ITGB2 and ICAM3 predict increased survival of sepsis with decreased intercellular communication in cytotoxic CD8+ T cells

Abstract Sepsis is closely linked to immunity. Our research aimed to identify key genes associated with sepsis immunity utilizing single-cell RNA sequencing (scRNA-seq) data. This study obtained the GSE167363 and GSE54514 datasets from the Gene Expression Omnibus (GEO). The GSE167363 dataset was sub...

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Main Authors: Min Lei, Yaping Zhang, Yijin Yu, Gaojian Wang, Nianqiang Hu, Junran Xie
Format: Article
Language:English
Published: Nature Portfolio 2025-04-01
Series:Scientific Reports
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Online Access:https://doi.org/10.1038/s41598-025-93685-z
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author Min Lei
Yaping Zhang
Yijin Yu
Gaojian Wang
Nianqiang Hu
Junran Xie
author_facet Min Lei
Yaping Zhang
Yijin Yu
Gaojian Wang
Nianqiang Hu
Junran Xie
author_sort Min Lei
collection DOAJ
description Abstract Sepsis is closely linked to immunity. Our research aimed to identify key genes associated with sepsis immunity utilizing single-cell RNA sequencing (scRNA-seq) data. This study obtained the GSE167363 and GSE54514 datasets from the Gene Expression Omnibus (GEO). The GSE167363 dataset was subjected to cluster analysis, cell proportion analysis, cell interaction analysis, and gene set enrichment analysis (GSEA). The differentially expressed genes (DEGs) of CD8+ T cells were intersected with the DEGs in the GSE54514 dataset, and key genes related to immunity in sepsis patients were identified through Kyoto Encyclopedia of Genes and Genomes (KEGG) analysis. Finally, we validated the gene expression levels in a mouse model of sepsis caused by cecum ligation and puncture (CLP). Findings indicated that intercellular communication of Cytotoxic CD8+ T cells was reduced in the sepsis survivors compared to non-survivors. The expression of 3 down-regulated key DEGs (ITGB2, SELL and ICAM3) was negatively correlated with the abundance of CD8+ T cells. Moreover, Cytotoxic CD8+ T cells with low expression of ITGB2, SELL and ICAM3 were more adverse to the survival of sepsis as compared to those with high expression of the above genes. These genes may predict increased survival in sepsis by regulating intercellular communication in cytotoxic CD8+ T cells, suggesting that they are potential therapeutic targets for improving sepsis prognosis.
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spelling doaj-art-5185d3c6ec7e41538ebfaef7e07fe75c2025-08-20T03:06:49ZengNature PortfolioScientific Reports2045-23222025-04-0115111410.1038/s41598-025-93685-zITGB2 and ICAM3 predict increased survival of sepsis with decreased intercellular communication in cytotoxic CD8+ T cellsMin Lei0Yaping Zhang1Yijin Yu2Gaojian Wang3Nianqiang Hu4Junran Xie5Department of Anesthesiology, Sir Run Run Shaw Hospital, Zhejiang University School of MedicineDepartment of Anesthesiology, Sir Run Run Shaw Hospital, Zhejiang University School of MedicineDepartment of Anesthesiology, Sir Run Run Shaw Hospital, Zhejiang University School of MedicineDepartment of Anesthesiology, Sir Run Run Shaw Hospital, Zhejiang University School of MedicineDepartment of Anesthesiology, Sir Run Run Shaw Hospital, Zhejiang University School of MedicineDepartment of Anesthesiology, Sir Run Run Shaw Hospital, Zhejiang University School of MedicineAbstract Sepsis is closely linked to immunity. Our research aimed to identify key genes associated with sepsis immunity utilizing single-cell RNA sequencing (scRNA-seq) data. This study obtained the GSE167363 and GSE54514 datasets from the Gene Expression Omnibus (GEO). The GSE167363 dataset was subjected to cluster analysis, cell proportion analysis, cell interaction analysis, and gene set enrichment analysis (GSEA). The differentially expressed genes (DEGs) of CD8+ T cells were intersected with the DEGs in the GSE54514 dataset, and key genes related to immunity in sepsis patients were identified through Kyoto Encyclopedia of Genes and Genomes (KEGG) analysis. Finally, we validated the gene expression levels in a mouse model of sepsis caused by cecum ligation and puncture (CLP). Findings indicated that intercellular communication of Cytotoxic CD8+ T cells was reduced in the sepsis survivors compared to non-survivors. The expression of 3 down-regulated key DEGs (ITGB2, SELL and ICAM3) was negatively correlated with the abundance of CD8+ T cells. Moreover, Cytotoxic CD8+ T cells with low expression of ITGB2, SELL and ICAM3 were more adverse to the survival of sepsis as compared to those with high expression of the above genes. These genes may predict increased survival in sepsis by regulating intercellular communication in cytotoxic CD8+ T cells, suggesting that they are potential therapeutic targets for improving sepsis prognosis.https://doi.org/10.1038/s41598-025-93685-zSepsisImmunityCytotoxic CD8+ T cellsITGB2ICAM3scRNA-seq
spellingShingle Min Lei
Yaping Zhang
Yijin Yu
Gaojian Wang
Nianqiang Hu
Junran Xie
ITGB2 and ICAM3 predict increased survival of sepsis with decreased intercellular communication in cytotoxic CD8+ T cells
Scientific Reports
Sepsis
Immunity
Cytotoxic CD8+ T cells
ITGB2
ICAM3
scRNA-seq
title ITGB2 and ICAM3 predict increased survival of sepsis with decreased intercellular communication in cytotoxic CD8+ T cells
title_full ITGB2 and ICAM3 predict increased survival of sepsis with decreased intercellular communication in cytotoxic CD8+ T cells
title_fullStr ITGB2 and ICAM3 predict increased survival of sepsis with decreased intercellular communication in cytotoxic CD8+ T cells
title_full_unstemmed ITGB2 and ICAM3 predict increased survival of sepsis with decreased intercellular communication in cytotoxic CD8+ T cells
title_short ITGB2 and ICAM3 predict increased survival of sepsis with decreased intercellular communication in cytotoxic CD8+ T cells
title_sort itgb2 and icam3 predict increased survival of sepsis with decreased intercellular communication in cytotoxic cd8 t cells
topic Sepsis
Immunity
Cytotoxic CD8+ T cells
ITGB2
ICAM3
scRNA-seq
url https://doi.org/10.1038/s41598-025-93685-z
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