Periodontal disease influences osteoclastogenic bone markers in subjects with and without rheumatoid arthritis.

<h4>Background</h4>Periodontal disease (PD) and rheumatoid arthritis (RA) are bone pathologies mediated through immuno-inflammatory mechanisms. The aim of this study was to investigate the serum markers osteopontin (OPN), tumor necrosis factor receptors 1 (TNFR1) and 2 (TNFR2) receptor a...

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Main Authors: Jeneen Panezai, Ambereen Ghaffar, Mohammad Altamash, Per-Erik Engström, Anders Larsson
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2018-01-01
Series:PLoS ONE
Online Access:https://storage.googleapis.com/plos-corpus-prod/10.1371/journal.pone.0197235/1/pone.0197235.pdf?X-Goog-Algorithm=GOOG4-RSA-SHA256&X-Goog-Credential=wombat-sa%40plos-prod.iam.gserviceaccount.com%2F20210218%2Fauto%2Fstorage%2Fgoog4_request&X-Goog-Date=20210218T134530Z&X-Goog-Expires=3600&X-Goog-SignedHeaders=host&X-Goog-Signature=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author Jeneen Panezai
Ambereen Ghaffar
Mohammad Altamash
Per-Erik Engström
Anders Larsson
author_facet Jeneen Panezai
Ambereen Ghaffar
Mohammad Altamash
Per-Erik Engström
Anders Larsson
author_sort Jeneen Panezai
collection DOAJ
description <h4>Background</h4>Periodontal disease (PD) and rheumatoid arthritis (RA) are bone pathologies mediated through immuno-inflammatory mechanisms. The aim of this study was to investigate the serum markers osteopontin (OPN), tumor necrosis factor receptors 1 (TNFR1) and 2 (TNFR2) receptor activator of nuclear factor-kappa B ligand (RANKL) and RANKL/ osteoprotegerin (OPG) ratio and compare them in PD and RA groups.<h4>Materials & methods</h4>RA (with PD = 19 and without PD = 19), PD (n = 38) and 14 healthy subjects underwent bleeding on probing (BOP) and probing pocket depth (PPD) measurement. PD was defined as PPD measuring ≥5mm registered in ≥3 sites. Marginal bone loss (MBL) for premolars and molars was measured on digital panoramic radiographs. Serum samples were collected from all subjects. OPN, TNFR1, TNFR2 and RANKL were measured by enzyme-linked immunosorbent assays (ELISAs). OPG was measured as part of a multiplex proximity extension assay (PEA).<h4>Results</h4>OPN, TNFR1, TNFR2 and RANKL serum levels were the highest in the RA group with PD, while the RA group without PD were comparable to PD subjects only. The RANKL/OPG ratios were comparable between PD group and both RA groups with (p = 0.051) and without PD (p = 0.37). Serum RANKL levels were associated with MBL (p = 0.008) and PPD ≥ 5mm (p = 0.01).<h4>Conclusion</h4>Peripheral osteoclastogenesis is a feature of periodontal disease with systemic levels of osteoclastogenic markers comparable to the effects observed in rheumatoid arthritis.
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spelling doaj-art-5213e7d0ec48475bbc46514768e1405f2025-08-20T02:45:57ZengPublic Library of Science (PLoS)PLoS ONE1932-62032018-01-01136e019723510.1371/journal.pone.0197235Periodontal disease influences osteoclastogenic bone markers in subjects with and without rheumatoid arthritis.Jeneen PanezaiAmbereen GhaffarMohammad AltamashPer-Erik EngströmAnders Larsson<h4>Background</h4>Periodontal disease (PD) and rheumatoid arthritis (RA) are bone pathologies mediated through immuno-inflammatory mechanisms. The aim of this study was to investigate the serum markers osteopontin (OPN), tumor necrosis factor receptors 1 (TNFR1) and 2 (TNFR2) receptor activator of nuclear factor-kappa B ligand (RANKL) and RANKL/ osteoprotegerin (OPG) ratio and compare them in PD and RA groups.<h4>Materials & methods</h4>RA (with PD = 19 and without PD = 19), PD (n = 38) and 14 healthy subjects underwent bleeding on probing (BOP) and probing pocket depth (PPD) measurement. PD was defined as PPD measuring ≥5mm registered in ≥3 sites. Marginal bone loss (MBL) for premolars and molars was measured on digital panoramic radiographs. Serum samples were collected from all subjects. OPN, TNFR1, TNFR2 and RANKL were measured by enzyme-linked immunosorbent assays (ELISAs). OPG was measured as part of a multiplex proximity extension assay (PEA).<h4>Results</h4>OPN, TNFR1, TNFR2 and RANKL serum levels were the highest in the RA group with PD, while the RA group without PD were comparable to PD subjects only. The RANKL/OPG ratios were comparable between PD group and both RA groups with (p = 0.051) and without PD (p = 0.37). Serum RANKL levels were associated with MBL (p = 0.008) and PPD ≥ 5mm (p = 0.01).<h4>Conclusion</h4>Peripheral osteoclastogenesis is a feature of periodontal disease with systemic levels of osteoclastogenic markers comparable to the effects observed in rheumatoid arthritis.https://storage.googleapis.com/plos-corpus-prod/10.1371/journal.pone.0197235/1/pone.0197235.pdf?X-Goog-Algorithm=GOOG4-RSA-SHA256&X-Goog-Credential=wombat-sa%40plos-prod.iam.gserviceaccount.com%2F20210218%2Fauto%2Fstorage%2Fgoog4_request&X-Goog-Date=20210218T134530Z&X-Goog-Expires=3600&X-Goog-SignedHeaders=host&X-Goog-Signature=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
spellingShingle Jeneen Panezai
Ambereen Ghaffar
Mohammad Altamash
Per-Erik Engström
Anders Larsson
Periodontal disease influences osteoclastogenic bone markers in subjects with and without rheumatoid arthritis.
PLoS ONE
title Periodontal disease influences osteoclastogenic bone markers in subjects with and without rheumatoid arthritis.
title_full Periodontal disease influences osteoclastogenic bone markers in subjects with and without rheumatoid arthritis.
title_fullStr Periodontal disease influences osteoclastogenic bone markers in subjects with and without rheumatoid arthritis.
title_full_unstemmed Periodontal disease influences osteoclastogenic bone markers in subjects with and without rheumatoid arthritis.
title_short Periodontal disease influences osteoclastogenic bone markers in subjects with and without rheumatoid arthritis.
title_sort periodontal disease influences osteoclastogenic bone markers in subjects with and without rheumatoid arthritis
url https://storage.googleapis.com/plos-corpus-prod/10.1371/journal.pone.0197235/1/pone.0197235.pdf?X-Goog-Algorithm=GOOG4-RSA-SHA256&X-Goog-Credential=wombat-sa%40plos-prod.iam.gserviceaccount.com%2F20210218%2Fauto%2Fstorage%2Fgoog4_request&X-Goog-Date=20210218T134530Z&X-Goog-Expires=3600&X-Goog-SignedHeaders=host&X-Goog-Signature=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