short Angiogenesis-Related Cytokines, RANKL, and Osteoprotegerin in Multiple Myeloma Patients in relation to Clinical Features and Response to Treatment
title_sort angiogenesis related cytokines rankl and osteoprotegerin in multiple myeloma patients in relation to clinical features and response to treatment
url http://dx.doi.org/10.1155/2011/867576
work_keys_str_mv AT ksfiridaki angiogenesisrelatedcytokinesranklandosteoprotegerininmultiplemyelomapatientsinrelationtoclinicalfeaturesandresponsetotreatment
AT capappa angiogenesisrelatedcytokinesranklandosteoprotegerininmultiplemyelomapatientsinrelationtoclinicalfeaturesandresponsetotreatment
AT gtsirakis angiogenesisrelatedcytokinesranklandosteoprotegerininmultiplemyelomapatientsinrelationtoclinicalfeaturesandresponsetotreatment
AT pkanellou angiogenesisrelatedcytokinesranklandosteoprotegerininmultiplemyelomapatientsinrelationtoclinicalfeaturesandresponsetotreatment
AT mkaparou angiogenesisrelatedcytokinesranklandosteoprotegerininmultiplemyelomapatientsinrelationtoclinicalfeaturesandresponsetotreatment
AT mstratinaki angiogenesisrelatedcytokinesranklandosteoprotegerininmultiplemyelomapatientsinrelationtoclinicalfeaturesandresponsetotreatment
AT gsakellaris angiogenesisrelatedcytokinesranklandosteoprotegerininmultiplemyelomapatientsinrelationtoclinicalfeaturesandresponsetotreatment
AT gkontakis angiogenesisrelatedcytokinesranklandosteoprotegerininmultiplemyelomapatientsinrelationtoclinicalfeaturesandresponsetotreatment
AT mgalexandrakis angiogenesisrelatedcytokinesranklandosteoprotegerininmultiplemyelomapatientsinrelationtoclinicalfeaturesandresponsetotreatment